Psychiatry department
Beni Suef University
cdepression,
 phobia,
 anxiety and
 psychoses
 pervasive developmental disorders
 attention deficit/hyperactivity disorder,
 conduct disorder and
 mental retardation.
 functional enuresis,
 functional encopresis, and
 separation anxiety.



This is a form of disruptive behavior in which
the basic rights of others and age appropriate
societal norms or rules are violated.

 Epidemiology
 It

usually starts before the age of 18 years
 male: female ratio 10:1.
 6-16 % of boys and 2-9 % of girls below 18y have
conduct disorder.
 The

disorder is either conducted solitary or
in a group (gang).
 Aggression may be either direct (overt) or
indirect.
 A-

Overt aggression is directed to people,
animals or property with the aim of
deliberate injury or destruction.

 B-

Indirect aggression as shoplifting, lying,
and staying out late at night despite of
parental prohibition.
 It

 1.

is a multifactorial disorder:

Genetic factors
 2. Organic factors
 3. Environmental factors
 4. Family factors
 5. Social Modeling
Family factors
• Neglecting unavailable mother with absence of
support
 • drug abuse or antisocial father

• Higher psychiatric morbidity among parents
with personality deviation

•Frequent inconsistent punishment

• Increased marital discord
 • Disturbed family structure, increased marital
conflicts, divorce and parental violence.

1-For the Child
 • Behavioral therapy
 • Group therapy
 • Pharmacotherapy (to control aggression &
impulsivity)
 a. Lithium carbonate
 b. Clonidine
 c. Anticonvulsants
2- Family therapy
3- Parental training
4- Institutionalization
 Epidemiology
 This

disorder is more common in males than
in females in the ratio 3-5 : l.
 In the United States, its incidence is 3-5 % of
primary school children.
 In Britain, it is less than 1 %.
It includes three main criteria:
 1-

Disturbed attention or concentration:
 2- Hyperactivity
 3- Impulsivity
 1.

Genetic factors
 2. Organic factors (frontal lobe)
 3. Environmental factors (food additives,
preservatives, toxins)
1. Pharmacotherapy:
 a. Psychostimulants, e.g.,
dextroamphetamine, methylphenidate
(Ritalin)
 b. Antidepressants
 c. Antipsychotics
 d. Lithium carbonate
2. Special education programs
3. Family therapy
 This

is a group of psychiatric conditions in
which the expected social skills, language
behavior and behavioral repertoire are
either not developed or are lost in early
childhood before the age of 3 years.
 The most common type is Autistic Disorder.
 Epidemiology
 Autistic

Disorder occurs at the rate of 2-5 per
10,000 children under the age of 12. Male to
female ratio is 3-5 to 1.
 1.

Inability to develop relationship with
people.
 2. Delayed development of language skill,
 3. Repetitive or stereotyped movements,
It is multifactorial including
 1. Psychogenic factors
 2. Genetic factors
 3. Perinatal complications, especially during
the first trimester.
 4. Biochemical factors
 5. Neurologphysiology: EEG changes in 10-85
% of autistic children
 The

goal is to decrease the behavioral
symptoms and to help the development of
the delayed functions.
 1. Supportive home environment
 2. Special educational programs
 3. Pharmacotherapy: useful in modifying and
controlling behavior
high potency neuroleptics
Selective Serotonin Reuptake Inhibitors
(SSRI)
Functional Enuresis
 Enuresis is the repeated voiding of urine into
the child's clothes or bed.
 It may be involuntary or intentional.
Nocturnal bed wetting is the most common
form.
 Daytime control usually precedes nocturnal
control by 1-2 years.

 Prevalence

of enuresis varies greatly in
different groups, in the States 7 % of 5 year
olds are enuretic.
 To
 1.

diagnose functional enuresis:

The child must be at least 5 years old
 2. Wetting is repetitive
 3. Medical causes should be ruled out
particularly in secondary enuresis.
 Most common medical causes are urinary
tract infection, diabetes, seizure disorders
and congenital abnormalities.
•

Primary: if bladder control has never been
achieved

•

Secondary: if urinary incontinence
reappearance after maintainmg competent
functions for 1 year.
 1.Restricting

fluids before bedtime
 2.Waking the child during the night.
 3. Rewarding successful dry nights.
 4. Bladder training during the day, i.e.,
delaying bladder emptying
 5. Medications: given before going to bed,
such as:
imipramine (Tofranil),
desmopressin (synthetic ADH)
anticholinergic drugs.
 It

is characterized by fecal soiling of clothes.
Medical evaluation is necessary before
labeling the disorder as functional.

 Epidemiology

After the age of four years, encopresis occurs
3-4 times more in boys than in girls. There is
a significant relation between encopresis and
enuresis.
 Diagnosis
 1.

The child is at least 4 years old.
 2. Encopresis occurs at least once a month
for at least 3 months.
 3. Medical causes should be excluded.


a. Primary or secondary: primary if no bowel
control has been achieved, and secondary if the
child has learned control for one year.

b. With constipation and overflow, or without
constipation:
 75 % of encopretic children have constipation.
 There is fecal concretion with overflow of fluid
fecal matter.
 Incontinence without constipation results in
intermittent production of formed stools.

 1.

For encopresis without constipation, a
behavioral program gives rewards for just
sitting on the toilet then later for moving
bowels appropriately.
 2. For children with severe retention or
impaction cleaning out the bowel initially (
enemas), followed by retraining the bowel
(high roughage diet, developing of a toilet
routine) are used in addition to behavioral
program
 3. In resistant cases individual and family
psychotherapeutic interventions are needed.
 These

disorders are termed academic skills
disorders.
 These children usually present with one of
the basic psychological problems involved in
understanding or in using spoken or written
language.
 They usually present with poor scholastic
achievement despite their average
intelligence as assessed by the individually
administered standardized intelligence tests.
 Impairment

in the academic areas includes
disorders in:
 • Reading
 • Mathematics
 • Written expression.
 It

might be associated with:
 1. Delayed speech
 2. Anxiety and other emotional problems.
 3. They may as well present behavioral
problems such as alienation or rebellion.
 Etiology
 It

includes a variety of neurocortical deficits
resulting in various
 disruptions of cognitive processing, e.g.
difficulty in visual spatial or linguistic
processing.
 Management
 1.

Special assessment including 1Q, EEG,
plain X ray skull, and CT scan brain
 2. Special educational programs with special
scholastic placements.
 3. Family counseling and training programs to
help in the education.
 4. Teacher's education to help in the
education progress
 5. Psychotherapy for the patient and family.
 The

diagnosis of Mental Retardation MR
requires both low intelligence (IQ less than
70) and
 deficits in adaptive functions i.e. impairment
of skills manifested during the
developmental period (before the age of 18
years)
 including cognitive, language, motor and
social abilities.
 Classification
 The

intelligence quotient was calculated
from the following formula:
 IQ= mental age/ chronological age x 100
 On basis of IQ : mental retardation is
classified into:
 Mild:
IQ 50-69
 Moderate:
IQ 35-49
 Severe:
IQ 20-34
 Profound:
IQ below 20
a. Biological Causes:
 Genetic Factors
 Prenatal Factors
 Perinatal Factors
 Causes during Infancy or childhood
b. Psychosocial Causes
Majority (85%) of those with M.R.
• Self care and living skills:
 Most have no difficulty in achieving full
independence in self-care (eating, washing,
dressing, and sphincteric control).
 They may need help with planning a budget.
• Language and communication skills:
 Most achieve the ability to use speech for
everyday purposes and can hold conversations
in normal circumstances.
• Education and occupation:
 Educable, many have difficulties reading and
writing, but can achieve an academic level of
grade 6.
 They can hold a job.

10% of those with M.R.
• Self care and living skills:
 Achievement of self care and motor skills is retarded, yet
they can be trained to attain considerable independence in
daily living but they need supervision.
 They are usually capable of managing pocket money but
find difficulty in calculating the change due.
• Language and communication skills:
 Slow in developing comprehension and use of language,
however they are usually able to communicate adequately.
• Education and occupation:
 Limited progress with school work, usually not beyond the
academic level of grade 2,
 They are trainable.
 Some adults can carry out simple manual work.

 4%

of those with M.R.
 • Self care and living skills: They need a
great deal of supervision as their self-care
and motor skills are markedly impaired.
 They are dependent on others for money
arrangement
 • Language and communication skills: The
development of comprehension and use of
language is very limited and communication
is often not by speech.
 • Education: Below first grade. They are not
trainable.
Profound M.R. (IQ below 20):
1% of MR
• Self care and living skills: Constant help
and supervision is needed for basic needs.
• Language and communication skills:
Severely limited in ability to understand
language.
They communicate in a very limited nonverbal way.
• Education: Extremely limited
 For

mental retardation at all levels of
severity, the developmental course is
SLOW but not deviant.



Although the normal sequence of
developmental stages occurs, the speed of
developmental change is slow and there is
a ceiling on ultimate achievement.
Mentally retarded children are four to five times
at a higher risk to have a psychiatric disorder
than children with normal intelligence.
 The most common constellation of symptoms
includes:
 irritability,
 hyperactivity,
 impulsivity,
 short attention span and
 language delay.
 aggressive temper outbursts.













1. Early detection of treatable causes as
hypothyroidism and malnutrition.
2. Proper comprehensive evaluation to address the
multiple disabilities and complications associated
with MR whether medical or psychiatric.
3. Parental guidance: support, education, genetic.
4. Detecting strengths and weaknesses
5. Specialists for speech therapy.
6. Behavior modification
7. Psychotherapy (mild MR) to enhance self-esteem,
social and emotional development and behavioral
control.
8. Treatment of co-morbid conditions e.g. depression
or ADHD.
Child psychiatry

Child psychiatry

  • 1.
  • 2.
    cdepression,  phobia,  anxietyand  psychoses  pervasive developmental disorders  attention deficit/hyperactivity disorder,  conduct disorder and  mental retardation.  functional enuresis,  functional encopresis, and  separation anxiety. 
  • 3.
     This is aform of disruptive behavior in which the basic rights of others and age appropriate societal norms or rules are violated.  Epidemiology  It usually starts before the age of 18 years  male: female ratio 10:1.  6-16 % of boys and 2-9 % of girls below 18y have conduct disorder.
  • 4.
     The disorder iseither conducted solitary or in a group (gang).  Aggression may be either direct (overt) or indirect.  A- Overt aggression is directed to people, animals or property with the aim of deliberate injury or destruction.  B- Indirect aggression as shoplifting, lying, and staying out late at night despite of parental prohibition.
  • 5.
     It  1. isa multifactorial disorder: Genetic factors  2. Organic factors  3. Environmental factors  4. Family factors  5. Social Modeling
  • 6.
    Family factors • Neglectingunavailable mother with absence of support  • drug abuse or antisocial father  • Higher psychiatric morbidity among parents with personality deviation  •Frequent inconsistent punishment  • Increased marital discord  • Disturbed family structure, increased marital conflicts, divorce and parental violence. 
  • 7.
    1-For the Child • Behavioral therapy  • Group therapy  • Pharmacotherapy (to control aggression & impulsivity)  a. Lithium carbonate  b. Clonidine  c. Anticonvulsants 2- Family therapy 3- Parental training 4- Institutionalization
  • 8.
     Epidemiology  This disorderis more common in males than in females in the ratio 3-5 : l.  In the United States, its incidence is 3-5 % of primary school children.  In Britain, it is less than 1 %.
  • 9.
    It includes threemain criteria:  1- Disturbed attention or concentration:  2- Hyperactivity  3- Impulsivity
  • 10.
     1. Genetic factors 2. Organic factors (frontal lobe)  3. Environmental factors (food additives, preservatives, toxins)
  • 11.
    1. Pharmacotherapy:  a.Psychostimulants, e.g., dextroamphetamine, methylphenidate (Ritalin)  b. Antidepressants  c. Antipsychotics  d. Lithium carbonate 2. Special education programs 3. Family therapy
  • 12.
     This is agroup of psychiatric conditions in which the expected social skills, language behavior and behavioral repertoire are either not developed or are lost in early childhood before the age of 3 years.  The most common type is Autistic Disorder.
  • 13.
     Epidemiology  Autistic Disorderoccurs at the rate of 2-5 per 10,000 children under the age of 12. Male to female ratio is 3-5 to 1.
  • 14.
     1. Inability todevelop relationship with people.  2. Delayed development of language skill,  3. Repetitive or stereotyped movements,
  • 15.
    It is multifactorialincluding  1. Psychogenic factors  2. Genetic factors  3. Perinatal complications, especially during the first trimester.  4. Biochemical factors  5. Neurologphysiology: EEG changes in 10-85 % of autistic children
  • 16.
     The goal isto decrease the behavioral symptoms and to help the development of the delayed functions.  1. Supportive home environment  2. Special educational programs  3. Pharmacotherapy: useful in modifying and controlling behavior high potency neuroleptics Selective Serotonin Reuptake Inhibitors (SSRI)
  • 18.
    Functional Enuresis  Enuresisis the repeated voiding of urine into the child's clothes or bed.  It may be involuntary or intentional. Nocturnal bed wetting is the most common form.  Daytime control usually precedes nocturnal control by 1-2 years. 
  • 19.
     Prevalence of enuresisvaries greatly in different groups, in the States 7 % of 5 year olds are enuretic.
  • 20.
     To  1. diagnosefunctional enuresis: The child must be at least 5 years old  2. Wetting is repetitive  3. Medical causes should be ruled out particularly in secondary enuresis.  Most common medical causes are urinary tract infection, diabetes, seizure disorders and congenital abnormalities.
  • 21.
    • Primary: if bladdercontrol has never been achieved • Secondary: if urinary incontinence reappearance after maintainmg competent functions for 1 year.
  • 22.
     1.Restricting fluids beforebedtime  2.Waking the child during the night.  3. Rewarding successful dry nights.  4. Bladder training during the day, i.e., delaying bladder emptying  5. Medications: given before going to bed, such as: imipramine (Tofranil), desmopressin (synthetic ADH) anticholinergic drugs.
  • 23.
     It is characterizedby fecal soiling of clothes. Medical evaluation is necessary before labeling the disorder as functional.  Epidemiology After the age of four years, encopresis occurs 3-4 times more in boys than in girls. There is a significant relation between encopresis and enuresis.
  • 24.
     Diagnosis  1. Thechild is at least 4 years old.  2. Encopresis occurs at least once a month for at least 3 months.  3. Medical causes should be excluded.
  • 25.
     a. Primary orsecondary: primary if no bowel control has been achieved, and secondary if the child has learned control for one year. b. With constipation and overflow, or without constipation:  75 % of encopretic children have constipation.  There is fecal concretion with overflow of fluid fecal matter.  Incontinence without constipation results in intermittent production of formed stools. 
  • 26.
     1. For encopresiswithout constipation, a behavioral program gives rewards for just sitting on the toilet then later for moving bowels appropriately.  2. For children with severe retention or impaction cleaning out the bowel initially ( enemas), followed by retraining the bowel (high roughage diet, developing of a toilet routine) are used in addition to behavioral program  3. In resistant cases individual and family psychotherapeutic interventions are needed.
  • 27.
     These disorders aretermed academic skills disorders.  These children usually present with one of the basic psychological problems involved in understanding or in using spoken or written language.  They usually present with poor scholastic achievement despite their average intelligence as assessed by the individually administered standardized intelligence tests.
  • 28.
     Impairment in theacademic areas includes disorders in:  • Reading  • Mathematics  • Written expression.  It might be associated with:  1. Delayed speech  2. Anxiety and other emotional problems.  3. They may as well present behavioral problems such as alienation or rebellion.
  • 29.
     Etiology  It includesa variety of neurocortical deficits resulting in various  disruptions of cognitive processing, e.g. difficulty in visual spatial or linguistic processing.
  • 31.
     Management  1. Specialassessment including 1Q, EEG, plain X ray skull, and CT scan brain  2. Special educational programs with special scholastic placements.  3. Family counseling and training programs to help in the education.  4. Teacher's education to help in the education progress  5. Psychotherapy for the patient and family.
  • 32.
     The diagnosis ofMental Retardation MR requires both low intelligence (IQ less than 70) and  deficits in adaptive functions i.e. impairment of skills manifested during the developmental period (before the age of 18 years)  including cognitive, language, motor and social abilities.
  • 33.
     Classification  The intelligencequotient was calculated from the following formula:  IQ= mental age/ chronological age x 100  On basis of IQ : mental retardation is classified into:  Mild: IQ 50-69  Moderate: IQ 35-49  Severe: IQ 20-34  Profound: IQ below 20
  • 34.
    a. Biological Causes: Genetic Factors  Prenatal Factors  Perinatal Factors  Causes during Infancy or childhood b. Psychosocial Causes
  • 35.
    Majority (85%) ofthose with M.R. • Self care and living skills:  Most have no difficulty in achieving full independence in self-care (eating, washing, dressing, and sphincteric control).  They may need help with planning a budget. • Language and communication skills:  Most achieve the ability to use speech for everyday purposes and can hold conversations in normal circumstances. • Education and occupation:  Educable, many have difficulties reading and writing, but can achieve an academic level of grade 6.  They can hold a job. 
  • 36.
    10% of thosewith M.R. • Self care and living skills:  Achievement of self care and motor skills is retarded, yet they can be trained to attain considerable independence in daily living but they need supervision.  They are usually capable of managing pocket money but find difficulty in calculating the change due. • Language and communication skills:  Slow in developing comprehension and use of language, however they are usually able to communicate adequately. • Education and occupation:  Limited progress with school work, usually not beyond the academic level of grade 2,  They are trainable.  Some adults can carry out simple manual work. 
  • 37.
     4% of thosewith M.R.  • Self care and living skills: They need a great deal of supervision as their self-care and motor skills are markedly impaired.  They are dependent on others for money arrangement  • Language and communication skills: The development of comprehension and use of language is very limited and communication is often not by speech.  • Education: Below first grade. They are not trainable.
  • 38.
    Profound M.R. (IQbelow 20): 1% of MR • Self care and living skills: Constant help and supervision is needed for basic needs. • Language and communication skills: Severely limited in ability to understand language. They communicate in a very limited nonverbal way. • Education: Extremely limited
  • 39.
     For mental retardationat all levels of severity, the developmental course is SLOW but not deviant.  Although the normal sequence of developmental stages occurs, the speed of developmental change is slow and there is a ceiling on ultimate achievement.
  • 40.
    Mentally retarded childrenare four to five times at a higher risk to have a psychiatric disorder than children with normal intelligence.  The most common constellation of symptoms includes:  irritability,  hyperactivity,  impulsivity,  short attention span and  language delay.  aggressive temper outbursts. 
  • 41.
            1. Early detectionof treatable causes as hypothyroidism and malnutrition. 2. Proper comprehensive evaluation to address the multiple disabilities and complications associated with MR whether medical or psychiatric. 3. Parental guidance: support, education, genetic. 4. Detecting strengths and weaknesses 5. Specialists for speech therapy. 6. Behavior modification 7. Psychotherapy (mild MR) to enhance self-esteem, social and emotional development and behavioral control. 8. Treatment of co-morbid conditions e.g. depression or ADHD.