Joel Arudchelvam
Consultant Vascular and Transplant Surgeon
Teaching Hospital Anuradhapura
Extremity Vascular Injuries
 Injury to the vessels of the limbs
 Common (at THA – 95% of vascular injuries)
 Results in limb loss at times loss of life
 Economic burden
Causes
 Road Traffic injuries – 60%
 Trap Gun
 Iatrogenic - 25%
 Penetrating / Sharp
 Blunt
Mechanism of disruption of flow at
arterial level
 Transection
 Laceration
 Contusion
 Kink
 Intimal flap
Clinical features
 Hard signs
 Active bleeding
 Thrills, Bruits
 Distal ischemia
 Pain
 Pallor
 Pulse – absent
 Perishing cold
 Paresthesia /anaesthesia
 Paresis / paralysis
 Expanding hematoma
 Soft signs
 Hematoma
 Injury close to a known neurovascular bundle
Injury to Popliteal Vessels
 Common
 Amputation rates are high.
 Our experience
 At NHSL
 Popliteal arterial injury – 32.5% of all injuries –commonest
Vascular injury
Vessel Number Mechanism
Iliac artery 2 Blunt
Femoral artery 5 Iatrogenic – 3
blunt (RTA) – 2
Femoral vein 1 Penetrating
Popliteal artery 5 blunt (RTA) -4
Popiteal vein 1 Penetrating
Tibial arteries 4 blunt (RTA)
Brachial artery 2 blunt (RTA)
Vascular injury
Vessel Number Mechanism
Iliac artery 2 Blunt
Femoral artery 5 Iatrogenic – 3
blunt (RTA) – 2
Femoral vein 1 Penetrating
Popliteal artery 5 (25%) blunt (RTA) -4
Popiteal vein 1 Penetrating
Tibial arteries 4 blunt (RTA)
Brachial artery 2 blunt (RTA)
Anatomy
 Fixed
 Proximally - Adductor
Hiatus
 Distally- Soleal Arch.
 Collaterals – Not Very
Effective
 Supplies Blood To Heavy
Muscle Load
HISTORY
 Ligation – common practice during world War I / II
 Amputation Rate – 72.5%
 Korean War / Vietnam Conflict -32%
Reasons given
 Lack of
 Transport
 Sterility Conditions / Antibiotics
 Lack of Blood For Transfusion
 Anesthesia
 Prevented Repair on a Large Scale.
PROGNOSTIC FACTORS
 Time Interval – Common Cause Of Limb Loss In Most Series
 In Our Series – THA – 8.2 Hours
 Mechanism
 Penetrating Wounds Better Outcomes Than From Blunt
Injury
 Because Surrounding Tissue Damage To Be Less Severe.
 Difficult To Diagnose Because Associated Organ And
Tissue Injuries
 Associated Injuries - Skeletal Injuries (Knee Dislocation ,
Popliteal Vein, Nerve, And Soft Tissue And Tendon)
Clinical features
 Hard signs
 Active bleeding
 Thrills, Bruits
 Distal ischemia
 Pain
 Pallor
 Pulse – absent
 Perishing cold
 Paresthesia /anaesthesia
 Paresis / paralysis
 Expanding hematoma
 Soft signs
 Hematoma
 Injury close to a known neurovascular bundle
Clinical features
 Hard signs
 Active bleeding
 Thrills, Bruits
 Distal ischemia
 Pain
 Pallor
 Pulse – absent
 Perishing cold
 Paresthesia /anaesthesia
 Paresis / paralysis
 Expanding hematoma
 Soft signs
 Hematoma
 Injury close to a known neurovascular bundle
Investigations
Investigations
• Hard signs
• urgent intervention
• Soft signs
• Observe
• Investigate
Investigations
Imaging modalities
 Duplex scan
 Angiography
 CT angiography
 Catheter angiography
Investigations
Imaging modalities
 Duplex scan
 Difficult in trauma due to
pain, dressing, wound,
patient not cooperating
 Angiography
 CT angiography
 Catheter angiography
Investigations
Arteriography
 On table
TREATMENT
Surgical Repair
 ABCD / resuscitation
 Repair as soon as possible
 General anesthesia
 Cleaning entire leg and be able to visualize the foot and
palpate distal pulses.
 Contra lateral limb – for venous harvest
 Fasciotomy
 Medial approach
Surgical Repair
 Arterial Ends Trimmed
 Balloon Thrombectomy
 Systemic And Distal
Heparinisation
 Interposition Graft
 Unit Experience
 NHSL - 93.3% RSVG
 ANP– RSVG Upto Now
? Prosthesis
 Infection
 Lower Patency
Surgical Repair
 lateral injury – patch angioplasty
 Our series – none underwent
 Extra-anatomic bypass
 Severe soft tissue injury
 Our series – none underwent
 Skeletal fixation
 When to fix depends on the urgency of vascular
repair, generally skeletal fixation first is
preferred
Compartment Syndrome
 Reduced organ perfusion due to increased
compartment pressure.
 Causes,
 Vascular injury
 Reperfusion
 Haematoma
 contusion
Compartment Syndrome
Treatment – Fasciotomy
Who should do it?
Reperfusion effects
 Local
 Reperfusion injury – paradoxical death of already dying
muscles after reperfusion
 Systemic
 Reperfusion syndrome;
 Hypotension
 ARDS
 Lactic acidosis
 Hyperkalemia
 Kidney shut down
Reperfusion effects
 Mangement
 Fasciotomy
 Hydration
 Mannitol, allopurinol
 O2
 Inotropes
 Ligation of vessel if not responding to above measures
Primary Amputation
 Extensive soft tissue damage / multiple skeletal fractures
with bone loss “Mangled limb”
Summary
 Vascular injury;
 Resuscitate
 Assess viability and extent of injury
 Assess need for fasciotomy
 Early intervention and post intervention monitoring
 Rehabilitation
Thank You

Arterial trauma

  • 1.
    Joel Arudchelvam Consultant Vascularand Transplant Surgeon Teaching Hospital Anuradhapura
  • 2.
    Extremity Vascular Injuries Injury to the vessels of the limbs  Common (at THA – 95% of vascular injuries)  Results in limb loss at times loss of life  Economic burden
  • 3.
    Causes  Road Trafficinjuries – 60%  Trap Gun  Iatrogenic - 25%  Penetrating / Sharp  Blunt
  • 4.
    Mechanism of disruptionof flow at arterial level  Transection  Laceration  Contusion  Kink  Intimal flap
  • 5.
    Clinical features  Hardsigns  Active bleeding  Thrills, Bruits  Distal ischemia  Pain  Pallor  Pulse – absent  Perishing cold  Paresthesia /anaesthesia  Paresis / paralysis  Expanding hematoma  Soft signs  Hematoma  Injury close to a known neurovascular bundle
  • 6.
    Injury to PoplitealVessels  Common  Amputation rates are high.  Our experience  At NHSL  Popliteal arterial injury – 32.5% of all injuries –commonest
  • 7.
    Vascular injury Vessel NumberMechanism Iliac artery 2 Blunt Femoral artery 5 Iatrogenic – 3 blunt (RTA) – 2 Femoral vein 1 Penetrating Popliteal artery 5 blunt (RTA) -4 Popiteal vein 1 Penetrating Tibial arteries 4 blunt (RTA) Brachial artery 2 blunt (RTA)
  • 8.
    Vascular injury Vessel NumberMechanism Iliac artery 2 Blunt Femoral artery 5 Iatrogenic – 3 blunt (RTA) – 2 Femoral vein 1 Penetrating Popliteal artery 5 (25%) blunt (RTA) -4 Popiteal vein 1 Penetrating Tibial arteries 4 blunt (RTA) Brachial artery 2 blunt (RTA)
  • 9.
    Anatomy  Fixed  Proximally- Adductor Hiatus  Distally- Soleal Arch.  Collaterals – Not Very Effective  Supplies Blood To Heavy Muscle Load
  • 10.
    HISTORY  Ligation –common practice during world War I / II  Amputation Rate – 72.5%  Korean War / Vietnam Conflict -32%
  • 11.
    Reasons given  Lackof  Transport  Sterility Conditions / Antibiotics  Lack of Blood For Transfusion  Anesthesia  Prevented Repair on a Large Scale.
  • 12.
    PROGNOSTIC FACTORS  TimeInterval – Common Cause Of Limb Loss In Most Series  In Our Series – THA – 8.2 Hours  Mechanism  Penetrating Wounds Better Outcomes Than From Blunt Injury  Because Surrounding Tissue Damage To Be Less Severe.  Difficult To Diagnose Because Associated Organ And Tissue Injuries  Associated Injuries - Skeletal Injuries (Knee Dislocation , Popliteal Vein, Nerve, And Soft Tissue And Tendon)
  • 13.
    Clinical features  Hardsigns  Active bleeding  Thrills, Bruits  Distal ischemia  Pain  Pallor  Pulse – absent  Perishing cold  Paresthesia /anaesthesia  Paresis / paralysis  Expanding hematoma  Soft signs  Hematoma  Injury close to a known neurovascular bundle
  • 14.
    Clinical features  Hardsigns  Active bleeding  Thrills, Bruits  Distal ischemia  Pain  Pallor  Pulse – absent  Perishing cold  Paresthesia /anaesthesia  Paresis / paralysis  Expanding hematoma  Soft signs  Hematoma  Injury close to a known neurovascular bundle
  • 15.
    Investigations Investigations • Hard signs •urgent intervention • Soft signs • Observe • Investigate
  • 16.
    Investigations Imaging modalities  Duplexscan  Angiography  CT angiography  Catheter angiography
  • 17.
    Investigations Imaging modalities  Duplexscan  Difficult in trauma due to pain, dressing, wound, patient not cooperating  Angiography  CT angiography  Catheter angiography
  • 18.
  • 19.
    TREATMENT Surgical Repair  ABCD/ resuscitation  Repair as soon as possible  General anesthesia  Cleaning entire leg and be able to visualize the foot and palpate distal pulses.  Contra lateral limb – for venous harvest  Fasciotomy  Medial approach
  • 20.
    Surgical Repair  ArterialEnds Trimmed  Balloon Thrombectomy  Systemic And Distal Heparinisation  Interposition Graft  Unit Experience  NHSL - 93.3% RSVG  ANP– RSVG Upto Now ? Prosthesis  Infection  Lower Patency
  • 21.
    Surgical Repair  lateralinjury – patch angioplasty  Our series – none underwent  Extra-anatomic bypass  Severe soft tissue injury  Our series – none underwent  Skeletal fixation  When to fix depends on the urgency of vascular repair, generally skeletal fixation first is preferred
  • 22.
    Compartment Syndrome  Reducedorgan perfusion due to increased compartment pressure.  Causes,  Vascular injury  Reperfusion  Haematoma  contusion
  • 23.
    Compartment Syndrome Treatment –Fasciotomy Who should do it?
  • 24.
    Reperfusion effects  Local Reperfusion injury – paradoxical death of already dying muscles after reperfusion  Systemic  Reperfusion syndrome;  Hypotension  ARDS  Lactic acidosis  Hyperkalemia  Kidney shut down
  • 25.
    Reperfusion effects  Mangement Fasciotomy  Hydration  Mannitol, allopurinol  O2  Inotropes  Ligation of vessel if not responding to above measures
  • 26.
    Primary Amputation  Extensivesoft tissue damage / multiple skeletal fractures with bone loss “Mangled limb”
  • 27.
    Summary  Vascular injury; Resuscitate  Assess viability and extent of injury  Assess need for fasciotomy  Early intervention and post intervention monitoring  Rehabilitation
  • 28.