Name : Ms. KAVITA Patient UID.

: 9296654
Age/Gender : 27 Yrs/Female Visit No. : 0442025070900001
Referred Client : LDPL8062-BHAGWAN LABORATORY DIGITA Collected on : 08-Jul-2025 10:00AM
Referred By : SELF Received on : 09-Jul-2025 03:06AM
Doctor Name : Reported on : 09-Jul-2025 03:54AM
Sample Type : - ,Serum - 18608321,Whole Blood EDTA - 18608317,Heparin - 18608316

HAEMATOLOGY
Test Name Results Unit Bio. Ref. Interval
HBA1C -GLYCOSYLATED HEMOGLOBIN
Hb A1C, GLYCOSYLATED Hb,wb edta 5.60 % Normal <5.7%
Methodology: by HPLC Prediabetes 5.7% to 6.4%
Diabetes 6.5% or higher
Estimated Average Glucose 114.02 mg/dL 68-125
INTERPRETATION

AS PER AMERICAN DIABETES ASSOCIATION (ADA)

Reference Group HbA1c in %

Non diabetic adults >=18 years < 5.7

At risk (Prediabetes) 5.7 - 6.4

Diagnosing Diabetes >= 6.5

CLINICAL NOTES
In vitro quantitative determination of HbA1c in whole blood is utilized in long term monitoring of [Link] HbA1c level correlates with the mean glucose concentration
prevailing in the course of the patient's recent history (approx - 6-8 weeks) and therefore provides much more reliable information for glycemia monitoring than do determinations
of blood glucose or urinary glucose. It is recommended that the determination of HbA1c be performed at intervals of 4-6 weeks during Diabetes Mellitus therapy. Results of HbA1c
should be assessed in conjunction with the patient's medical history, clinical examinations and other findings.

Some of the factors that influence HbA1c and its measurement [Adapted from Gallagher et al ]
1. Erythropoiesis
- Increased HbA1c: iron, vitamin B12 deficiency, decreased erythropoiesis.
- Decreased HbA1c: administration of erythropoietin, iron, vitamin B12, reticulocytosis, chronic liver disease.
2. Altered Haemoglobin-Genetic or chemical alterations in hemoglobin: hemoglobinopathies, HbF, methemoglobin, may increase or decrease HbA1c.
3. Glycation
- Increased HbA1c: alcoholism, chronic renal failure, decreased intraerythrocytic pH.
- Decreased HbA1c: certain hemoglobinopathies, increased intra-erythrocyte pH.
4. Erythrocyte destruction
- Increased HbA1c: increased erythrocyte life span: Splenectomy.
- Decreased A1c: decreased RBC life span: hemoglobinopathies, splenomegaly, rheumatoid arthritis or drugs such as antiretrovirals, ribavirin & dapsone.
5. Others
- Increased HbA1c: hyperbilirubinemia, carbamylated hemoglobin, alcoholism, large doses of aspirin, chronic opiate use,chronic renal failure
- Decreased HbA1c: hypertriglyceridemia,reticulocytosis, chronic liver disease, aspirin, vitamin C and E,splenomegaly, rheumatoid arthritis or drugs

Note:
[Link] RBC life span –HbA1c test will not be accurate when a person has a condition that affects the average lifespan of red blood cells (RBCs), such as hemolytic anemia
or blood loss. When the lifespan of RBCs in circulation is shortened, the A1c result is falsely low and is an unreliable measurement of a person's average glucose over time.
[Link] forms of hemoglobin – The presence of some hemoglobin variants, such as hemoglobin S in sickle cell anemia, may affect certain methods for measuring A1c. In
these cases, fructosamine can be used to monitor glucose control.

estimated Average Glucose (eAG) : based on value calculated according to National Glycohemoglobin Standardization Program (NGSP) criteria.

*** End Of Report ***

Page 1 of 15
 Name : Ms. KAVITA Patient UID. : 9296654
Age/Gender : 27 Yrs/Female Visit No. : 0442025070900001
Referred Client : LDPL8062-BHAGWAN LABORATORY DIGITA Collected on : 08-Jul-2025 10:00AM
Referred By : SELF Received on : 09-Jul-2025 03:06AM
Doctor Name : Reported on : 09-Jul-2025 03:58AM
Sample Type : - ,Serum - 18608321,Whole Blood EDTA - 18608317,Heparin - 18608316

HAEMATOLOGY
Test Name Results Unit Bio. Ref. Interval
COMPLETE BLOOD COUNT (CBC),WHOLE BLOOD EDTA
HAEMOGLOBIN (Hb) 10.4 g/dL 12.0-15.0
Methodology: colorimetric method
RED BLOOD CELLS- RBC COUNT 4.04 millions/mm³ 4.5 - 5.5
Methodology: DC Impedance with hydrodynamic focusing
PACKED CELL VOLUME (PCV) -HEMATOCRIT 35.2 % 40.0-50.0
Methodology: Pulse Height detection method
MCV 87.13 fL 83-101
Methodology: Automated/Calculated
MCH 25.74 pg 27.0-32.0
Methodology: by Automated/Calculated
MCHC 29.55 g/dL 31.5-34.5
Methodology: Automated/Calculated
RED CELL DISTRIBUTION WIDTH (RDW-CV) 15.7 % 11.6-14.0
Methodology: Automated/Calculated
RED CELL DISTRIBUTION WIDTH (RDW-SD) 51.4 fL 39.0- 46.0
Methodology: Automated/Calculated
MENTZER INDEX 21.57
Methodology: Calculated
PLATELET COUNT 216 10^3/µL 150-410
Methodology: DC Impedance with hydrodynamic focusing/Microscopy
PLATELET DISTRIBUTION WIDTH (PDW) 16.0 fL 9.00-17.00
Methodology: Calculated
PCT(PLATELETCRIT) 0.282 % 0.108-0.282
Methodology: Calculated
MEAN PLATELET VOLUME - MPV 13.1 fL 7.00-12.0
Methodology: Calculated
P-LCR 47.10 % 11.0-45.0
Methodology: Calculated
P-LCC 102.00 % 30.0-90.0
Methodology: Calculated
TOTAL LEUKOCYTE COUNT (TLC) 6.98 10^3/µL 4.00-10.0
Methodology: electric impedance
DIFFERENTIAL LEUCOCYTE COUNT
Neutrophils 61.4 % 40 - 80
Methodology: Flow cytometry/Manual
Lymphocytes 24.9 % 20 - 40
Methodology: Flow cytometry/Manual
Eosinophils 2.8 % 1.00-6.00

Page 2 of 15
 Name : Ms. KAVITA Patient UID. : 9296654
Age/Gender : 27 Yrs/Female Visit No. : 0442025070900001
Referred Client : LDPL8062-BHAGWAN LABORATORY DIGITA Collected on : 08-Jul-2025 10:00AM
Referred By : SELF Received on : 09-Jul-2025 03:06AM
Doctor Name : Reported on : 09-Jul-2025 03:58AM
Sample Type : - ,Serum - 18608321,Whole Blood EDTA - 18608317,Heparin - 18608316
Methodology: Flow cytometry/Manual
Monocytes 10.8 % 2.00-10.0
Methodology: Flow cytometry/Manual
Basophils 0.1 % 0.00-1.00
Methodology: Flow cytometry/Manual
ABSOLUTE NEUTROPHIL COUNT 4.29 10^3/µL 2.00-7.00
Methodology: Calculated
ABSOLUTE LYMPHOCYTE COUNT 1.74 10^3/µL 1.00-3.00
ABSOLUTE EOSINOPHIL COUNT 0.2 10^3/µL 0.02-0.50
Methodology: Calculated
ABSOLUTE MONOCYTE COUNT 0.75 10^3/µL 0.20-1.00
Methodology: Calculated
ABSOLUTE BASOPHIL COUNT 0.01 10^3/µL 0.02-0.10
Methodology: Calculated
CLINICAL NOTES
A complete blood count (CBC) is used to evaluate overall health and detect wide range of disorders, including anemia, infection and [Link] have been some reports
of WBC and platelet counts being lower in venous blood than in capillary blood samples ,although still within these reference ranges.

POSSIBLE CAUSES OF ABNORMAL PARAMETERS

High RBC, Hb, or HCT - dehydration, Low RBC, Hb, or HCT - anemia,
polycythemia,shock, chronic hypoxia thalassemia, and other hemoglobinopathies.
High MCV - macrocytic anemia, liver disease Low MCV - microcytic anemia.
High WBC -acute stress,
Low WBC - sepsis, marrow hypoplasia
infection,malignancies
High platelets - risk of thrombosis Low platelets - risk of bleeding

Notes
[Link] Anemia/Dimorphic Anemia can have low platelet count.
[Link] Anemia/Leucocytosis can have Reactive thrombocytosis.
ERYTHROCYTE SEDIMENTATION RATE (ESR),WHOLE BLOOD EDTA
ESR [WESTERGREN] 22 mm/1st 0 - 15
Methodology: Sedimentation
CLINICAL NOTES
The erythrocyte sedimentation rate (ESR ) is a relatively simple, inexpensive, non-specific test that has been used for many years to help detect inflammation associated
with conditions such as infections, cancers, and autoimmune [Link] test may also be used to monitor disease activity and response to therapy in both of the above
diseases as well as some others, such as lupus.

Factors increasing ESR Factors decreasing ESR

Old age,Pregnancy,Anemia Microcytosis
Elevated fibrinogen Low fibrinogen
Macrocytosis Polycythemia
Macrocytosis Marked leukocytosis

*** End Of Report ***

Page 3 of 15
 Name : Ms. KAVITA Patient UID. : 9296654
Age/Gender : 27 Yrs/Female Visit No. : 0442025070900001
Referred Client : LDPL8062-BHAGWAN LABORATORY DIGITA Collected on : 08-Jul-2025 10:00AM
Referred By : SELF Received on : 09-Jul-2025 03:10AM
Doctor Name : Reported on : 09-Jul-2025 04:20AM
Sample Type : - ,Serum - 18608321,Whole Blood EDTA - 18608317,Heparin - 18608316

BIOCHEMISTRY
Test Name Results Unit Bio. Ref. Interval
CREACTIVE PROTEIN - CRP (QUANTITATIVE)
CREACTIVE PROTEIN - CRP 8.07 mg/L 0-5
(QUANTITATIVE),Serum
Methodology: Turbidimetric
CLINICAL NOTES
CRP is an acute phase reactant, a protein made by the liver and released into the blood within a few hours after tissue injury, the start of an infection, or other cause of
inflammation. The CRP test is not diagnostic of any condition, but it can be used together with signs and symptoms and other tests to evaluate an individual for an acute or
chronic inflammatory [Link] example, CRP may be used to detect or monitor significant inflammation in an individual who is suspected of having an acute condition,
such as:A serious bacterial infection like sepsis, fungal infection,Pelvic inflammatory disease (PID).

Markedly increased levels can occur, for example, after trauma or a heart attack, with active or untreated autoimmune disorders, and with serious bacterial infections,
such as in sepsis. The level of CRP can jump as much as a thousand-fold in response to bacterial infection, and its rise in the blood can precede pain, fever, or other signs
and symptoms.

The CRP test is useful in monitoring people with chronic inflammatory conditions to detect flare-ups and/or to determine if treatment is effective. Some examples
include:Inflammatory bowel disease,Some forms of arthritis,Autoimmune diseases, such as lupus or vasculitis.

CRP may sometimes be ordered along with erythrocyte sedimentation rate (ESR), another test that detects inflammation. While the CRP test is not specific enough to
diagnose a particular disease, it does serve as a general marker for infection and inflammation, thus alerting health practitioners that further testing and treatment may be
necessary. Depending on the suspected cause, a number of other tests may be performed to identify the source of inflammation.

Interpretation of CRP levels:-

Refrence National library of Medicine-C Reactive Protein Sara M. Nehring; Amandeep Goyal; Bhupendra C. Patel(Last Update: July 18, 2022)
Less than 3 mg/L: Normal (level seen in most healthy adults).
3 to 10 mg/L: Normal or minor elevation (can be seen in obesity, pregnancy, depression, diabetes, common cold, gingivitis, periodontitis, sedentary lifestyle, cigarette
smoking, and genetic polymorphisms).
10 to 100 mg/L: Moderate elevation (Systemic inflammation such as RA, SLE, or other autoimmune diseases, malignancies, myocardial infarction, pancreatitis, bronchitis).
More than 100 mg/L: Marked elevation (Acute bacterial infections, viral infections, systemic vasculitis, major trauma).
More than 500 mg/L: Severe elevation (Acute bacterial infections).

*** End Of Report ***

Page 4 of 15
 Name : Ms. KAVITA Patient UID. : 9296654
Age/Gender : 27 Yrs/Female Visit No. : 0442025070900001
Referred Client : LDPL8062-BHAGWAN LABORATORY DIGITA Collected on : 08-Jul-2025 10:00AM
Referred By : SELF Received on : 09-Jul-2025 03:10AM
Doctor Name : Reported on : 09-Jul-2025 04:12AM
Sample Type : - ,Serum - 18608321,Whole Blood EDTA - 18608317,Heparin - 18608316

BIOCHEMISTRY
Test Name Results Unit Bio. Ref. Interval
CALCIUM-SERUM
CALCIUM , Serum 9.12 mg/dL 8.4 - 10.6
Methodology: BAPTA
CLINICAL NOTES
A blood calcium test is ordered to screen for, diagnose, and monitor a range of conditions relating to the bones, heart, nerves, kidneys, and teeth. The test may also be
ordered if a person has symptoms of a parathyroid disorder, malabsorption, or an overactive thyroid. To help diagnose the underlying problem, additional tests are often
done to measure ionized calcium, urine calcium, phosphorus, magnesium, vitamin D, parathyroid hormone (PTH) and PTH-related peptide (PTHrP). PTH and vitamin D are
responsible for maintaining calcium concentrations in the blood within a narrow range of values. Measuring urine calcium can help determine whether the kidneys are
excreting the proper amount of calcium,

Serum calcium is decreased (hypocalcemia) in following conditions-

-Hypoparathyroidism,Pseudohypoparathyroidism
-Vitamin D deficiency (either from intake deficiency or decreased conversion/activation) or resistance (osteomalacia and rickets)
-Chronic renal diseases (eg, renal acidosis, Fanconi syndrome),Chronic liver disease and biliary obstructive diseases
-Magnesium deficiency (PTH glandular release is magnesium-dependent),Hyperphosphatemia,Hypoalbuminemia
-Overexpression of fibroblast growth factor 23 (oncogenic osteomalacia)
-Severe calcium dietary deficiency,Hungry bone syndrome,Severe pancreatitis (calcium saponification),Massive transfusion

Serum calcium is Increased (hypercalcemia) in following conditions-

-Hyperparathyroidism (primary, such MEN type 1, hyperplasia, adenoma, or carcinoma; or secondary, from chronic kidney injury and hyperphosphatemia)
-Malignancies (humoral hypercalcemia of malignancy) that secrete PTH–related protein, especially squamous cell carcinoma of lung and renal cell carcinoma
-Vitamin D excess,Vitamin A intoxication,Milk-alkali syndrome
-Multiple myeloma, owing to bone lesions,Paget disease of bone with prolonged immobilization,Sarcoidosis,Other granulomatous disorders
-Familial hypocalciuria hypercalcemia,Addison disease
-Thyrotoxicosis,Hypothyroidism, owing to prolongation of vitamin D action as its metabolism is slowed down
-Drug exposure: Some drugs that can increase serum calcium are as follows antacids (some), calcium salts, long-term thiazide therapy, lithium

*** End Of Report ***

Page 5 of 15
 Name : Ms. KAVITA Patient UID. : 9296654
Age/Gender : 27 Yrs/Female Visit No. : 0442025070900001
Referred Client : LDPL8062-BHAGWAN LABORATORY DIGITA Collected on : 08-Jul-2025 10:00AM
Referred By : SELF Received on : 09-Jul-2025 03:10AM
Doctor Name : Reported on : 09-Jul-2025 04:12AM
Sample Type : - ,Serum - 18608321,Whole Blood EDTA - 18608317,Heparin - 18608316

BIOCHEMISTRY
Test Name Results Unit Bio. Ref. Interval
LIVER FUNCTION TEST (LFT) - EXTENDED
BILIRUBIN TOTAL,Serum 0.29 mg/dL 0.10 - 1.20
Methodology: Diazonium Ion
DIRECT BILIRUBIN(CONJUGATED), Serum 0.05 mg/dl 0.00-0.20
Methodology: Diazo Method
INDIRECT BILIRUBIN,Serum 0.24 mg/dL 0.80
Methodology: Calculated
SGPT (ALT), SERUM 57.30 U/L 0-35
Methodology: UV without P5P
SGOT (AST) ,SERUM 37.60 U/L 0-40
Methodology: UV without P5P
ALKALINE PHOSPHATASE ,Serum 99.0 U/L 53-128
Methodology: IFCC
GAMMA GLUTAMYL TRANSFERASE (GGT),Serum 19.00 U/L 12.0-58.0
Methodology: IFCC
TOTAL PROTEIN , Serum 8.09 g/dL 6.00-8.30
Methodology: Biuret
Albumin,Serum 4.39 g/dL 3.2-5.20
Methodology: BCG
GLOBULIN,SERUM 3.7 g/dL 2.30-4.50
Methodology: Calculated
A/G Ratio ,Serum 1.19 1.0 - 2.3
Methodology: Calculated
SGOT/SGPT RATIO 0.66
COMMENT
These are group of tests that can be used to detect the presence of liver disease, distinguish among different types of liver disorders, gauge the extent of known liver
damage, and monitor the response to treatment. Most liver diseases cause only mild symptoms initially, but these diseases must be detected early. Some tests are
associated with functionality (e.g., albumin), some with cellular integrity (e.g., transaminase), and some with conditions linked to the biliary tract (gamma-glutamyl transferase
and alkaline phosphatase). Conditions with elevated levels of ALT and AST include hepatitis A,B ,C ,paracetamol toxicity [Link] biochemical tests are useful in the
evaluation and management of patients with hepatic dysfunction. Some or all of these measurements are also carried out (usually about twice a year for routine cases) on
those individuals taking certain medications, such as anticonvulsants, to ensure that the medications are not adversely impacting the person's liver.

Reference ranges are from Teitz fundamental of clinical chemistry 8th ed (2018)

*** End Of Report ***

Page 6 of 15
 Name : Ms. KAVITA Patient UID. : 9296654
Age/Gender : 27 Yrs/Female Visit No. : 0442025070900001
Referred Client : LDPL8062-BHAGWAN LABORATORY DIGITA Collected on : 08-Jul-2025 10:00AM
Referred By : SELF Received on : 09-Jul-2025 03:10AM
Doctor Name : Reported on : 09-Jul-2025 04:58AM
Sample Type : - ,Serum - 18608321,Whole Blood EDTA - 18608317,Heparin - 18608316

BIOCHEMISTRY
Test Name Results Unit Bio. Ref. Interval
KIDNEY FUNCTION TEST (KFT)-BASIC
UREA - SERUM 19.5 mg/dL 15.0 - 40.0
Methodology: Urease UV
CREATININE-SERUM 0.57 mg/dL 0.40-1.10
Methodology: Jaffe Kinetic
URIC ACID - SERUM 4.20 mg/dL 2.60 - 6.00
Methodology: URICASE-POD
SODIUM (SERUM) 139.6 mmol/L 135 - 150
Methodology: ISE
POTASSIUM-SERUM 4.18 mmol/L 3.5 - 5.5
Methodology: ISE
CHLORIDE ,Serum 105.70 mmol/L 94 - 110
Methodology: ISE
BLOOD UREA NITROGEN (BUN) 9.11 mg/dL 8.00-23.0
Methodology: Calculated
BUN/CREATININE RATIO 15.98 Ratio 10-20:1 Normal
Methodology: Calculated
UREA / CREATININE RATIO 34.21 Ratio 40-100:1 Normal
Methodology: Calculated

INTERPRETATION
Kidney function tests are group of tests that can be used to evaluate how well the kidneys are [Link] is a waste product produced by muscles from the breakdown
of a compound called creatine. In blood, it is a marker of GFR ,in urine, it can remove the need for 24-hour collections for many analytes or be used as a quality assurance tool
to assess the accuracy of a 24-hour collection . It is removed from the body by the kidneys, which filter almost all of it from the blood and release it into the urine. This test
measures the amount of creatinine in the blood and/or [Link] is part of the cycle that produces energy needed to contract muscles. Both creatine and creatinine are
produced by the body at a relatively constant rate. Since almost all creatinine is filtered from the blood by the kidneys and released into the urine, blood levels are usually a
good indicator of how well the kidneys are working.
REMARK-The amount of creatinine you produce depends on your body size and your muscle mass. For this reason, creatinine levels are usually slightly higher in men than in
women and [Link] drugs are nephrotoxic hence KFT is done before and after initiation of treatment with these drugs.

Higher creatinine than normal level may be due to: • Blockage in the urinary tract • Kidney problems, such as kidney damage or failure, infection, or reduced blood flow • Loss of
body fluid (dehydration) • Muscle problems, such as breakdown of muscle fibers • Problems during pregnancy, such as seizures (eclampsia)), or high blood pressure caused by
pregnancy (preeclampsia)
Lower than normal creatinine level may be due to: • Myasthenia Gravis • Muscular [Link] serum creatinine values are rare; they almost always reflect low muscle mass.

*** End Of Report ***

Page 7 of 15
 Name : Ms. KAVITA Patient UID. : 9296654
Age/Gender : 27 Yrs/Female Visit No. : 0442025070900001
Referred Client : LDPL8062-BHAGWAN LABORATORY DIGITA Collected on : 08-Jul-2025 10:00AM
Referred By : SELF Received on : 09-Jul-2025 03:10AM
Doctor Name : Reported on : 09-Jul-2025 04:58AM
Sample Type : - ,Serum - 18608321,Whole Blood EDTA - 18608317,Heparin - 18608316

BIOCHEMISTRY
Test Name Results Unit Bio. Ref. Interval
LIPID PROFILE BASIC
CHOLESTEROL TOTAL - Serum 152.60 mg/dl <200 Desirable
Methodology: Cholesterol Oxidase,Esterase,Peroxidase 200-239 Borderline high risk
>240 High risk
TRIGLYCERIDES - SERUM 103.10 mg/dL <150
Methodology: GPO-POD
CHOLESTEROL - HDL (DIRECT) 33.20 mg/dL >40 Recommended Range
Methodology: Direct measure ,polymer-polyanion
NON-HDL CHOLESTEROL 119.40 mg/dL <130
CHOLESTEROL-LDL (DIRECT) 98.78 mg/dL <130 Recommended Range
Methodology: Calculated
VLDL ,SERUM 20.62 mg/dL 0.00 - 45.0
Methodology: Calculated
CHOL/HDL Ratio 4.60 Ratio 3.40-4.40
Methodology: Calculated
LDL/HDL Ratio 2.98 Ratio 1.0-3.5
Methodology: Calculated
HDL/LDL CHOLESTEROL RATIO 0.34 Ratio <3.50
Methodology: Calculated
REFERENCE RANGES AS PER NCEP ATP III GUIDLINES

TOTAL CHOLESTEROL mg/dl HDL mg/dl LDL mg/dl TRIGLYCERIDES mg/dl

Desirable <200 Low <40 Optimal <100 Normal <150
Near Optimal 100-129
Borderline High 200-239 High >60 Borderline High 150-199
Borderline High 130-159
High 160-189 High 200-499
High >240 - -
Very High >190 Very High >500

ALERT!!! 10-12 hours fasting is mandatory for lipid [Link] not,values might fluctuate.
CLINICAL NOTES-Lipid profile is initial screening tool for abnormalities in lipids. The results of this test can identify certain genetic diseases & can determine approximate risks
for cardiovascular disease, certain forms of pancreatitis. Hypertriglyceridemia is indicative of insulin resistance when present with low HDL & elevated LDL, while elevated TG is
risk factor for coronary artery disease,especially when low HDL is [Link] of 500mg/dL or more can be concerning for development of pancreatitis.*The calculated value for
LDL-C is typically reported as part of the lipid profile as per friedewald equation. When triglycerides are high(>350mg/dl), the equation is no longer valid. In this situation,
the only way to accurately determine LDL-C is to measure it directly.

Remark-Measurements in the same patient can show physiological & analytical variations. 3 serial samples 1 week apart are recomended for Total Cholesterol, TG, HDL & LDL
[Link] per NCEP guidelines, all adults above the age of 20 years should be screened for lipid [Link] screening of children above the age of 2 years with a
family history of premature cardiovascular disease or those with at least one parent with high total cholesterol is [Link] Identifies elevated Triglycerides as an
independent risk factor for Coronary Heart Disease (CHD) .RefFriedewald WT, Levy RI, Fredrickson DS. Estimation of the concentration of low-density lipoprotein cholesterol in
plasma, without use of the preparative ultracentrifuge. Clin Chem. 1972, 18;499-502. PubMed ID: 4337382)

*** End Of Report ***

Page 8 of 15
 Name : Ms. KAVITA Patient UID. : 9296654
Age/Gender : 27 Yrs/Female Visit No. : 0442025070900001
Referred Client : LDPL8062-BHAGWAN LABORATORY DIGITA Collected on : 08-Jul-2025 10:00AM
Referred By : SELF Received on : 09-Jul-2025 03:10AM
Doctor Name : Reported on : 09-Jul-2025 04:58AM
Sample Type : - ,Serum - 18608321,Whole Blood EDTA - 18608317,Heparin - 18608316

BIOCHEMISTRY
Test Name Results Unit Bio. Ref. Interval
IRON PROFILE BASIC
IRON -Serum 71.30 ug/dL 59.0-158.0
Methodology: Ferrozine-no Deproteinization
UIBC-SERUM 326.50 ug/dL 110 - 370
Methodology: NiTRO-PSAP
TOTAL IRON BINDING CAPACITY 397.80 ug/dL 240-450
Methodology: Calculated
TRANSFERRIN SATURATION 17.92 % 15.0-50.0
Methodology: Calculated
CLINICAL NOTES
The serum iron test is used to measure the amount of iron that is in transit in the body – the iron that is bound to transferrin in the blood. Along with other tests, it is used to
help detect and diagnose iron deficiency or iron overload. Testing may also be used to help differentiate various causes of [Link] amount of iron present in the blood
will vary throughout the day and from day to day. For this reason, serum iron is almost always measured with other iron tests, including ferritin, transferrin, and calculated
total iron-binding capacity (TIBC) and transferrin [Link] ferritin appears to be in equilibrium with tissue ferritin and is a good indicator of storage iron in normal
subjects and in most disorders. In patients with some hepatocellular diseases, malignancies and inflammatory diseases, serum ferritin is a disproportionately high estimate of
storage iron because serum ferritin is an acute phase reactant. In such disorders iron deficiency anemia may exist with a normal serum ferritin conc. In the presence of
inflammation, persons with low serum ferritin are likely to respond to iron therapy.

Increased Levels
-Iron overload – Hemochromatosis, Thalassemia & Sideroblastic anemia
-Malignant conditions - Acute myeloblastic & Lymphoblastic leukemia, Hodgkin’s disease & Breast carcinoma
-Inflammatory diseases - Pulmonary infections, Osteomyelitis, Chronic UTI,
-Rheumatoid arthritis, SLE, burns,Acute & Chronic hepatocellular disease

Decreased Levels
-Iron deficiency anemia

*** End Of Report ***

Page 9 of 15
 Name : Ms. KAVITA Patient UID. : 9296654
Age/Gender : 27 Yrs/Female Visit No. : 0442025070900001
Referred Client : LDPL8062-BHAGWAN LABORATORY DIGITA Collected on : 08-Jul-2025 10:00AM
Referred By : SELF Received on : 09-Jul-2025 03:10AM
Doctor Name : Reported on : 09-Jul-2025 04:20AM
Sample Type : - ,Serum - 18608321,Whole Blood EDTA - 18608317,Heparin - 18608316

BIOCHEMISTRY
Test Name Results Unit Bio. Ref. Interval
RHEUMATOID FACTOR (RA FACTOR) -QUANTITATIVE
RA FACTOR 3.40 IU/mL <14
Methodology: Turbidimetric
CLINICAL NOTES
Rheumatoid factor (RF) is an autoantibody, an immunoglobulin IgM that is produced by the body's immune system. Autoantibodies attack a person's own tissues, mistakenly
identifying the tissue as "foreign." While the biologic role of RF is not well understood, its presence is useful as an indicator of inflammatory and autoimmune activity. This
test detects and measures RF in the blood and may be used, along with other tests, to help in the diagnose of Rheumatoid Arthritis (RA).RA is a chronic systemic
autoimmune disease that causes inflammation, pain, stiffness, and destructive changes in the hands, feet, and other joints throughout the body. Some patients may show
signs of fatigue, low-grade fevers, and weight loss. Rheumatoid factor has been used to detect RA. Because the sensitivity and specificity of RF are not ideal, other
laboratory tests are often performed in conjunction with RF testing. About 80% of those with RA will have a positive RF test, but it can be negative in people who have
clinical signs of RA.

REMARK
-Elevated RF can be found in a small percentage (5-10%) of healthy people.
-RF may also be elevated in the elderly, though they may not demonstrate clinical signs.
-In addition, elevated levels of RF may be detected in people who do not have RA (false positive) but may have another disorder:

Sjogren syndrome
Systemic lupus erythmatosus
Bacterial, viral and parasitic infections (hepatitis, TB, syphilis, leprosy)
Certain cancers
Lung disease, liver disease, and kidney disease

*** End Of Report ***

Page 10 of 15
 Name : Ms. KAVITA Patient UID. : 9296654
Age/Gender : 27 Yrs/Female Visit No. : 0442025070900001
Referred Client : LDPL8062-BHAGWAN LABORATORY DIGITA Collected on : 08-Jul-2025 10:00AM
Referred By : SELF Received on : 09-Jul-2025 03:10AM
Doctor Name : Reported on : 09-Jul-2025 04:58AM
Sample Type : - ,Serum - 18608321,Whole Blood EDTA - 18608317,Heparin - 18608316

BIOCHEMISTRY
Test Name Results Unit Bio. Ref. Interval
EGFR (ESTIMATED GLOMERULAR FILTRATION RATE)
CREATININE-SERUM 0.57 mg/dL 0.40-1.10
Methodology: Jaffe Kinetic
eGFR 127.66 mL/min/1.73m2 60-180
COMMENT
The Kidney Disease Improving Global Outcomes (KDIGO) guideline defines CKD by the presence of glomerular filtration rate (GFR) 3 months and/or evidence of kidney
damage (eg, structural abnormalities, histologic abnormalities, albuminuria, urinary sediment abnormalities, renal tubular disorders, and/or history of kidney transplantation)
for >3months.2 Thus, monitoring should include tests for GFR, albuminuria, and urine [Link] young adults with normal kidneys will have an eGFR as low as 75
ml/min, and this falls by about 1 ml/min per year as people get older, so many healthy people aged 75 will have an eGFR of 50-60 ml/min

CLINICAL USE
Detect chronic kidney disease (CKD) in adults.
Monitor CKD therapy and/or progression in adults.

Interpretation of eGFR Values(MDRD)

eGFR (mL/min/1.73m2) Interpretation
90-180 Normal
60-89 Mild decrease
45-59 Mild to moderate decrease
30-44 Moderate to severe decrease
15-29 Severe decrease
<15 Kidney failure

Individuals Suitable for Testing

[Link] aged 18 years and older who are at risk of or who have CKD
[Link] with diabetes, hypertension, autoimmune disease, systemic infections, and family history of kidney disease are some of those at increased risk.
[Link] test is not suitable for people with unstable creatinine concentrations such as seen in patients hospitalized with acute illness, in pregnant women, and in those with
serious co-morbid conditions.
[Link] is this test suitable for people with extremes of muscle mass, ie, amputees, paraplegics, bodybuilders, patients with muscle-wasting disease, and patients with a
neuromuscular disorder.
[Link] test is also not suitable for obese people, patients suffering from malnutrition, those with a vegetarian or low-meat diet, and those taking creatine dietary supplements

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Page 11 of 15
 Name : Ms. KAVITA Patient UID. : 9296654
Age/Gender : 27 Yrs/Female Visit No. : 0442025070900001
Referred Client : LDPL8062-BHAGWAN LABORATORY DIGITA Collected on : 08-Jul-2025 10:00AM
Referred By : SELF Received on : 09-Jul-2025 03:10AM
Doctor Name : Reported on : 09-Jul-2025 04:12AM
Sample Type : - ,Serum - 18608321,Whole Blood EDTA - 18608317,Heparin - 18608316

IMMUNOLOGY
Test Name Results Unit Bio. Ref. Interval
TOTAL IGE
Total IgE,Serum 37.20 IU/mL 2-214
Methodology: ECLIA
Comments
Immunoglobulin E (IgE) is the most important trigger molecule for allergic information. The level of IgE is low during the first year of [Link] IgE is a mediator of the allergic
response, quantitative measurement of serum IgE, when integrated with other clinical indicators, can provide useful information for the differential clinical diagnosis of atopic
and not-atopic disease. Patients with atopic disease, including allergic asthma, allergic rhinitis, and atopic dermatitis commonly have moderately elevated serum IgE levels. .
Total serum IgE levels may also be elevated in the presence of some clinical conditions that are not related to allergy eg immunodeficiency states, auto immune disease,
hodgkins disease, bronchopulmonary aspergillosis, IgE myeloma, and Sezary syndrome. . Patients with atopic diseases like Allergic asthma, Allergic rhinitis & Atopic
dermatitis have moderately elevated IgE levels.

Increased Levels - Atopic/Non-atopic allergy, Hyper IgE syndrome, Parasitic infections, IgE Myeloma, Pulmonary Aspergillosis & Autoimmune diseases
Uses
• Evaluation of children with strong family history of allergies and early clinical signs of disease
• To confirm clinical expression of sensitivity to foods in patients with Anaphylactic sensitivity or with Asthma, Angioedema or Cutaneous disease
• To evaluate sensitivity to insect venom allergens particularly as an aid in defining venom specificity in those cases in which skin tests are equivocal
• To confirm the presence of IgE antibodies to certain occupational allergens
Note:
1. Normal levels of IgE do not rule out possibility of IgE dependent allergies as the diagnostic sensitivity of the test depends upon elapsed time between exposure to an
allergen and testing, patient age and affected target organs.
2. No close correlation has been demonstrated between severity of allergic reaction and IgE levels.

Reference ranges
[Link] and Laboratory Standards Institute. Defining, establishing, and verifying reference intervals in the clinical laboratory: approved guideline. CLSI document C28–
A3. Clinical and Laboratory Standards Institute, Wayne, Pa; [Link], F.R.K. Reference values for serum IgE in healthy non-atopic children and adults. Clin
Chem. 1982; 28: 1556

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Page 12 of 15
 Name : Ms. KAVITA Patient UID. : 9296654
Age/Gender : 27 Yrs/Female Visit No. : 0442025070900001
Referred Client : LDPL8062-BHAGWAN LABORATORY DIGITA Collected on : 08-Jul-2025 10:00AM
Referred By : SELF Received on : 09-Jul-2025 03:10AM
Doctor Name : Reported on : 09-Jul-2025 05:14AM
Sample Type : - ,Serum - 18608321,Whole Blood EDTA - 18608317,Heparin - 18608316

IMMUNOLOGY
Test Name Results Unit Bio. Ref. Interval
VITAMIN B12 : CYANOCOBALAMIN
VITAMIN B12 : CYANOCOBALAMIN,Serum 233.10 pg/mL 211 - 911
Methodology: ECLIA
CLINICAL NOTES

Vitamin B12 performs many important functions in the body, but the most significant function is to act as coenzyme for reducing ribonucleotides to deoxyribonucleotides, a
step in the formation of genes. Inadequate dietary intake is not the commonest cause for cobalamine deficiency. The most common cause is malabsorption either due to
atrophy of gastric mucosa or diseases of terminal ileum. Cobalamine deficiency leads to Megaloblastic anemia and demyelination of large nerve fibres of spinal cord.
Sources of Vitamin B12 are liver, shellfish, fish, meat, eggs, milk, cheese & yogurt.

Decreased Levels
-Dietary deficiency: Vegetarians
-Lack of Intrinsic factor: Total or partial gastrectomy, Atrophic gastritis, Intrinsic factor antibodies
-Malabsorption: Regional ileitis, resected bowel, Tropical Sprue, Celiac disease, pancreatic
-insufficiency, bacterial overgrowth & achlorhydria
-Loss of ingested vitamin B12: fish tapeworm
-Congenital disorders: Orotic aciduria & transcobalamine deficiency
-Increased demand: Pregnancy specially last trimester

Increased Levels
-Chronic renal failure, Congestive heart failure, Acute & Chronic Myeloid Leukemia, Polycythemia vera,Carcinomas with liver metastasis, Liver disease, Drug induced
cholestasis & Protein malnutrition

Note: To differentiate vitamin B12 & folate deficiency, measurement of Methyl malonic acid in urine & serum Homocysteine level is suggested

*** End Of Report ***

Page 13 of 15
 Name : Ms. KAVITA Patient UID. : 9296654
Age/Gender : 27 Yrs/Female Visit No. : 0442025070900001
Referred Client : LDPL8062-BHAGWAN LABORATORY DIGITA Collected on : 08-Jul-2025 10:00AM
Referred By : SELF Received on : 09-Jul-2025 03:10AM
Doctor Name : Reported on : 09-Jul-2025 04:12AM
Sample Type : - ,Serum - 18608321,Whole Blood EDTA - 18608317,Heparin - 18608316

IMMUNOLOGY
Test Name Results Unit Bio. Ref. Interval
VITAMIN D 25-HYDROXY CHOLECALCIFEROL
VITAMIN D(25 OH) ,SERUM 16.50 ng/mL Deficiency <20
Methodology: ECLIA Insufficiency 20-30
Sufficiency 30-100
Toxicity >100
CLINICAL NOTES-
Vitamin D is essential for strong bones, because it helps the body use calcium from the diet. Traditionally, vitamin D deficiency has been associated with rickets, a disease
in which the bone tissue doesn't properly mineralize, leading to soft bones and skeletal deformities. But increasingly, research is revealing the importance of vitamin D in
protecting against a host of health problems.

Symptoms and Health risks of vitamin D deficiency

Symptoms of bone pain and muscle weakness can mean you have a vitamin D deficiency. However, for many people, the symptoms are subtle. Yet, even without
symptoms, too little vitamin D can pose health risks. Low blood levels of the vitamin have been associated with Increased risk of cardiovascular disease, Cognitive
impairment in older adults, Severe asthma in children & Cancer.
Research suggests that vitamin D could play a role in the prevention and treatment of a number of different conditions, including type1 and type 2 diabetes, hypertension,
glucose intolerance, and multiple sclerosis.

Causes of vitamin D deficiency

-Peoplle following a strict vegan diet.
-Exposure to sunlight is limited,
-Vitamin D malabsorption- like Crohn's disease, cystic fibrosis, and celiac disease,
-Dietary deficiency
-Severe Hepatocellular disease
-Drugs like Anticonvulsants
-Nephrotic syndrome

Increased levels
-Vitamin D intoxication

*** End Of Report ***

Page 14 of 15
 Name : Ms. KAVITA Patient UID. : 9296654
Age/Gender : 27 Yrs/Female Visit No. : 0442025070900001
Referred Client : LDPL8062-BHAGWAN LABORATORY DIGITA Collected on : 08-Jul-2025 10:00AM
Referred By : SELF Received on : 09-Jul-2025 03:10AM
Doctor Name : Reported on : 09-Jul-2025 05:14AM
Sample Type : - ,Serum - 18608321,Whole Blood EDTA - 18608317,Heparin - 18608316

IMMUNOLOGY
Test Name Results Unit Bio. Ref. Interval
THYROID PROFILE : T3, T4 & TSH(TFT)
TRIODOTHYRONINE TOTAL (T3),Serum 1.32 ng/mL 0.70-2.04
Methodology: ECLIA
THYROXINE TOTAL (T4),Serum 8.59 ug/dl 5.1-14.1
Methodology: ECLIA
THYROID STIMULATING HORMONE (TSH),Serum 2.110 µIU/ml 0.35-5.50
Methodology: ECLIA
NOTE-TSH levels are subject to circardian variation,reaching peak levels between 2-4 AM and min between 6-10 PM. The variation is the order of 50% hence time of the day has influence on the
measures serum TSH [Link] and time of drug intake also influence the test result.
Transient increase in TSH levels or abnormal TSH levels can be seen in some non thyroidal conditions,simoultaneous measurement of TSH with free T4 is useful in evaluating differantial diagnosis.

INTERPRETATION-Ultra Sensitive 4th generation assay

[Link] hyperthyroidism is accompanied by ↑serum T3 & T4 values along with ↓ TSH level.
[Link] TSH,high FT4 and TSH receptor antibody(TRAb) +ve seen in patients with Graves disease
[Link] TSH,high FT4 and TSH receptor antibody(TRAb) -ve seen in patients with Toxic adenoma/Toxic Multinodular goiter
[Link],Low FT4 and Thyroid microsomal antibody increased seen in patients with Hashimotos thyroiditis
[Link],Low FT4 and Thyroid microsomal antibody normal seen in patients with Iodine deficiency/Congenital T4 synthesis deficiency
[Link] TSH,Low FT4 and TRH stimulation test -Delayed response seen in patients with Tertiary hypothyroidism
[Link] hypothyroidism is accompanied by ↓ serum T3 and T4 values & ↑serum TSH levels
[Link] T4 levels accompanied by ↑ T3 levels and low TSH are seen in patients with T3 Thyrotoxicosis
[Link] or↓ T3 & ↑T4 levels indicate T4 Thyrotoxicosis ( problem is conversion of T4 to T3)
[Link] T3 & T4 along with ↓ TSH indicate mild / Subclinical Hyperthyroidism .
[Link] T3 & ↓ T4 along with ↑ TSH is seen in Hypothyroidism .
[Link] T3 & T4 levels with ↑ TSH indicate Mild / Subclinical Hypothyroidism .
[Link] ↑ T3 levels may be found in pregnancy and in estrogen therapy while ↓ levels may be encountered in severe illness , malnutrition , renal failure and during therapy with drugs like
propanolol.
[Link] ↑ TSH levels are nearly always indicative of Primary Hypothroidism ,rarely they can result from TSH secreting pituitary tumours.

DURING PREGNANCY - REFERENCE RANGE for TSH IN uIU/mL (As per American Thyroid Association)
1st Trimester : 0.10-2.50 uIU/mL
2nd Trimester : 0.20-3.00 uIU/mL
3rd Trimester : 0.30-3.00 uIU/mL
The production, circulation, and disintegration of thyroid hormones are altered throughout the stages of pregnancy.

REMARK-Assay results should be interpreted in context to the clinical condition and associated results of other investigations. Previous treatment with corticosteroid therapy may result in lower TSH
levels while thyroid hormone levels are normal. Results are invalidated if the client has undergone a radionuclide scan within 7-14 days before the test. Abnormal thyroid test findings often found in
critically ill patients should be repeated after the critical nature of the condition is [Link] is an important marker for the diagnosis of thyroid [Link] studies have shown that the
TSH distribution progressively shifts to a higher concentration with age ,and it is debatable whether this is due to a real change with age or an increasing proportion of unrecognized thyroid disease in
the elderly.

*** End Of Report ***

Page 15 of 15