Cat Surg Greenhorns
Cat Surg Greenhorns
SURGERY
FOR
GREENHORNS
Thomas A. Oetting
C ATA R A C T S U R G E RY F O R G R E E N H O R N S
University of Iowa
VAMC Iowa City
Copyright ©
Thomas A. Oetting, MS, MD
Contents
1. Training Plan . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 1
1.1. First Year – Beginner . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 1
1.2. Second Year – Advanced Beginner. . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 2
1.3. Third Year – Proficient . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 4
2. Assessment . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 8
2.1. Classic Types of Cataract . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 8
2.1.1. Nuclear Cataract . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 8
2.1.2. Posterior Subcapsular Plaque (PSCP) . . . . . . . . . . . . . . . . . . . . . . . . 8
2.1.3. Cortical Cataract . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 9
2.2. Evaluation of Patients with Cataract . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 10
2.2.1. Symptoms of a Cataract . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 10
2.2.2. Indications for Cataract Surgery. . . . . . . . . . . . . . . . . . . . . . . . . . . . 10
2.2.3. General Issues . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 11
2.2.4. Past Ocular History . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 11
2.2.5. Preoperative Exam . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 11
2.3. Difficulty Factors . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 12
3. Preoperative . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 15
3.1. Consent . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 15
3.2. Selecting the Intraocular Lens (IOL) . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 16
3.2.1. Brief History of the IOL. . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 16
3.2.2. Today. . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 16
3.2.3. IOL Material Considerations . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 16
3.2.4. IOL Design Considerations . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 17
3.2.5. Four Things You Need to Know to Calculate Correct IOL Power . . . . . . . . 17
3.2.6. Estimating the IOL Power for Emmetropia . . . . . . . . . . . . . . . . . . . . . 19
3.2.7. Selecting the IOL Power for Your Patient . . . . . . . . . . . . . . . . . . . . . . 19
3.3. Operating Microscope Basics . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 20
4. Anesthesia . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 22
4.1. Retrobulbar Procedure. . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 22
4.2. Subtenon’s Procedure . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 23
4.3. Topical Anesthesia . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 24
Our training plan sets up Dreyfus stages for the acquisition of the skill of cataract surgery
and more specifically phacoemulsification. A beginner has some skills in the wet lab and can
do parts of cases. An advanced beginner can do simple cases with one hand. A proficient
surgeon can do routine cases with both hands and some complex cases. Residents would not be
expected to be an expert who could do routine cases with little thought and develop techniques
for more complex cases. Each stage has expectations associated with it.
1
Expectation Assessment Resources
iPod,
Know accepted names of all instruments in VA demonstrate in
Greenhorns,
cataract tray. OR
[Link]
2
Resources to develop these skills:
• wet lab fully equipped, dedicated facility at VAMC, work in wet lab w/ cadaver or pig
eyes
• [Link] or download to iPad using
iTunes
• [Link]
Phacoemulsification/[Link]
• [Link]
• Observed Professional Communication Competency Consent feedback on EPIC
• OR formative feedback form for portfolio
– VA rotation – Wed and Friday AM, observe 3rd year 1st case
– VA rotation – Thursday AM, Dr. Oetting, 1–3 cases (Dr. Oetting will help with
second instrument at first usually with retrobulbar anesthesia), all cases video taped
for review
– UIHC rotation – Tuesday, Dr. Johnson transition to topical anesthesia, deliberate
practice on capsulorhexis, video formative feedback
• M&M Conf Practice Based Learning Competency, every 10 weeks
• Phaco Course Madison Wisconsin (UW, UI, MCW residents), advanced lectures 1⁄2 day /
wet lab 1⁄2 day
• texts in library: David F. Chang. Phaco Chop: Mastering Techniques, Optimizing
Technology, and Avoiding Complications. 1st edition. Slack Inc, 2005; Barry Seibel.
Phacodynamics: Mastering the Tools and Techniques of Phacoemulsification Surgery. 4th
edition. Slack Inc, 2005.
3
1.3. Third Year – Proficient
4
Demonstrate or deeply understand conversion to Greenhorns,
oral
ECCE. Facebook
Demonstrate or deeply understand anterior vit- Greenhorns,
oral
rectomy. Facebook
Demonstrate or understand sulcus IOL place- Greenhorns,
oral
ment. Facebook
Understand phacoemulsification machine set- Greenhorns,
oral
tings. Facebook
Greenhorns,
Understand OVD selection. oral
Facebook
Greenhorns,
Demonstrate ability to use iris hooks. observe in OR
Facebook
5
Typical % Resources
Dreyfus Expected samples of rotation grads to grow
Level
Stage behavior for this level at this at this beyond
level level this level
books,
starting Desire to learn. n/a video,
observe
Demonstrate sterile technique,
know all instruments in tray, books,
assistant know all steps of cataract video,
VA 1st yr
Novice surgeon surgery, demonstrate prep and wet lab,
drape, demonstrate IOL fold, observe
demonstrate RB injection.
wet lab,
wet lab Demonstrate microscope use, video,
VA 1st yr
surgeon pig/cadaver eye with faculty. back into
cases
wet lab,
Demonstrate suture technique,
neophyte video,
demonstrate IOL placement, VA 1st yr
Beginner surgeon back into
demonstrate use of I/A device.
cases
Demonstrate 5 cases < 45 wet lab,
min, know steps to convert to video,
basic
ECCE, know steps for vitreous develop
cataract VA 2nd yr 100%
loss, demonstrate use of cap- non-
surgeon
sule dye, demonstrate effective dominant
Advanced consent. hand
Beginner
assistant Demonstrate capsulorhexis video,
topical during topical case, assist UI 2nd yr 100% deliberate
surgeon efficient cataract surgeon. practice
Demonstrate 5 cases < 30
min using both hands, demon-
strate topical cases, demon-
two
strate the use of small pupil
handed VA 3rd yr 95% video
techniques, demonstrate the
surgeon
use of CTR, demonstrate chop-
ping techniques, demonstrate
Proficient
IOL suturing techniques.
VA 3rd yr
advanced video,
DM 3rd yr 50%
surgeon practice
UI 3rd yr
6
efficient
innova- Demonstrate 5 cases < 15 min, video,
Expert graduates rare
tive develop new techniques. practice
surgeon
7
2. Assessment
Epidemiology/risk factors:
• age
• riboflavin, Vit C, Vit E and carotene may decrease risk of nuclear sclerosis
• cigarette smoking increases the risk of nuclear sclerosis
Symptoms/history:
• gradual progressive loss of vision
• second sight – development of myopia due to increased lenticular refractive index
• monocular diplopia
• decreased color discrimination, especially blue
Clinical features:
• central yellow to brown discoloration of the lens
• myopic shift – increased AP diameter better converging lens
• bilateral
• decreased penetration of cobalt blue slit beam through lens
Epidemiology/risk factors:
• younger patients than with nuclear or cortical cataracts
• diabetes mellitus
• radiation
• corticosteroids
• uveitis including RP
8
• smoking
Symptoms/history:
• progressive loss of vision, sometimes rapid
• glare, halos
• monocular diplopia
Clinical features:
• axial opacity of the posterior cortical material
– initially an iridescent sheen appears in the posterior cortex
– followed by granular and plaque like opacities
• can be confused with posterior polar cataract and Mittendorf dot
• can see with direct but best viewed with red reflex through slit lamp
Figure 1. A) Using direct to see PSCP, B) view through direct, C) view through slit lamp
Epidemiology/risk factors:
• younger patients than with nuclear cataracts
• diabetes mellitus
• sunlight
• trauma
• smoking
Symptoms/history:
• progressive loss of vision, sometimes rapid
• glare, halos
• monocular diplopia
Clinical features:
• opacity of the cortical fibres from posterior to central
• sometimes wedge shaped forming cortical spokes
• can progress to intumescent or hypermature cataract
• usually medial and inferior from UV exposure
9
Figure 2. Cortical cataract: A) right eye, B) left eye
Ask yourself!
• Is the cataract causing the visual decline?
• Is the cataract secondary to a systemic or ocular condition?
• Could the eye/patient survive cataract surgery if indicated?
Visual acuity:
• usually a gradual decline over years
• w/ post subcapsular cataract (PSCC) VA can decline over days
• often near VA decline is greater than far VA decline in PSCC
Glare:
• night driving problems, halos, especially with PSCC and cortical
Myopic shift:
• “second sight”, especially in nuclear sclerotic cataract
Diplopia:
• monocular, especially in PSCC and cortical
10
2.2.3. General Issues
11
Pupils:
• RAPD – as always critical – especially if patient’s vision remains poor after surgery
• dilated pupil size – useful when selecting among surgeons (see difficulty factors)
CVF – LP in all four quadrants in dense cataracts (instead of echo).
• which surgeon should do case (e.g. attending vs. 1st year resident)
12
• estimate length of case
• determine need for additional supplies/equipment
• determine the type of anesthesia
Difficulty factors (in decreasing order of importance):
• zonular laxity (PXF, h/o trauma, Marfan’s)
• small pupil – consider why? PXF, DM s/p laser, CPS, alpha blocker (e.g. tamsulosin)
• cannot lay flat for very long (e.g. COPD, claustrophobia, tremor, severe obesity)
• poor red reflex – white/black cataract making CCC difficult
• big brow limiting superior access
• narrow angle limiting AC space
• predisposition to corneal decompensation (e.g. guttata, PPMD, hard nucleus)
• past surgery such as existing trab or past PPVx
• predisposition to exposure (e.g. BOTOX® , past lid trauma, DM)
• anticoagulants (e.g. Coumadin, ASA)
• monocular
13
Consider smaller phaco tip.
Beware of IFIS.
Consider iris hooks diamond config-
narrow angle > 50 cases add 25%
uration.
Consider BSS+ (w/ glutathione).
Arshinoff soft shell w/ OVD.
Consider BSS+ (w/ glutathione).
predisposition K Phaco chop.
> 50 cases 0%
decomp Arshinoff soft shell w/ OVD.
Consider ECCE , MSICS.
Avoid fixation ring.
Avoid conj manipulation.
existing trab > 20 cases 0%
Malyugin w/ small pupil.
Suture wound following surgery.
Topical w/ long eye to avoid RB.
past PPVx > 20 cases 0% Possible CTR.
Careful during I/A.
MAC.
cannot lay flat > 100 cases 0%
Consider general.
Topical to avoid injection risk.
anticoagulants > 20 cases 0% If needed, subtenon’s infusion.
Plavix is most scary.
Topical for faster rehabilitation.
monocular > 100 cases 0%
Try to forget about it.
14
3. Preoperative
3.1. Consent
15
3.2. Selecting the Intraocular Lens (IOL)
Harold Ridley placed first lens in 1949, a huge PMMA IOL (about the size of the crystalline
lens).
1950s rigid anterior chamber (AC) IOLs were used with ECCE and ICCE:
• bullous keratopathy was common
• chronic inflammation led to CME and glaucoma
Later, iris fixation lenses were used to avoid contact with the angle:
• some IOLs would suture onto the iris; others would clip on like Artisan today
• these lenses would frequently dislocate
Closed-loop flexible anterior chamber (AC) lenses were next:
• kept PK surgeons in business
• caused UGH syndrome (Ellingson syndrome)
Foldable IOLs came in the ’90s and allowed smaller incisions.
Multifocal and toric IOLs came more recently.
Rare and weird names for IOL: pseudophakos, lenticulus.
3.2.2. Today
16
more inflammation,
cost,
silicone oil (for RD repair)
silicone injectable,
adheres to IOL and becomes
less dysphotopsia
opaque
3.2.5. Four Things You Need to Know to Calculate Correct IOL Power
1) Desired postoperative SE
17
2) Axial eye length (AEL)
The more anterior the IOL, the less power the IOL needs.
• e.g. IOL placed in the sulcus needs less power
• AC depth is a factor in some IOL formula (e.g. Holladay 2)
Goal is to place a posterior chamber (PC) lens.
• these can end up in the bag (best) or sulcus (anterior to ant. capsule)
• placement of IOL measured for bag in the sulcus results in myopic surprise
– decrease power by 0.5 to 1 diopter (shorter eye larger shift)
– if primary lens is a single piece acrylic (e.g. SN60WF) have 3-piece available for sulcus
– see: [Link]
Always plan to have available anterior chamber (AC) lenses.
• these are placed anterior to the iris w/haptics that settle into the angle
• these are used when the capsule is lost and cannot hold an IOL
• when too small they can tilt and when too large they can hurt
• place peripheral iridotomy before AC IOL to prevent iris bombé
18
3.2.6. Estimating the IOL Power for Emmetropia
N N
IOL = −
AEL − ACD K − ACD
But you don’t know ACD or postoperative depth of the IOL preop!
SRK – classic regression formula (developed in 1980 by Sanders, Retzlaff, and Kraff):
IOL = A-constant - 2, 5 × AL - 0, 9 × K
The SRK computes the lens power for emmetropia, but you may have a different goal.
The U/S or IOLMaster produces a table with IOL power mapped to desired postop SE.
• roughly a change in IOL power of 1.5 produces a change of 1.0 in glasses
• e.g. formula gives 19 diopters for emmetropia; about 20.5 will give −1.00 SE p/o
• if your estimated IOL power is unusual, you are probably wrong
• double check your calculations
19
• trust what happened w/ the other eye’s IOL if applicable
• if the eye seems too short, ask yourself was the patient hyperopic as a young person (e.g. in
the big war before the myopic shift from the cataract)
Estimate the power for both the AC and the PC lens. Compare several formulas.
Then, if convinced that the calculations are right, make sure that all potentially needed
IOL powers are available in the OR.
• you will need a posterior chamber lens for the bag (1-piece or 3-piece)
• you will need a 3-piece IOL for the sulcus
– the more anterior sulcus lens will need 0.5–1.0 less diopters of power than it would if
placed in the bag
– cannot use 1-piece acrylic (Alcon SA60AT or SN60WF) in sulcus
• you will need an AC IOL
– typical AC lenses come in 3 diameters: 12.5, 13.0, and 13.5 mm
– sized at surgery by adding 1 mm to the “white to white” limbal diameter
Learn how to use your foot pedal and practice before your first case.
20
Sometimes hard to get legs un-
Avoid brow. der table.
temporal
Avoid bleb. Iris trauma can cause glare.
May not allow incision on axis.
21
4. Anesthesia
Pros:
• great for long cases (> 45 minutes)
• great for inexperienced surgeon (get akinesia, proptosis)
• proptosis helps to increase exposure
• quiets nystagmus (can be used for Nd:YAG laser w/ nystagmus also)
Cons:
• blood thinners (+/- sev. studies show bleeding risk low for ASA & Coumadin)
• monocular (RB injection can force admission until patch removed)
• risk of globe injury, especially with long eyes
• tricky with patients following scleral buckle
22
Procedure:
1. Place 1 gtt of topical anesthetic into both eyes.
2. Clean lower lid with alcohol wipe.
3. Fill 5 cc syringe with mixture of lidocaine/bupivacaine/widase without epinephrine.
4. Place blunt 23 gauge needle on needle (blunt needle limits risk of globe perforation).
5. Start at the lateral lower lid about 3/4 of the way from the medial side.
6. Use the index finger of non-dominant hand to create space between floor and globe.
7. Aim perpendicular to lid until passing through the septum (1st pop).
8. Then redirect more superiorly advancing about 1 – 1.5 inches (2nd pop) into muscle cone.
9. First pull syringe back to ensure you are not in a blood vessel.
10. Inject 4 cc slowly into retrobulbar space.
11. Retract needle until just under skin to level of orbicularis mm.
12. Inject remaining 1 cc to block facial nerve to prevent squeezing.
13. Have patient look straight ahead during procedure.
14. Apply pressure on closed eye for a minute or so – be alert for retrobulbar hemorrhage.
15. See video at: [Link]
Pros:
• great when topical case is getting complicated (e.g. convert to ECCE, ant vit)
• great for pts on blood thinners to limit risk of retrobulbar injection
Cons:
• conjunctiva gets red
• postop foreign body sensation
• conjunctival chemosis can be a problem
Procedure:
1. Give topical anesthesia (probably already done if converting from topical case).
2. Prepare 3 cc syringe with lidocaine/bupivacaine or use preservative free lidocaine.
23
3. Place lacrimal cannula with gentle curve to approximate that of the globe (also can get
Masket cannula (or others) designed for this purpose).
4. Pick a quadrant for the block (best to go for a lateral quadrant to avoid oblique mm).
5. Have the patient look away from the chosen quadrant to increase exposure.
6. Use .12 forceps to retract conjunctiva.
7. Make small incision down to sclera with Westcott scissors.
8. Redirect Westcott scissors with curve down and bluntly dissect through quadrant.
9. Dissect past the equator (similar to using Stevens tenotomy scissors in peds/retina).
10. Use .12 forceps on posterior conjunctiva for counter traction.
11. Place cannula through incision and direct past the equator before injecting.
12. Inject the anesthetic which should flow easily and cause minimal chemosis.
13. If anesthetic does not flow easily, dissect further posterior with Westcott scissors.
Pros:
• experienced fast surgeon
• monocular patients get fast rehab
• great for long eyes to limit risk of injection
• decreased risk of retrobulbar bleeding injection (esp. with Plavix > ASA ≥ Coumadin)
Cons:
• greenhorn surgeons need akinesia
• cannot use in patients with nystagmus
Intracameral:
• 1% nonpreserved lidocaine in anterior chamber can supplement topical
• many studies have shown no comfort benefit of intracameral
• helps with mydriasis
• if the case is long or if iris is moving, it seems to help in my hands
• usually placed just after paracentesis
• use about 0.5 cc of preservative free 1% lidocaine (can add epinephrine )
24
• may sting a bit so I usually warn the patient: “I’m giving you the rest of the numbing
medicine and you may feel it for a second or two and then it will do its magic”
25
5. Cataract Surgery – Old School
5.1. ICCE
Indications:
• rarely indicated today – I do about one case a year
• unstable lenses with severe zonular laxity
Be careful:
• children
• capsular rupture
• high myopia
• Marfan’s
26
• vitreous present
Preop:
• orbital massage or osmotic agents to reduce vitreous pressure
Anesthesia:
• retrobulbar and lid block
• rarely general anesthesia, e.g. claustrophobia, dementia, tremor
Procedure:
1. Superior bridle suture.
2. May need a scleral support ring in high myopes.
3. Peritomy of about 170 degrees.
4. Limbal incision of about 170 degrees chord length in the 11–12 mm range.
5. Safety sutures are preplaced – usually 7-0 vicryl.
6. Small peripheral iridotomy is placed.
7. Alpha-chymotrypsin was placed to degrade zonules (no longer avail in US).
8. Anterior surface of the lens is dried with a cellulose sponge.
9. Cryoprobe is placed on mid-periphery of the lens and frozen.
10. Lens is removed with a side to side motion through incision.
11. Wound is closed with safety sutures.
12. Vitreous is attended to if needed.
13. Anterior chamber lens is placed after placing PI with anterior vitrector.
14. Wound is closed with 10-0 nylon.
Indications:
• still indicated today (small incision variant, MSICS, very popular worldwide)
• hard lenses with tentative corneal endothelium
Contraindications:
• poor zonular support
27
• soft lens
Preop:
• consider orbital massage or osmotic agents to reduce vitreous pressure
Anesthesia:
• retrobulbar and lid block
• subtenon’s block
• rarely general anesthesia, e.g. claustrophobia, dementia, tremor
Procedure:
1. Superior bridle suture.
2. Peritomy of about 170 degrees.
3. Initial limbal groove in sclera with a chord length in the 11 mm range.
4. Initial entry into anterior chamber to allow capsulotomy (3 mm).
5. Instill viscoelastic.
6. Remove anterior capsule (usually with can opener approach).
7. Mobilize lens (physically with cystitome or with hydrodissection – be careful).
8. Extend initial incision to full length of groove (with scissors or knife).
9. Safety sutures are preplaced, usually 7-0 vicryl.
10. Lens removed w/ lens loop or w/ counter pressure technique.
11. Wound is closed with safety sutures.
12. Cortical material is removed using I/A device (either automated or manual).
13. Instill ophthalmic viscoelastic device (OVD).
14. Lens is placed in the posterior chamber.
15. Wound is closed with 10-0 nylon.
16. OVD is removed.
Indications:
• budget constrained
• hard lenses with tentative corneal endothelium
Contraindications:
• soft lenses
Preop:
• consider orbital massage or osmotic agents to reduce vitreous pressure
Anesthesia:
• retrobulbar and lid block
• subtenon’s block
• rarely general anesthesia, e.g. claustrophobia, dementia, tremor
Procedure:
1. Superior bridle suture (if incision is superior).
2. Peritomy of about 170 degrees.
3. Make frown shaped incision to reduce astigmatism.
28
4. Center of frown incision is 1–2 mm posterior to limbus.
5. Frown incision is 6–7 mm wide.
6. Incision dissection is carried into cornea widely to about 10 mm width.
7. Instill viscoelastic.
8. Initial entry into anterior chamber to allow capsulotomy with keratome.
9. Remove anterior capsule (large CCC, can opener, other).
10. Mobilize lens (physically with cystitome or with hydrodissection with CCC).
11. Lens removed w/ irrigating lens loop under lens.
12. Allow fluid pressure too push lens out of eye.
13. Cortical material is removed using I/A device (either automated or manual).
14. Instill ophthalmic viscoelastic device (OVD).
15. Lens is placed in the posterior chamber.
16. Wound may seal or use a couple of 10-0 nylon sutures.
17. OVD is removed.
18. Reapproximate the conjunctiva.
29
6. Phacoemulsification – Step by Step
Please read:
• Paul S. Koch. Simplifying Phacoemulsification: Safe and Efficient Methods for Cataract
Surgery. 5th edition. Slack Inc, 1997.
• Bonnie An Henderson. Essentials of Cataract Surgery. 1st edition. Thorofare NJ, Slack
Inc, 2007.
• David F. Chang. Phaco Chop: Mastering Techniques, Optimizing Technology, and
Avoiding Complications. 1st edition. Slack Inc, 2005.
• David F. Chang, Terry Kim, Thomas A. Oetting. Curbside Consultation in Cataract
Surgery. 1st edition. Thorofare NJ, Slack Inc, 2007.
Indications:
• almost all cataract surgery
Contraindications:
• few, maybe: almost no zonular support or extremely hard lens
Anesthesia:
• topical +/− intracameral non preserved lidocaine
• retrobulbar and lid block
• subtenon’s block
• rarely general anesthesia, e.g. claustrophobia, dementia, tremor
30
Potential What to do about it?
complications
drive needle into
Delay case and cryo/laser area.
vitreous
6.1. Paracentesis
31
6.2. Place Ophthalmic Viscoelastic Device (OVD)
Arshinoff soft shell technique: 1st dispersive (e.g. Viscoat® ), then cohesive (e.g. Healon® ).
Allows dispersive to coat corneal endothelium to protect from U/S energy.
Allows cohesive to maintain chamber during the first part of procedure.
Or use just one. Healon® is cheapest at our VA.
6.3. Wound
32
Limbal incision:
• peritomy of 4–7 mm depending on IOL size
• cauterize sclera
• 1⁄2 depth groove into limbus with crescent blade or 64 beaver
• enter eye with keratome (sized for phaco needle)
Scleral tunnel:
• peritomy of 4–7 mm depending on IOL size
• cauterize sclera
• 1⁄2 depth groove into sclera with crescent blade
• tunnel at 1⁄2 depth through sclera into cornea with crescent blade
• enter eye with keratome (sized for phaco needle)
Corneal incision:
• 1⁄3 depth tunnel into cornea with keratome
• enter eye with keratome
33
Potential What to do about it?
complications
6.4. Capsulorhexis
Goal is CCC with a central circular opening slightly smaller than the optic diameter.
Three basic techniques for CCC (best way to learn about this is to watch video):
34
• cystitome - initial cut and control of tear with cystitome (best with cohesive OVD)
• combo – initial cut w/ cystitome, most of tear w/ forceps (most common technique)
• forceps – sharp forceps cut and then grab capsule to complete tear
• femtosecond laser – cheating
6.5. Hydrodissection
35
May prolapse lens with a large capsulorhexis – not always bad.
6.6. Phacoemulsification
Goal is to remove lens with the minimum ultrasound (U/S) damage to the cornea.
Trend is to use increasing vacuum and decreasing U/S power to remove nucleus.
Energy can be torsional (Alcon, AMO) or longitudinal (standard U/S).
36
• supracapsular – prolapsing nucleus into sulcus during phaco
• AC shell - prolapsing shelled out nucleus into AC
• 1⁄2 bag 1⁄2 AC – tipping nucleus on side 1⁄2 in bag, 1⁄2 in AC – Brown, Pop’n’Chop
Fragmentation
Advantages Disadvantages
style
slow,
sculpt and
can do with one hand energy close to cornea,
prolapse
lots of U/S power
classic,
divide and easy to do,
lots of U/S power
conquer energy away from cornea,
can do with one hand
does not require
hydrodissection,
slow,
V groove does not require rotation,
lots of U/S power
useful with known capsule
damage
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fairly easy to do,
stop’n’chop needs two hands
less U/S power
little stress on bag, hard to do,
least U/S power, needs two hands,
chop
fast, hit ant capsule with chopper,
easy on zonules jigsaw problem
38
6.7. Cortical Aspiration
Aspiration to grab and peel the cortex off the capsule, not suck it off the capsule.
Dangerous procedure – common time for vitreous loss in experienced surgeon.
Subincisional removal is most difficult, esp with small rhexis.
Adequate hydrodissection makes this step easier.
39
Wound may need to be extended to allow placement of the lens
PMMA (doesn’t fold) IOL needs slightly more than optic size.
Old school now – forceps loaded IOLs need 3.5 mm or so to insert.
Most injected IOLs don’t need extension from incision for phaco needle.
A bit bigger, well constructed wound seals better than stretched small wound.
PMMA IOL:
1. grasp IOL and trailing haptic with forceps (e.g. Kelman-McPherson)
2. place leading haptic into bag, optic into AC, release forceps
3. place optic into bag
4. place trailing haptic into bag with hook or forceps
Folded IOL:
• folded and placed in special forceps
• incision size grows a bit with increased power of IOL – 3.5 mm range
• moustache style fold: wider incision, but haptics flow into bag (great when suturing IOL
to the iris with no capsule)
• axial style fold: smaller incision, but haptics need guidance
Scheme 2. IOL implantation with forceps: A) moustache style fold, B) axial style fold
Injected IOL:
• most common – many different systems
• single piece acrylic (SA60AT, SN60WF) and plate IOL – most simple
• 3-piece IOL requires some haptic care and manipulation
• be careful of Descemet’s membrane w/ IOL insertion (especially w/ injectors)
40
Scheme 3. IOL implantation with injector: A) toe up on injector can tear Descemet’s membrane, B)
toe down slips under Descemet’s membrane
Scheme 4. A) IOL with correct side up (top haptic looks like a 7, optic looks like an O, bottom
haptic looks like an L), B) IOL is designed for right handed surgeon to easily rotate, C) when upside
down the IOL looks like an S, so Stop
Upside down angulated 3-piece IOL creates myopic shift w/ anterior IOL shift.
41
Scheme 6. IOL in capsular bag: A) centered, B) decentered, 1⁄2 in bag
42
Potential What to do about it?
complications
Can leave as is – accept myopic shift, or take one
haptic out of wound with hook:
place IOL upside down 1. Fill with OVD above and below IOL.
2. One hook above and one below – flip IOL.
43
6.10. OVD is Removed with I/A Device
6.12. Other
44
7. Phaco Machine Settings Primer
Look over the classic definitive text: Barry Seibel. Phacodynamics: Mastering the Tools and
Techniques of Phacoemulsification Surgery. 4th edition. Slack Inc, 2005.
Flow rate – amount of fluid passing through the tubing (cc/min), also aspiration flow rate.
Vacuum – difference in fluid pressure in two points, e.g. tip of needle and AC (mmHg).
E.g. Venturi pump (Stellaris, Accurus), diaphragm. Increasing pump power increases vacuum
directly, flow rate indirectly.
Venturi pump requires external source of compressed air or compressor.
This has limited acceptance of this pump (ASC may not have air lines).
45
Compressed gas flows over open top of rigid cassette attached to tubing.
Flow of gas creates vacuum much as flow over airplane wing creates lift.
Flow rate is a function of vacuum and resistance of flow and not directly set.
Roughly analogous to electric current voltage relationship (Ohm’s law):
Pump settings:
• no settings for flow rate, only vacuum
• fixed:
– no matter how deep you are in position 2 or 3, vacuum is fixed
– great for chopping and quadrant removal
• variable:
– vacuum increases from 0 to max as you push on the pedal
– great for I/A (can slowly increase vacuum to just what you need)
Pump settings:
• set vacuum cutoff and flow rate
• vacuum cut off:
– seems like you are setting the vacuum
– really setting the vacuum at which the pump stops
– increasing the vacuum does not increase pump speed
– flow rate or aspiration flow rate (AFR) sets pump speed cc/min
• w/ modern peristaltic pumps (e.g. Infiniti) for each foot pos you can have :
– fixed or variable flow
46
– fixed or variable vacuum cut off
Flow rate Vacuum cut off Comment/Application
Table 32. Application of different flow rate and vacuum cut off settings
Continuous:
• phaco is on in position three
• usually increasing U/S power with depth into foot position 3
Pulse:
• phaco pulses with duty cycle on and off
• usually with equal on and off time or 50% duty cycle (time on/cycle time)
• usually the rate is fixed (Hz)
• usually increasing U/S power with depth into foot position
Burst:
• bursts of power come with off time that decreases with depth into foot position
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• usually when floored in position 3 – U/S power becomes continuous
• U/S power is fixed
Hyperpulse:
• uses short on time pulses e.g. 25% on; 75% off
• fixed duty cycle; fixed pulse rate; usually high frequency like 200 Hz
• usually increasing U/S power with depth into foot position 3
Torsional:
• OZil® uses rotary motion of angled (Kelman type) tip rather than longitudinal U/S power
• AMO oscillatory system does not require Kelman tip
• can use supplemental longitudinal to clear and prevent occlusion
48
I/A 500 n/a n/a default settings
Alcon Legacy Vacuum Flow
Type U/S Comment
20 000 (mmHg) (cc/min)
sculpt 50 22 continuous
49
8. Ophthalmic Viscoelastic Devices (OVD)
8.1. Indications
Table 36. Differences between cohesive and dispersive OVDs during different surgical steps
50
8.2. Removal
Properties of dispersive OVD at high shear rate (e.g. during phaco) and of cohesive OVD at low
shear rate (e.g. during IOL placement) – e.g. Healon® 5.
Very long fragile chain molecules that break with flow rate.
Difficult to remove.
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Scheme 8. Arshinoff Shell technique
52
9. Capsular Staining
The white cataract used to be the most feared cataract surgery to perform.
Capsular staining has changed these cases from complex to routine.
Capsular stains (ICG and trypan blue) are useful whenever capsule is hard to see:
• classic white cataract
• traumatic cataract with possible anterior capsular tear
• dark red or brown cataract with limited red reflex
• started rhexis and then loose capsule in an area of dense lens
• useful for delineating anterior capsular trauma
VisionBlue® – premixed and approved by the FDA (2005) making it cheaper, better, and faster
than ICG.
Reference: Horiguchi M et al. Staining of the lens capsule for circular continuous capsulorrhexis
in eyes with white cataract. Arch Ophthalmol. 1998;116:535–7. ICG is used to stain the lens
capsule, but is rarely used with trypan blue available now.
Stain may be harmful to the retina, so use the least amount possible.
Preparation:
• draw up 0.5 cc of aqueous solvent (comes with ICG) into syringe
• place aqueous solvent into vial of 25 mg ICG and shake
• draw up 4.5 cc of BSS into syringe
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• place BSS (original article was BSS+, but BSS is OK) into ICG vial and shake some more
• Osm 270 (plasma 285) with a concentration 0.5%
54
10. Routine Postoperative Care
10.1. Phacoemulsification
Day 1
55
Week 1
Week 2–4
Day 1
56
• look for K abrasion, esp if patched
IOP:
• < 8 mmHg: look hard for leak with Seidel test
• 9 – 29 mmHg: probably OK
• > 30 mmHg: start with Cosopt, Alphagan, recheck in 45 min
• > 40 mmHg: suppress aqueous and bleed until pressure is stable (< 30), consider seeing
the next day
• lower these guidelines in patients with h/o DM, AION, etc.
Fundus:
• usually can see fundus w/o dilation
• document no RD or choroidal effusion/hemorrhage
Plan:
• floroquinolone 1 gtt qid
• prednisolone acetate 1 gtt qid
• cyclogyl 1% bid
F/u:
• one week later usually
• next day with wound leak, big corneal abrasion, etc.
Give a postoperative instruction sheet.
Week 1
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Week 6
After that
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11. Principles of Anterior Vitrectomy
We will cover the causes and signs of vitreous prolapse and the principles of anterior vitrectomy
in various situations.1-3 This handout is modified from my blog1 which also includes video and
may be of some interest to those learning about anterior vitrectomy.
Causes of vitreous prolapse. The vitreous either comes around the zonules or through a
tear in the posterior capsule. Posterior capsular tears are caused commonly by: anterior tear
extending posteriorly (most common), posterior tear secondary to phaco needle being too deep,
a chopper or from the I/A instrument, or a pre-existing injury (e.g. posterior polar cataract,
iatrogenic from PPVx, or from penetrating lens trauma). Zonular problems are often pre-existing
such as from trauma, PXF, or Marfan’s but can also be iatrogenic from forceful rotation of the
lens or pulling on the capsule during I/A.
Signs of vitreous prolapse. The first sign of vitreous prolapse is denial. Something seems
wrong, but you can’t quite pin point the issue. At first you deny that an issue exists, but soon
it becomes clear. Less mysterious signs of vitreous prolapse include: the chamber deepens, the
pupil widens, lens material no longer centered, particles no longer come to phaco or I/A, and
the lens no longer rotates freely. When you suspect vitreous prolapse, you should keep the
chamber formed by placing dispersive OVD into the eye before removing the phaco needle or
I/A from the eye and can check the wound with a WECK-CEL® sponge for vitreous.
The second step is to separate the irrigation device from the aspiration/cutting device (this is
standard on modern phaco machines). In general, you will want to place the vitreous cutter
low (at the level of the posterior capsule) while holding the irrigating cannula high (anterior
chamber) which allows you to create a pressure differential such that the vitreous is encouraged
to move posteriorly toward the aspiration/cutter and away from the anterior chamber.
Scheme 9. Anterior vitrectomy: A) close the chamber, B) separate irrigation and cutter, C) cut low,
irrigate high
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In general, the bottle height should be low – just high enough to keep the AC formed and not
so high that fluid is forced out around the instruments which can bring vitreous with it. The
smaller the bore of the infusion cannula, the higher the bottle height will need to be. The
higher the vacuum, the higher the bottle height will need to be. The cutting rate should be as
high as possible when cutting vitreous and low when cutting cortical lens material or removing
viscoelastic. We will separately discuss early, mid, and late case vitreous loss below.
This is the worst time for vitreous to prolapse. The strategy will depend on how the vitreous
presented. If the vitreous has come from a strike in the posterior capsule while grooving or
from a radial tear which has gone posterior with almost all of the lens remaining, then one
should strongly consider converting to ECCE. If the pieces are smaller, then another option
is to sequester the residual nuclear material with viscoelastic or an IOL scaffold,4 perform the
anterior vitrectomy, and continue with slow motion phacoemulsification.5 If the vitreous has
come from loose zonules, then the solution may be better support of the capsule with a capsular
tension ring (CTR), a capsular tension segment (CTS), or capsular hooks.
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to the equator and place 2 cc non-preserved lidocaine behind the eye.
2. Use only viscous dispersive viscoelastic (e.g. Viscoat® ) from this point forward as it causes
less ocular hypertension and sticks to other structures.
3. Use viscoelastic to lift lens material into the anterior chamber.
4. If the material heads south, don’t chase it and leave it for the vitreous surgeons.
5. Try to wedge the residual lens material into a safe position anterior to the iris and away
from the posterior capsular tear.
6. Make separate 1.5 mm incision for anterior vitrectomy.
7. Separate irrigation (through paracentesis) and asp/cutter (through larger paracentesis).
8. May need to suture original wound to keep chamber formed.
9. Irrigate away from the sequestered material and cut/suck in the area of the posterior
tear/hole.
10. Try to get some of the residual cortical material with the anterior vitrector or with removal
of residual cortical material with syringe on 27 gauge cannula or 23 gauge Visitec cannula
with viscoelastic to support chamber (dry technique).
11. Consider staining with Kenalog (see below).
12. Replace viscoelastic often to keep residual material sequestered.
13. Consider placing a 3-piece IOL in the anterior chamber or sulcus and below the residual
material as a scaffold for residual material to prevent the material from dropping posterior.5
14. Now, with anterior chamber free of vitreous and lens material sequestered from tear with
IOL scaffold or viscoelastic, use slow motion phaco to remove (low bottle height, low
vacuum).
15. If pieces are small, you can use the Malyugin ring inserted to grab nuclear bits (Neuzil
technique).
Vitreous presenting early due to zonulopathy:
1. If topical, consider subtenon’s injection. Incise conjunctiva in a quadrant and dissect
posteriorly to the equator and place 2 cc non-preserved lidocaine behind the eye.
2. Use triamcinolone stain to identify vitreous and area of weak zonules (see below).
3. Trim with anterior vitrector under viscoelastic with anterior approach or consider pars
plana approach (if comfortable with this technique).
4. Sideways Arshinoff shell to force dispersive viscoelastic into area of weak zonules. First
place dispersive in area of weak zonules, then place cohesive across from weak area forcing
dispersive into area of weak zonules sealing it off.
5. Perform CCC if not already done.
6. Use cohesive viscodissection between capsule and the cortical material to allow space for
CTR or CTS.
7. Place CTR with lead eyelet of the ring heading out of inserter toward the area of weak
zonules to minimize stress of insertion.
8. Use hooks or a sutured CTS to support the CTR if needed during the rest of the case.
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Figure 8. Capsular hooks: A) hooking of the capsule, B) hooks keep lens in place during I/A
This seems to be the most common time for vitreous loss. Often one will get the posterior
capsule just as the last nuclear fragment is taken. Of course there is no reason to convert to
ECCE in this case. The following steps are usefull:1
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this setting (ring has ripples to ease cortex removal following placement).
8. Place a 3-piece IOL with haptics toward weak area if small defect, place CTR if less than
4 clock hours, or place sutured CTS if 4–7 clock hours.
9. Miochol to bring pupil down.
This is the least problematic and least common time to loose vitreous. The main issue is to
make sure the IOL is stable while attending to the vitreous and then to secure a proper IOL in
either the AC, sulcus, or bag.1 The strategy will depend on whether the vitreous is presenting
through a later tear or zonular weakness (more common).
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11.4. Staining the Vitreous with Triamcinolone
Scott Burk at Cincinnati Eye described using triamcinolone (not approved by the FDA for this
indication) to stain vitreous to better visualize vitreous prolapsed into the anterior chamber.8
As triamcinolone is not approved by the FDA for this indication and as some retinal surgeons
have had sterile and even infectious endophthalmitis with triamcinolone injection, its use is
controversial. However, I find it to be a very useful adjunct to anterior vitrectomy. You can
simply dilute the nonpreserved triamcinolone (FDA approved for posterior segment inflammation)
1:10 (e.g. Triesence).
An alternative to the more expensive non preserved triamcinolone is to wash the preservative off
the triamcinolone using a filter as described by Burk and then dilute 1:10 as described below:
1. TB syringe to withdraw 0.2 ml of well shaken triamcinoloone 40 mg/ml.
2. Remove the needle and replace with a 5 (or 22) micron syringe filter (e.g. Sherwood
Medical).
3. Depress the syringe so the large triamcinolone molecules will be stopped by the filter while
the preservative and solvent will pass through the filter.
4. The triamcinolone will be trapped on the syringe side of the filter.
5. Transfer the filter to a 5 ml syringe filled with balanced salt solution (BSS).
6. Gently force the BSS through the filter to further rinse off preservative.
7. Repeat rinsing a few times.
8. Place a 22 gauge needle on the distal end of the filter.
9. Draw 2 ml of BSS into the syringe through the filter to resuspend the Kenalog.
10. The Kenalog (now without preservative and diluted 1:10) will stain vitreous white.
11.5. References
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4. Kumar DA, Agarwal A, Prakash G, Jacob S, Agarwal A, Sivagnanam S. IOL scaffold
technique for posterior capsule rupture. J Refract Surg. 2012;28(5):314–5.
5. Osher RH. Slow motion phacoemulsification approach. J Cataract Refract Surg. 1993;19(5):667.
6. Chang DF, Masket S, Miller KM, Braga-Mele R, Little BC, Mamalis N, Oetting TA,
Packer M. ASCRS Cataract Clinical Committee. Complications of sulcus placement of
single-piece acrylic intraocular lenses: recommendations for backup IOL implantation
following posterior capsule rupture. J Cataract Refract Surg. 2009;35(8):1445–58.
7. Jones JJ, Oetting TA, Rogers GM, Jin GJ. Reverse Optic Capture of the Single-Piece
Acrylic Intraocular Lens in Eyes With Posterior Capsule Rupture. Ophthalmic Surg Lasers
Imaging. 2012;6:1–9.
8. Burk SE, Da Mata AP, Snyder ME, Schneider S, Osher RH, Cionni RJ. Visualizing
vitreous using Kenalog suspension. J Cataract Refract Surg. 2003;29(4):645–51.
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12. Conversion to ECCE
Conversion to ECCE often comes at a difficult time. The lens is about to fall south, the
vitreous has prolapsed and the surgeon is stressed. Understanding the steps and process of
conversion to ECCE is essential and study before the crisis will help soothe the stress when
this inevitable process occurs. We will cover several areas: identifying patients at risk for the
need for conversion to ECCE, indications for conversion, conversion from topical to subtenon’s,
wound preparation, expressing the lens material, closure of the wound, placement of the IOL,
postoperative issues and a brief section on anterior vitrectomy.
One of the most important parts of the preoperative process for cataract patients is to assess
the difficulty factors (see section on difficulty factors) that may lead to conversion to ECCE
or otherwise complicate the procedure. You may want to add operative time to your schedule
or ask for additional equipment. You may want to change to a superior limbal wound which
facilitates conversion to an ECCE rather than a temporal clear corneal incision. You may want
to do a retrobulbar block rather than topical anesthesia as the case may last longer or is more
likely to become complicated. Or, you may want someone more experienced to do the case.
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with continued phacoemulsification. Rarely now with trypan blue dye, a surgeon will choose to
convert to ECCE when the anterior capsule is hard to see and capsulorhexis must be completed
with the can opener technique. More often the conversion is indicated when the crystalline lens
is loose from weak zonules or a posterior capsule tear which make phacoemulsification less safe
than extending the wound and removing the residual lens material. Indications for conversion
to ECCE include:
• hard crystalline lens or unstable endothelium
• radial tear in anterior capsule with hard lens
• poor visualization despite trypan dye
• posterior capsular tear
• zonular dialysis
Often we convert cases from topical clear corneal to ECCE. While the ECCE can be done
under topical, it is usually more comfortable and safer to give additional anesthetic which is
typically a subtenon’s injection of bupivacaine and lidocaine. This will provide some akinesia
and additional anesthesia. There is usually subconjunctival hemorrhage and if the injection is
made too anterior, it can cause chemosis and ballooning of the conjunctiva. The steps of the
subtenon’s injection are outlined in the anesthesia section above.
The major step toward converting to ECCE is to either extend the existing wound or close and
make another. The ECCE will require a large incision of from 9–12 mm which is closed with
suture. The decision to extend the existing wound or make a new wound hinges on several
factors: location of the original wound, size of the brow, past surgical history, and possible need
for future surgery.
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Already have sup incision.
superior None.
No need to change position.
Table 37. Advantages of making new or extending existing wound for ECCE
Making a new incision during conversion is identical to that for a planned ECCE. The
original incision is closed with a 10-0 nylon suture. The surgeon and microscope are rotated as
the surgeon should sit superior. The steps to make a new superior incision are:
• conjunctival peritomy of about 170 degrees
• use 64 or crescent blade to make limbal groove with a chord length of 11 mm
• bipolar cautery for hemostasis
• use keratome to make initial incision starting in groove into AC
• extend initial incision to full length of groove (with scissors or knife)
• safety sutures are preplaced, usually 7–0 vicryl
Extending an existing incision can be tricky and the technique is different for scleral tunnels
compared to clear corneal incisions. However, in both cases the original extension is brought to
the limbus. In the case of an original scleral incision, the incision is brought anterior to join
the limbus on either end before extending along the limbus for a chord length of about 11 mm.
In the case of an existing corneal incision, the corneal incision is brought posterior toward the
limbus before extending the wound along the limbus for a chord length of about 11 mm. When
iris hooks are being used in a diamond configuration, the wound can be extended to preserve
the subincisional hook and the large pupil.2
• conjunctival peritomy of about 170 degrees
• use 64 or crescent blade on either side of the existing wound to make a limbal groove with
a chord length of 11 mm
• bipolar cautery for hemostasis
• use crescent to bring existing scleral wound anterior or existing corneal wound posterior
to join limbus
• extend initial incision to full length of groove (with scissors or knife)
• safety sutures are preplaced, usually 7–0 vicryl
One has to be far more careful when removing the nucleus during the typical conversion to
ECCE which comes along with vitreous loss. First, the anterior capsule must be large enough
to allow the nucleus to express which may require relaxing incisions in some cases. When the
zonules are weak or the posterior capsule is torn, the lens cannot be expressed with fluid or
external pressure as is often done with a planned ECCE with intact capsule/zonules. After any
vitreous is removed (see below), the lens must be carefully looped out of the anterior chamber
with minimal pressure on the globe. If the posterior capsule and zonules are intact, than the
lens can be expressed as described with a planned ECCE.
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12.5.1. Removing Lens with Intact Capsule Complex
• mobilize lens with Viscoat® cannula – tip lens so that wound side is anterior
• slip lens loop under lens, toe up, remove lens
• wound is closed with safety sutures and additional central vicryl suture
• anterior vitrectomy (see below)
• cortical material is removed using dry technique or anterior vitrector
• instill ophthalmic viscoelastic device (OVD)
• lens is placed in the sulcus or in the anterior chamber
• wound is closed with 10-0 nylon and vicryl sutures are removed
• OVD is removed
IOL selection with ECCE conversion depends on the residual capsular complex. The key to
IOL centration is to get both of the haptics in the same place: either both in the bag or both in
the sulcus.
When the posterior capsule is intact following a conversion to ECCE, the anterior capsular
opening is usually poorly defined which can make bag placement difficult. If the anterior capsule
and thus the bag is well defined, then place a single-piece acrylic IOL without folding it directly
and gently into the bag using Kelman forceps.
When the posterior capsule is intact and the anterior capsule is poorly defined, then place a
3-piece IOL in the sulcus, such as a large silicone IOL or the MA50 acrylic, by placing these
directly and unfolded into the sulcus with Kelman forceps. Make sure that both haptics are in
the sulcus.
When the posterior capsule is damaged, if enough anterior capsule and posterior capsule is left
to support the IOL, define the sulcus with Viscoat® and place the IOL directly in the sulcus.
Make sure both haptics are in the sulcus. If the IOL does not seem stable, then place McCannel
sutures to secure the IOL to the iris or remove and replace with an AC IOL (don’t forget to
place a PI with vitrector).
When the capsule is severely damaged and cannot support an IOL, then place the IOL in the
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anterior chamber. Use Kelman forceps to place the IOL, then secure the chamber and use
a Sinskey hook to place the AC IOL into its final position (don’t forget to place a PI with
vitrector).
Postoperative care for patients following conversion from phaco to ECCE is a bit more com-
plicated and focuses on preventing cystoid macular edema and limiting induced astigmatism.
Often, the care is very similar to that of a planned ECCE with about 3 postoperative visits:
one the same day or next, one a week later, and one about 5–6 weeks later. Depending on the
amount of astigmatism, the patient may require several visits to sequentially remove sutures
while eliminating induced astigmatism.
Often on the same afternoon 4–6 hours following surgery or next morning with the primary
emphasis to check the IOP, look for wound leaks and scan for residual lens material or vitreous
in the anterior chamber. Most wound leaks should be sutured, but if the AC is not formed,
closing these is mandatory. Residual nuclear material should be removed in the next few days if
present, but residual cortical material will often dissolve away with little inflammation. You
would expect poor vision in the 20/200 range due to astigmatism and edema. The anterior
chamber should be formed and typically has moderate cell (10–20 cells/hpf with 0.2 mm beam).
If the IOP is less than 10, search hard for a leak using Seidel testing. If the IOP is in the 10–29
range, all is probably OK unless the patient is a vasculopath and then the upper limit of IOP
tolerance should be lowered. If the IOP is in the 30–39 range, consider aqueous suppression.
If the IOP is > 40, than consider aqueous suppression and bleeding down the IOP with the
paracentesis or anterior chamber tap. The IOP should be rechecked 60–90 minutes later to
ensure success with your treatment. Look at the fundus and rule out retinal detachment and
choroidal effusion or hemorrhage. Typically, patients are placed on prednisolone acetate 1% 1
drop 4 times a day, cyclogyl 1% 1 drop 2 times a day, and an antibiotic 1 drop 4 times a day for
the next week.
The vision and pressure should dramatically improve in patients over the next week where you
have converted to ECCE. The vision should be in the 20/100 range with an improvement with
pin hole to 20/50. The vision is usually limited by residual edema and astigmatism. In a study
of our ECCE we found about 7 diopters of cylinder at the one week visit. You should expect
very little inflammation and document that no RD exists. Search for residual lens material in
the anterior segment and posterior pole. You can discontinue the cyclogyl and the antibiotic.
Slowly taper the prednisolone acetate like 1 gtt qid for 7 more days, then 1 gtt tid for 7 days,
then 1 gtt bid for 7 days, then 1 gtt qd for 7 days, then discontinue. If the patient is at risk for
CME (e.g. vitreous loss), then keep on prednisolone qid and start a non steroidal like acular 1
gtt qid until the next visit 4–6 weeks later.
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12.7.3. Week 5 Postoperative Visit
The vision should continue to improve as the astigmatism settles and the cornea clears further.
The eye should be comfortable. The vision should be in the 20/80 range with an improvement
to 20/40 with pin hole. In our study the astigmatism induced by ECCE sutures was about 5.0
diopters at the incision. The anterior segment should be quiet and the IOP normal (unless the
patient is a steroid responder). Consider CME as a possibility in patients where conversion was
required as these cases are often long and can involve vitreous loss (with OCT, FFA, or clinical
exam).
But the main issue is astigmatic control with suture removal. Use keratometry, refraction, streak
retinoscopy, or topography to guide in suture removal. If the keratometry is 45.00 at 90, and
40.00 at 180 then look for tight sutures at around 90 degrees (12 o’clock) that are causing 5
diopters of cylinder. You can take only one suture at 5 weeks, then can take maybe 2 at a
time by 8 weeks. The plan is to remove a suture and see how the cornea settles. When the
astigmatism is less than about 1.0 to 1.5 diopters you should stop. Use antibiotic drops for a
few days after suture removal. After this visit, you should consider the following choices with
each visit (don’t waste too much time thinking about other possibilities and remember not
everybody is going to be 20/20):
1. pull a stitch (i.e. cyl at axis of stitch is greater than 1 on MR),
2. give glasses (i.e. no stitch to pull or cylinder is less than 1 on MR),
3. get FFA or OCT because you suspect CME.
12.8. References
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13. Approaching Different Kinds of Cataract
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13.2. Intumescent Cortical Cataract
Etiology:
• opacification of the cortical lens fibers
• swelling of the lens material creates intumescent cataract
Clinical features:
• initially vacuoles and water left in the lens cortex
• wedge shaped opacities or cortical spokes
• progresses to form white intumescent cortical cataract
• risk of phacolytic glaucoma
Risk factors:
• smoking
• ultraviolet light exposure
• diabetes mellitus
• poor nutrition
• trauma
Phaco/ECCE:
• capsular staining techniques
• capsulorhexis techniques
– initial small tear
– removal of liquid cortical material to relieve capsular tension
– liberal use of viscoelastic material
Complications of surgery:
• increased risk of capsular radial tear
• increased risk of vitreous loss
• increased risk of loss of lens material into vitreous
Etiology:
• opacification of the cortical lens fibers
• swelling of the lens material creates intumescent cataract
• degenerated cortical material leaks through capsule leaving wrinkled capsule
Pertinent clinical features:
• wrinkled anterior capsule
• increased anterior chamber flare
• calcium deposits in lens
• white cortical material
• risk of phacolytic glaucoma
Phaco/ECCE:
• capsular staining with trypan blue
• capsulorhexis techniques
– initial small tear
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– removal of liquid cortical material
– use of viscoelastic material in anterior chamber and bag
Complications of surgery:
• increased risk of capsular radial tear
• increased risk of vitreous loss
• increased risk of zonular dialysis
• increased risk of loss of lens into the vitreous
Etiology:
• opacification of the cortical lens fibers
• can be swelling of the lens material as in intumescent cataract
• can be wrinkled capsule as in hypermature cataract
• hallmark – liquified cortex allows nucleus to move freely in bag
Pertinent clinical features:
• wrinkled anterior capsule
• increased anterior chamber flare
• dense brown nucleus freely moving in capsular bag
• calcium deposits within the lens
Phaco/ECCE:
• capsular staining techniques
• capsulorhexis techniques
– initial small tear
– removal of liquid cortical material
– use of viscoelastics material in anterior chamber and bag
• stabilize nucleus with viscoelastic
Complications of surgery:
• increased risk of capsular radial tear
• increased risk of vitreous loss
• increased risk of zonular dialysis
• increased risk of loss of lens into the vitreous
Etiology:
• opacity of the anterior subcapsular cortex and capsular
• bilateral
• non progressive usually
• frequently autosomal dominant
Clinical features:
• usually asymptomatic – good vision
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• central opacity involving the anterior capsule
• associated with microphthalmos, persistent pupillary membrane, anterior lenticonus
• differential diagnosis includes penetrating capsule trauma
Phaco/ECCE – w/ capsulorhexis start away from polar cataract, make bigger and go around
polar cataract if possible.
Etiology:
• opacity of the posterior capsular cortex and capsule
• familial, autosomal dominant, bilateral; sporadic, unilateral
• slowly progressive
Pertinent clinical features:
• good vision, but at nodal point, more symptomatic than anterior polar
• central opacity involving the posterior capsule
• glare
• differential diagnosis includes:
– posterior subcapsular cataract
– penetrating capsule trauma
– Mittendorf dot
Phaco/ECCE:
• no hydrodissection
• sculpt out a bowl to relieve capsular tension or use the V groove technique
• gentle hydrodelineation and slow careful viscodissection
• leave central opacity or take at the end of surgery
Complications:
• increased risk of posterior capsular tear
• increased risk of vitreous loss
• increased risk of loss of lens material into vitreous
Etiology:
• penetrating injury results in cortical opacification at site
• rarely can seal resulting in a focal opacity
• usually progresses to complete opacification
Pertinent clinical features:
• focal cortical cataract
• white cataract with capsular irregularity/scar
• full thickness corneal scar
Laboratory testing:
• B-scan ultrasound – posterior capsule intact? Intraocular foreign body?
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• CT scan to rule out intraocular foreign body
Phaco/ECCE:
• capsular staining to identify traumatic tear
• treat similar to posterior polar cataract:
– no hydrodissection if posterior penetration suspected
– consider use of viscodissection and hydrodelineation
• usually can aspirate in younger patients without need for nucleofractis
Complications:
• increased risk of anterior radial capsular tear
• increased risk of vitreous loss
• increased risk of lens material in vitreous
• increased risk of retinal detachment
Etiology:
• increased aqueous glucose concentration drives glucose into lens
• glucose converted into sorbitol that is not metabolized by lens
• sorbitol creates an osmolar gradient forcing hydration of the lens
• this sorbitol induced lenticular hydration:
– decreases accommodation
– changes the refractive power of the lens
– generates cataract
Pertinent clinical features:
• snowflake or true diabetic cataract:
– bilateral
– posterior and anterior subcapsular, cortical vacuoles and clefts
• typical nuclear, cortical or posterior subcapsular cataracts
Phaco/ECCE:
• indicated when view of posterior pole is poor
• standard technique
• consider monofocal acrylic IOL with any retinopathy
Complications:
• exacerbation of diabetic macular edema:
– focal or grid laser therapy prior to surgery if indicated/possible
– anti-VEGF agents commonly used prior to surgery
– sutured wound to allow early laser therapy if indicated
• increased risk of cystoid macular edema:
– pretreatment with steroid and non-steroidal drops
– prophylactic treatment for 1–3 months with steroid and/or non-steroidal drops
• can present with rapid white cataract under tension:
– will need trypan blue (or ICG, but this is not approved by the US FDA)
– be careful with initial capsule tear as it is prone to go radial (Argentinean flag sing)
– make an initial tear, remove anterior cortical material, then add more OVD
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– consider very viscous OVD like Healon® 5 or Healon® GV
Etiology:
• posterior subcapsular cataract:
– initially an iridescent sheen appears in the posterior cortex
– followed by granular and plaque like opacities
• may progress to or involve anterior subcapsular cortical fibres
• may present as cortical cataract without posterior subcapsular component
• associated with uveitis and corticosteroids to treat uveitis
• may progress rapidly to a mature cataract
Pertinent clinical features:
• central opacity of the posterior cortical fibers
• cortical cataract
• posterior synechiae
• pupillary membrane
• anterior chamber cell or flare
Prior to phaco/ECCE:
• several months without inflammation
• 1 week prior to surgery suppress immune system
– topical agents in those patients who are typically quiet with topical agents alone
– oral prednisone in those that typically require oral steroid with a flare
– consider intraoperative IV steroids
Phaco/ECCE:
• synechiolysis with viscoelastic agents/hooks
• may require iris hooks to stabilize floppy iris and control pupillary aperture
• capsular dye to allow continuous tear
• IOL material: acrylic, heparin coated PMMA better than silicone
• consider aphakia in children with JRA
Complications:
• increased risk of postoperative inflammation
• increased risk of postoperative pressure spike
• increased risk of cystoid macular edema
• consider using steroid and non-steroidal drops for months following surgery
Etiology:
• systemic disease in which a fibrillar material is deposited in the eye:
– similar material to the basement membrane proteoglycan
– the material is found throughout the body
• within the eye the fibrillar material comes from the lens capsule, iris, and ciliary body
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• the zonules are weak in this condition
• often asymmetric or even unilateral
• glaucoma develops when the fibrillar material blocks the trabecular meshwork
Epidemiology:
• patients tend to be over 60 years of age
• geographic clustering suggests a hereditary pattern (in Scandinavia for example, pseudoex-
foliation causes 75% of glaucoma)
• glaucoma develops in 22–82% of patients with exfoliative material
• increased incidence of age related cataract
Pertinent clinical features:
• ground glass appearing deposition of fibrillar material on anterior lens capsule:
– iris may sweep material into rings on the lens capsule
– best viewed with dilation
• transillumination defect and fibrillar material at the pupillary margin
• open angle with brown clumps of fibrillar material on trabecular meshwork
• flakes of fibrillar material on corneal endothelium
• evidence of zonular weakness:
– phaco- or iridodonesis
– lens subluxation or even luxation
Phaco/ECCE:
• use of iris hooks for capsular support during phacoemulsification
• use of capsular tension ring with or without Cionni modification
• placement of AC IOL, sutured Cionni ring with capsular IOL, sutured PC IOL
• sutured iris IOL
• consider surgery sooner, while zonules are relatively strong
• minimize zonule stress during surgery
Complications of phaco/ECCE:
• increased risk of capsular radial tear
• increased risk of zonular dialysis
• increased risk of loss of lens material into vitreous
• increased risk of late dislocation of IOL capsular bag complex into vitreous
• postoperative intraocular pressure spike:
– completely remove OVD
– intraoperative miotic and postoperative aqueous suppressant
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