Basics of ECG

DR MD ARIFIN KHAN
MBBS,MPH,MD
INTERNIST
 What is an ECG?

ECG = Electrocardiogram
Tracing of heart’s electrical activity

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 Anatomy

Revisited
RCA (Right Coronary Artery)
 Right ventricle
 Inferior wall of LV
 Posterior wall of LV (75%)
 SA Node (60%)
 AV Node (>80%)
 LCA (Left Coronary Artery)
 Septal wall of LV
 Anterior wall of LV
 Inferior wall of LV
 Posterior wall of LV (10%)
 Anatomy

Revisited
SA node
 Intra-atrial pathways
 AV node
 Bundle of His
 Left and Right bundle
branches
 Left anterior fascicle
 Left posterior fascicle
 Purkinje fibers
Bipolar Leads

 1 positive and 1
negative electrode
 RA always negative
 LL always positive
 Traditional limb leads
are examples of these
 Lead I
 Lead II
 Lead III
 View from a vertical
plane
Unipolar Leads

 1 positive electrode & 1

negative “reference point”
 Calculated by using
summation of 2 negative
leads
 Augmented limb leads
 aVR, aFV, aVL

 View from a vertical

plane
 Precordial or chest leads
 V1-V6

 View from a horizontal

plane
12 lead ECG Format

Leads that are

produced by
devices used in the
Pre Hospital setting
 Electrode placement
 10 electrodes in total are placed on the
patient

 Firstly self-adhesive ‘dots’ are attached to

the patient. These have single electrical
contacts on them

 The 10 leads on the ECG machine are

then clipped onto the contacts of the ‘dots’
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 Electrode placement in 12 lead
ECG
 6 are chest electrodes
 Called V1-6 or C1-6

 4 are limb electrodes

 Right arm Ride
 Left arm Your
 Left leg Green
 Right leg Bike

 Remember
 The right leg electrode
is a neutral or “dummy”!
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Lead
Views
 Electrode placement
For the chest electrodes
 V1 4th intercostal space right sternal edge
 V2 4th intercostal space left sternal edge
 (to find the 4th space, palpate the manubriosternal angle
(of Louis)
 Directly adjacent is the 2nd rib, with the 2nd intercostal
space directly below. Palpate inferiorly to find the 3rd and
then 4th space

 V4 over the apex (5th ICS mid-clavicular

line)
 V3 halfway between V2 and V4
 V5 at the same level as V4 but on the
anterior axillary line
 V6 at the same level as V4 and V5 but on
the mid-axillary line
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Electrophysiology

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 Electrophysiology
 Pacemaker = sinoatrial node

 Impulse travels across atria

 Reaches AV node

 Transmitted along interventricular septum in Bundle of

His

 Bundle splits in two (right and left branches)

 Purkinje fibres
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Overall
direction
of
cardiac
impulse

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 How does the ECG work?
 Electrical impulse (wave of depolarisation) picked up by
placing electrodes on patient

 The voltage change is sensed by measuring the current

change across 2 electrodes – a positive electrode and a
negative electrode

 If the electrical impulse travels towards the positive

electrode this results in a positive deflection

 If the impulse travels away from the positive electrode

this results in a negative deflection
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Away from Towards
the the
electrode electrode
= negative = positive
deflection deflection

Direction of impulse (axis)

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 Types of Leads
 Coronal plane (Limb Leads)
1. Bipolar leads — l , l l , l l l
2. Unipolar leads — aVL , aVR , aVF

 Transverse plane
V1 — V6 (Chest Leads)

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Electrodes around the heart
 Leads
How are the 12 leads on the
- Lead I +
ECG (I, II, III, aVL, aVF,
aVR, V1 – 6) formed
using only 9 electrodes
(and a neutral)?

 Lead I is formed using the

right arm electrode (red)
as the negative electrode
and the left arm (yellow)
electrode as the positive

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Leads
- Lead I +

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 Leads
 Lead II is formed
using the right arm
electrode (red) as the
negative electrode Lead II
and the left leg
electrode as the
positive

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Lead II

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 Leads
 Lead III is formed using the left arm
electrode as the negative electrode and
the left leg electrode as the positive

 aVL, aVF, and aVR are composite leads,

computed using the information from the
other leads

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Lead
Groups
I aVR VI V4

II aVL V2 V5

III aVF V3 V6
Limb Leads Chest Leads
 Inferior Wall MI

 II, III, aVF

 View from Left Leg 
 inferior wall of left
ventricle

I aVR VI V4

II aVL V2 V5

III aVF V3 V6
Inferior Wall MI
 Posterior View
 Portion resting on
diaphragm
 ST elevation….suspect
inferior injury

I aVR VI V4

II aVL V2 V5

III aVF V3 V6
 Lateral Wall MI
 1 and AVL
 View from Left Arm 
 Lateral wall of left
ventricle

I aVR VI V4

II aVL V2 V5

III aVF V3 V6
Lateral Wall MI
 V5 and V6
 Left lateral chest
 Lateral wall of left
ventricle

I aVR VI V4

II aVL V2 V5

III aVF V3 V6
Lateral Wall MI
 I, aVL, V5, V6 I aVR VI V4

 ST elevation… II aVL V2 V5
suspect lateral
wall injury III aVF V3 V6

Lateral Wall
 Anterior Wall MI
 V3, V4
 Lateral anterior chest
 + electrode on
anterior chest

I aVR VI V4

II aVL V2 V5

III aVF V3 V6
 Anterior Wall MI
 V3, V4
 ST segment
elevation….suspect
anterior wall injury

I aVR VI V4

II aVL V2 V5

III aVF V3 V6
Septal Wall
MI  V1, V2
 Along sternal borders
 Look through right
ventricle and see
septal wall

I aVR VI V4

II aVL V2 V5

III aVF V3 V6
 Septal Wall
MIV1, V2


 Septum is left
ventricular tissue

I aVR VI V4

II aVL V2 V5

III aVF V3 V6
 Leads and what they tell you
Limb leads
Limb leads look at the heart in the coronal
plane

 aVL, I and II = lateral

 II, III and aVF = inferior
 aVR = right side of the heart

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Leads look at the heart from
different directions

axis
 Leads and what they tell you
Each lead can be thought of as ‘looking at’ an area
of myocardium

Chest leads
V1 to V6 ‘look’ at the heart on the transverse plain
 V1 and V2 look at the anterior of the heart and R
ventricle
 V3 and V4 = anterior and septal
 V5 and V6 = lateral and left ventricle
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Elements of the trace

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 What do the components
represent?

 P wave = atrial depolarisation

 QRS = ventricular depolarisation

 T= repolarisation of the
ventricles

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Interpreting the ECG

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 Interpreting the ECG
 Check
 Name
 DoB
 Time and date
 Indication e.g. “chest pain” or “routine pre-op”
 Any previous or subsequent ECGs
 Is it part of a serial ECG sequence? In which case it may be
numbered
 Calibration
 Rate
 Rhythm
 Axis
 Elements of the tracing in each lead

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 Calibration
Check that your ECG is calibrated correctly

Height
 10mm = 1mV
 Look for a reference pulse which should be the
rectangular looking wave somewhere near the
left of the paper. It should be 10mm (10 small
squares) tall

Paper speed
 25mm/s
 25 mm (25 small squares / 5 large squares)
equals one second
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 Rate
 If the heart rate is regular
 Count the number of large squares between
R waves
 i.e. the RR interval in large squares

 Rate = 300
RR

e.g. RR = 4 large squares

300/4 = 75 beats per minute

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 Rate
If the rhythm is irregular (see next slide on rhythm
to check whether your rhythm is regular or not) it
may be better to estimate the rate using the
rhythm strip at the bottom of the ECG (usually
lead II)

The rhythm strip is usually 25cm long (250mm i.e.

10 seconds)
If you count the number of R waves on that strip
and multiple by 6 you will get the rate

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 Rhythm
Is the rhythm regular?

 The easiest way to tell is to take a sheet of paper and line up one
edge with the tips of the R waves on the rhythm strip.

 Mark off on the paper the positions of 3 or 4 R wave tips

 Move the paper along the rhythm strip so that your first mark lines
up with another R wave tip

 See if the subsequent R wave tips line up with the subsequent

marks on your paper

 If they do line up, the rhythm is regular. If not, the rhythm is irregular

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 Rhythm
Sinus Rhythm
 Definition Cardiac impulse originates from the
sinus node. Every QRS must be
preceded by a P wave.

 (This does not mean that every P wave must be

followed by a QRS – such as in 2nd degree heart
block where some P waves are not followed by a
QRS, however every QRS is preceded by a P wave
and the rhythm originates in the sinus node, hence it
is a sinus rhythm. It could be said that it is not a
normal sinus rhythm)

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 Rhythm
Sinus arrhythmia

 There is a change in heart rate depending on the phase of

respiration

 Q. If a person with sinus arrhythmia inspires, what happens to their

heart rate?

 A. The heart rate speeds up. This is because on inspiration there is

a decrease in intrathoracic pressure, this leads to an increased
venous return to the right atrium. Increased stretching of the right
atrium sets off a brainstem reflex (Bainbridge’s reflex) that leads to
sympathetic activation of the heart, hence it speeds up)

 This physiological phenomenon is more apparent in children and

young adults

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 Rhythm
Sinus bradycardia
 Rhythm originates in the sinus node
 Rate of less than 60 beats per minute

Sinus tachycardia
 Rhythm originates in the sinus node
 Rate of greater than 100 beats per minute

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 Axis
 The axis can be though of as the overall
direction of the cardiac impulse or wave of
depolarisation of the heart

 An abnormal axis (axis deviation) can give

a clue to possible pathology

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 Axis An axis falling
outside the normal
range can be left
or extreme axis deviation
axis
deviation

A normal axis
can lie
anywhe re
between -30
and +90
right axis
degrees
deviation
or +120
degrees
according to
some
 Axis deviation - Causes
 Wolff-Parkinson-White
syndrome can cause both Left
and Right axis deviation

A useful mnemonic:

 “RAD RALPH the LAD from

VILLA”
 Left Axis Deviation
 Right Axis Deviation
 Ventricular tachycardia
 Right ventricular  Inferior MI
hypertrophy  Left ventricular
 Anterolateral MI hypertrophy
 Left Posterior Hemiblock Left Anterior Hemiblock
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 The P wave
The P wave represents atrial
depolarisation

It can be thought of as being

made up of two separate
waves due to right atrial
depolarisation and left atrial Sum of
depolarisation. right and
left waves

Which occurs first? right atrial depolarisation

Right atrial depolarisation left atrial depolarisation

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 The P wave
Dimensions
 No hard and fast rules

Height
 a P wave over 2.5mm should arouse suspicion

Length
 a P wave longer than 0.08s (2 small squares) should
arouse suspicion

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 The P wave
Height

 A tall P wave (over

2.5mm) can be called P
pulmonale

 Occurs due to R atrial

hypertrophy normal P pulmonale
 Causes include: >2.5mm
 pulmonary hypertension,
 pulmonary stenosis
 tricuspid stenosis

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 The P wave
Length

 A P wave with a length

>0.08 seconds (2 small
squares) and a bifid
shape is called P mitrale

 It is caused by left atrial normal P mitrale

hypertrophy and delayed
left atrial depolarisation

 Causes include:
 Mitral valve disease
 LVH
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 The PR interval
 The PR interval is measured between the
start of the P wave to the start of the QRS
complex

 (therefore if there is a Q wave before the R

wave the PR interval is measured from the
start of the P wave to the start of the Q
wave, not the start of the R wave)

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 The PR interval
 The PR interval corresponds to the time
period between depolarisation of the atria
and ventricular depolarisation.

 A normal PR interval is between 0.12 and

0.2 seconds ( 3-5 small squares)

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 The PR interval
 If the PR interval is short (less than 3 small
squares) it may signify that there is an accessory
electrical pathway between the atria and the
ventricles, hence the ventricles depolarise early
giving a short PR interval.

 One example of this is Wolff-Parkinson-White

syndrome where the accessory pathway is
called the bundle of Kent. See next slide for an
animation to explain this

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Depolarisation begins at
the SA node
The wave of
depolarisation spreads
across the atria
It reaches the AV node
and the accessory bundle
Conduction is delayed as
usual by the in-built delay
in the AV node
However, the accessory
bundle has no such delay
and depolarisation begins
early in the part of the
ventricle served by the
bundle

As the depolarisation in this part of the ventricle Until rapid depolarisation

does not travel in the high speed conduction resumes via the normal
pathway, the spread of depolarisation across the pathway and a more normal
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ventricle is slow, causing a slow rising delta wave complex follows medics.cc
 The PR interval
 If the PR interval is long (>5 small squares
or 0.2s):

 If there is a constant long PR interval 1st

degree heart block is present

 First degree heart block is a longer than

normal delay in conduction at the AV node
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 The PR interval
 If the PR interval looks as though it is widening
every beat and then a QRS complex is missing,
there is 2nd degree heart block, Mobitz type I.
The lengthening of the PR interval in
subsequent beats is known as the Wenckebach
phenomenon
 (remember (w)one, Wenckebach, widens)

 If the PR interval is constant but then there is a

missed QRS complex then there is 2nd degree
heart block, Mobitz type II

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 The PR interval
 If there is no discernable relationship
between the P waves and the QRS
complexes, then 3rd degree heart block is
present

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 Heart block (AV node block)
Summary

 1st degree
 constant PR, >0.2 seconds

 2nd degree type 1 (Wenckebach)

 PR widens over subsequent beats then a QRS is dropped
 2nd degree type 2
 PR is constant then a QRS is dropped

 3rd degree
 No discernable relationship between p waves and QRS
complexes

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 The Q wave
Are there any pathological Q
waves?

 A Q wave can be pathological Normal if in

if it is: I,II,III,aVL,V5-6
 Deeper than 2 small squares
(0.2mV)
and/or Pathological
 Wider than 1 small square anywhere
(0.04s)
and/or
 In a lead other than III or one
of the leads that look at the
heart from the left (I, II, aVL,
V5 and V6) where small Qs
(i.e. not meeting the criteria
above) can be normal

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 The QRS height
 If the complexes in the chest leads look
very tall, consider left ventricular
hypertrophy (LVH)

 If the depth of the S wave in V1 added to

the height of the R wave in V6 comes to
more than 35mm, LVH is present

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 QRS width
 The width of the QRS complex should be less
than 0.12 seconds (3 small squares)

 Some texts say less than 0.10 seconds (2.5

small squares)

 If the QRS is wider than this, it suggests a

ventricular conduction problem – usually right or
left bundle branch block (RBBB or LBBB)

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LBBB
 If left bundle branch block
is present, the QRS
complex may look like a
‘W’ in V1 and/or an ‘M’
shape in V6.

 New onset LBBB with

chest pain consider
Myocardial infarction

 Not possible to interpret

the ST segment.

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 RBBB
 It is also called RSR
pattern
 If right bundle branch
block is present, there
may be an ‘M’ in V1
and/or a ‘W’ in V6.

 Can occur in healthy

people with normal QRS
width – partial RBBB

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 QRS width
It is useful to look at leads V1 and V6

 LBBB and RBBB can be remembered by the

mnemonic:

 WiLLiaM MaRRoW

 Bundle branch block is caused either by

infarction or fibrosis (related to the ageing
process)
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 The ST segment
 The ST segment should sit on the isoelectric line

 It is abnormal if there is planar (i.e. flat) elevation

or depression of the ST segment

 Planar ST elevation can represent an MI or

Prinzmetal’s (vasospastic) angina

 Planar ST depression can represent ischaemia

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 Myocardial infarction
 Within hours:
 T wave may become peaked
 ST segment may begin to rise

 Within 24 hours:
 T wave inverts (may or may not persist)
 ST elevation begins to resolve
 If a left ventricular aneurysm forms, ST elevation may persist

 Within a few days:

 pathological Q waves can form and usually persist

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 Myocardial infarction
 The leads affected determine the site of
the infarct

 Inferior II, III, aVF

 Anteroseptal V1-V4
 Anterolateral V4-V6, I, aVL
 Posterior Tall wide R and ST↓ in V1
and V2
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Acute Anterior MI

ST elevation
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 Inferior MI

ST elevation
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 The ST segment
 If the ST segment is elevated but slanted,
it may not be significant

 If there are raised ST segments in most of

the leads, it may indicate pericarditis –
especially if the ST segments are saddle
shaped. There can also be PR segment
depression

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Pericarditis

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 The T wave
 Are the T waves too tall?
 No definite rule for height
 T wave generally shouldn’t
be taller than half the size
of the preceding QRS

 Causes:
 Hyperkalaemia
 Acute myocardial
infarction

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 The T wave
 If the T wave is flat, it may indicate
hypokalaemia

 If the T wave is inverted it may indicate

ischaemia

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 The QT interval
 The QT interval is measured from the start of the
QRS complex to the end of the T wave.

 The QT interval varies with heart rate

 As the heart rate gets faster, the QT interval gets
shorter

 It is possible to correct the QT interval with

respect to rate by using the following formula:
 QTc = QT/√RR (QTc = corrected QT)

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 The QT interval
 The normal range for QTc is 0.38-0.42

 A short QTc may indicate hypercalcaemia

 A long QTc has many causes

 Long QTc increases the risk of developing

an arrhythmia
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 The U wave
 U waves occur after the T wave and are
often difficult to see

 They are thought to be due to

repolarisation of the atrial septum

 Prominent U waves can be a sign of

hypokalaemia, hyperthyroidism
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 Supraventricular tachycardias
 These are tachycardias where the impulse is initiated in
the atria (sinoatrial node, atrial wall or atrioventricular
node)

 If there is a normal conduction pathway when the

impulse reaches the ventricles, a narrow QRS complex
is formed, hence they are narrow complex tachycardias

 However if there is a conduction problem in the

ventricles such as LBBB, then a broad QRS complex is
formed. This would result in a form of broad complex
tachycardia

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medics.cc
 Atrial Fibrillation
Features:

 There maybe tachycardia

 The rhythm is usually irregularly irregular
 No P waves are discernible – instead
there is a shaky baseline
 This is because there is no order to atrial
depolarisation, different areas of atrium
depolarise at will
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Atrial Fibrillation

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medics.cc
 Atrial flutter
 There is a saw-tooth baseline which rises above and
dips below the isoelectric line.
 Atrial rate 250/min
 This is created by circular circuits of depolarisation
set up in the atria

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Ventricular Tachycardia

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 Ventricular Tachycardia
 QRS complexes are wide and irregular in shape
 Usually secondary to infarction
 Circuits of depolarisation are set up in damaged
myocardium
 This leads to recurrent early repolarisation of the
ventricle leading to tachycardia
 As the rhythm originates in the ventricles, there is a
broad QRS complex
 Hence it is one of the causes of a broad complex
tachycardia
 Need to differentiate with supraventricular tachycardia
with aberrant conduction
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Ventricular Fibrillation

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 Ventricular fibrillation
 Completely disordered ventricular
depolarisation

 Not compatible with a cardiac output

 Results in a completely irregular trace

consisting of broad QRS complexes of
varying widths, heights and rates
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 Elements of the tracing
P wave ST segment
 Magnitude and shape,  Should be isoelectric
 e.g. P pulmonale, P mitrale

T wave
PR interval (start of P to start of QRS)  Magnitude and direction
 Normal 3-5 small squares,
0.12-0.2s
QT interval (Start QRS to end of T)
Pathological Q waves?  Normally < 2 big squares or
0.4s at 60bpm
 Corrected to 60bpm
QRS complex
 (QTc) = QT/RRinterval
 Magnitude, duration and
shape
  3 small squares or 0.12s
duration

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THANKS