PULP PERIODONTIUM
Confusion & controversy
Pathways of
spread of
infection
Diagnosis,
prognosis,
treatment
EMBRYONIC ANATOMIC FUNCTIONAL
ENDO- PERIO LESION
Lesions due to inflammatory products found in varying
degrees in both periodontium and pulpal tissues.
• Tooth with necrotic pulp reaching apical
periodontium
• Root perforations
• Root fractures
• Chronic periapical abscess with sinus tract
• Replants
• Transplants
• Teeth requiring hemisection
• Occlusal trauma causing reversible pulpitis
• Supra/infra bony pockets caused during
periodontal treatment resulting in pulpal
inflammation
• Occlusal trauma and gingival inflammation
resulting in pocket formation
• Any lesion of type I which result in irreversible
reaction to periodontium requiring periodontal
treatment
• Any lesion of type II which results in irreversible
damage to pulp tissue requiring endodontic
- Pulpal infection may cause a tissue
destructive process that proceeds from
the apical or furcal region of a tooth
towards the gingival margin, as
opposed to MARGINAL
PERIODONTITIS in which infection
spreads from the gingival margin
toward the root apex.
Endo-periodontal lesion with root
damage
Root fracture or cracking
Root canal or pulp chamber perforation
External root resorption
Endo-periodontal lesion without root
damage
Endo-periodontal lesion
in periodontitis patients
Grade 1- narrow deep
periodontal pocket in 1 tooth
surface
Grade 2- wide deep periodontal
pocket in 1 tooth surface
Grade 3- deep periodontal
pockets in more than 1 tooth
surface
Endo-periodontal lesion
in non-periodontitis
patients
Grade 1- narrow deep
periodontal pocket in 1 tooth
surface
Grade 2- wide deep periodontal
pocket in 1 tooth surface
Plaque Foreign bodies
Contributing
factors
Pulpal inflammation/necrosis
Inflammatory response in PDL
Gingival sulcus of involved tooth
Gingival sulcus of adjacent tooth
Severe – destruction of supporting
apparatus viz., PDL, socket, etc.
Localized swelling
Draining sinus tract
Diffuse swelling
Attached gingiva
Alveolar mucosa
Pulpal pathosis
Accessory
canal/ apical
foramen
Retrograde
periodontitis
Pathogenic bacteria &
inflammatory products
of periodontal disease
Accessory canals/
lateral canals/apical
foramen
Retrograde pulpitis
Pulpal response – remaining dentin
thickness
Negligible response
Repair & Healing
Pulpal response
Necrosis
Prophylactic root canal
treatment to be completed
before periodontal treatment
 SCALING & ROOT PLANING
- REASONS:
1. Dissolved smear layer–
- increased hydraulic conductance of involved
dentinal tubules
- decreased peripheral resistance to fluid flow
across dentin
2. Open dentinal tubules– pathway for diffuse
transport of bacterial elements in oral cavity to the
pulp
3. Peripheral sensitization of pulp nociceptors due to
release of inflammatory mediators as seen by A∂
fibers.
 IN CONCLUSION, unless periodontal disease extends
all the way to the tooth apex, the effect of periodontal
disease treatment on dental pulp is ineffective
ENDODONTIC LESION PERIODONTAL LESION
Pain:
Acute, severe, throbbing type, poor localization, postural
variation, referred to other sites, not much response to
analgesics etc.
Chronic onset. Mild to moderate in nature, able to
localize pain, no postural variation, responds to mild
analgesics etc.
Swelling:
Mucobuccal fold area, spreads to facial plane, extraoral
swelling
Attached gingiva, rarely spreads beyond MGJ, rarely
facial swellings
Mobility:
Isolated tooth mobility if acute abscess
Usually generalized mobility
Periodontal probing:
Precipitous drop in probing depth, lesion is narrow
coronally than at the apex, no calculus
Generalized deep probing, lesion is broader at its point of
origin at the apex, calculus on the root
Fistulation:
Traced to radiographic apex with negative vitality
Traced to mid-root
Percussion: vertical Horizontal
Pulp vitality: Nonvital Vital
MOBILITY
HISTORY
PULP VITALITY
VISUAL EXAMINATION
PALPATION PERIODONTAL PROBING
RADIOGRAPH
PERCUSSION
SINUS TRACING, CRACKED TOOTH TEST
(TRANSILLUMINATION, WEDGING), SELECTIVE
ANESTHESIA TEST to localize pain to specific site
PRIMARY ENDODONTIC
LESION
Soft tissue- presence of sinus opening
Tooth- presence of decay/large restoration/ #
restoration or tooth/
erosions/abrasions/cracks/discolorations/poor
RCT
PRIMARY PERIODONTAL
LESION
Inflamed gingiva/gingival recession around
multiple teeth
Accumulation of plaque and subgingival
calculus around multiple teeth
Intact teeth
Presence of swelling indicating periodontal
abscess
PRIMARY ENDO-
SECONDARY PERIO
Plaque forms at the gingival margin of the
sinus tract and leads to inflammation of
marginal gingiva exudate
Root perforation/fracture/misplaced post
PRIMARY PERIO-
SECONDARY ENDO
Presence of plaque, subgingival calculus and
swelling around multiple teeth
Presence of pus, exudate
Presence of localized/generalized gingival
recession and exposure of root
TRUE COMBINED LESION/
CONCOMITANT PULPAL
Plaque, calculus and periodontitis will be
present in varying degrees
PRIMARY ENDODONTIC
LESION
Sharp
Does not indicate whether inflammation is of
periodontal/endodontic origin
PRIMARY PERIODONTAL
LESION
Usually dull ache
Sharp only in acute condition
PRIMARY ENDO-
SECONDARY PERIO
Usually sharp shooting pain
Dull ache in chronic conditions
PRIMARY PERIO-
SECONDARY ENDO
Usually dull ache
Sharp only in periodontal abscess
TRUE COMBINED LESION/
CONCOMITANT PULPAL
AND PERIODONTAL
LESIONS
Dull ache
Severe pain only in acute conditions
PRIMARY ENDODONTIC
LESION
Normally tender on percussion
PRIMARY PERIODONTAL
LESION
The sensitivity of the proprioceptive fibres in
an inflamed periodontal ligament will help
identify the location of the pain
PRIMARY ENDO-
SECONDARY PERIO
Tender on percussion
PRIMARY PERIO-
SECONDARY ENDO
Tender on percussion
TRUE COMBINED LESION/
CONCOMITANT PULPAL
AND PERIODONTAL
LESIONS
Tender on percussion
PRIMARY ENDODONTIC
LESION
Fractured roots and recently traumatized teeth
often present high mobility
PRIMARY PERIODONTAL
LESION
Localized to generalized mobility
PRIMARY ENDO-
SECONDARY PERIO
Localized mobility
PRIMARY PERIO-
SECONDARY ENDO
Generalized mobility
TRUE COMBINED LESION/
CONCOMITANT PULPAL
AND PERIODONTAL
LESIONS
Generalized mobility with higher grade of
mobility related to the involved tooth
PRIMARY ENDODONTIC
LESION
A lingering response– irreversible pulpitis
No response– necrotic pulp (non-vital)
PRIMARY PERIODONTAL
LESION
The pulp is vital and responsive to testing
PRIMARY ENDO-
SECONDARY PERIO
Pulp vitality tests negative
PRIMARY PERIO-
SECONDARY ENDO
Pulp vitality may be positive in multirooted
teeth
TRUE COMBINED LESION/
CONCOMITANT PULPAL
AND PERIODONTAL
LESIONS
Usually negative because of non-vital pulp.
Vitality tests may give a positive response in
multirooted teeth
PRIMARY ENDODONTIC
LESION
A deep narrow solitary pocket in the absence
of periodontal disease may indicate the
presence of a lesion of endodontic origin or a
vertical root fracture
PRIMARY PERIODONTAL
LESION
Multiple wide and deep pockets
PRIMARY ENDO-
SECONDARY PERIO
Presence of solitary wide pocket but if
periodontal lesion is due to fracture of root
then solitary deep narrow pocket (mainly
localized)
PRIMARY PERIO-
SECONDARY ENDO
Presence of multiple wide and deep
periodontal pockets
TRUE COMBINED LESION/
CONCOMITANT PULPAL
AND PERIODONTAL
LESIONS
Probing reveals the typical conical periodontal
type of probing with the exception that at the
base of the periodontal lesion, the probe will
abruptly drop further down the lateral root
surface and may even extend to the apex of
the tooth
PRIMARY ENDODONTIC
LESION
Presence of deep carious lesions/extensive or
defective restorations/ previous poor RCT/
possible mishaps/root fracture/root resorption
with periapical radiolucency
PRIMARY PERIODONTAL
LESION
Vertical bone loss and more generalized than
to lesions of endodontic origin
Bone loss wider coronally
PRIMARY ENDO-
SECONDARY PERIO
Presence of deep carious lesions/extensive or
defective restorations/previous poor
RCT/diminution of the pulp canal
space/possible mishaps/root fractures/root
resorption with a wide base radiolucency
around the apex of the root
PRIMARY PERIO-
SECONDARY ENDO
Angular bone loss in multiple teeth with a wide
base coronally and narrow at the apex of the
root
TRUE COMBINED LESION/
CONCOMITANT PULPAL
AND PERIODONTAL
LESIONS
The radiographic appearance of combined
endodontic-periodontal disease may be similar
to that of a vertically fractured tooth
PRIMARY ENDODONTIC
LESION
A radiograph with gutta percha points to apex
or furcation area in molars
PRIMARY PERIODONTAL
LESION
Sinus tract mainly at the lateral aspect of the
root
PRIMARY ENDO-
SECONDARY PERIO
Sinus tract mainly at the apex of=r furcation
area
PRIMARY PERIO-
SECONDARY ENDO
Sinus tract mainly at the lateral aspect of the
root
TRUE COMBINED LESION/
CONCOMITANT PULPAL
AND PERIODONTAL
LESIONS
Difficult to trace out the origin of the lesion, if a
sinus tract is present, it maybe necessary to
raise a flap to determine the etiology of the
lesion
PRIMARY ENDODONTIC
LESION
Painful response at the time of chewing,
especially on releasing the biting pressure
PRIMARY PERIODONTAL
LESION
No symptoms
PRIMARY ENDO-
SECONDARY PERIO
Painful response at the time of chewing,
especially on releasing the biting pressure
PRIMARY PERIO-
SECONDARY ENDO
No symptoms
TRUE COMBINED LESION/
CONCOMITANT PULPAL
AND PERIODONTAL
LESIONS
Painful response at the time of chewing,
especially on releasing the biting pressure
Parolia A, Gait TC, Porto IC, Mala K.
Endo-perio lesion: A dilemma from 19 th
until 21 st century. Journal of
Interdisciplinary Dentistry. 2013 Jan
Hoyle P, Tank M, Modarres-Simmons S,
Storey CA. Assessment and management
of endo-periodontal lesions. Dental
Update. 2019 Nov 2;46(10):930-41.
 Identify occlusal trauma and eliminate– reduce risk of exacerbating inflamed periodontium
 C/F: fremitus test +ve on lateral/protrusive movements
 Splinting– indicated when not possible to eliminate such forces absolutely
- Also indicated for accurate occlusal adjustment (mobility)
 Primary endodontic disease– necrotic pulp– clinically seen with drainage from sulcus/sinus + swelling in
buccal attached gingiva
 Patient may be ASYMPTOMATIC
 In absence of adequate healing– further periodontal treatment is indicated.
Herrera D, Retamal‐Valdes B, Alonso B, Feres M. Acute periodontal lesions (periodontal abscesses and necrotizing periodontal diseases) and endo‐periodontal lesions. J
Clin Periodontol 2018
 Combination treatment
 Endodontic therapy BEFORE periodontal therapy
TREATMENT
Endodontic treatment only- root canal
therapy
Bakland LK, Andreasen FM, Andreasen JO. Management of traumatized teeth. In: Walton RE, Torabinejad T, editors. Principles and Practice of Endodontics. 3rd ed. Philadelphia, PA: WB
Bakland LK, Andreasen FM, Andreasen JO. Management of traumatized teeth. In: Walton RE, Torabinejad T, editors. Principles and Practice of Endodontics. 3rd ed. Philadelphia, PA: WB
TREATMENT
1. Periodontal treatment only
2. SRP
3. Removal of poorly contoured restorations
and developmental grooves
Bakland LK, Andreasen FM, Andreasen JO. Management of traumatized teeth. In: Walton RE, Torabinejad T, editors. Principles and Practice of Endodontics. 3rd ed. Philadelphia, PA: WB
TREATMENT
1) Endodontic treatment, evaluate
2) Periodontal treatment– initially a close
procedure
3) Revaluation at 4-6 wks. post-treatmen
Bakland LK, Andreasen FM, Andreasen JO. Management of traumatized teeth. In: Walton RE, Torabinejad T, editors. Principles and Practice of Endodontics. 3rd ed. Philadelphia, PA: WB
Bakland LK, Andreasen FM, Andreasen JO. Management of traumatized teeth. In: Walton RE, Torabinejad T, editors. Principles and Practice of Endodontics. 3rd ed. Philadelphia, PA: WB
Bakland LK, Andreasen FM, Andreasen JO. Management of traumatized teeth. In: Walton RE, Torabinejad T, editors. Principles and Practice of Endodontics. 3rd ed. Philadelphia, PA: WB
2014
Periodontal considerations:
- Severe vertical bone loss involving
1 root
- Through-and-through furcation
destruction
- Proximity of adjacent roots-prevent
oral hygiene maintenance
- Root exposure- dehiscence
- GOOD PROGNOSIS observed
with proper occlusion, absence of
mobility, and healthy periodontal
condition with regular follow-up.
HEMISECTIONING
2016
Advocated the role of endotherapy
preceding periodontal treatment
Periodontal regenerative therapy of
furcation-involved teeth is MORE
COST EFFECTIVE than
extraction/implant.
Use of enamel matrix derivative>>
more effective in class 1 and 2
furca involved
ACCESS FLAP+ EMD
2019
Periodontal regenerative
procedures
Endodontic treatment before
definitive periodontal regenerative
procedures  SUPPORTS
PERIODONTAL HEALING in case
of apical involvement
Strict SPT program
DBBM+ 10% COLLAGEN
2020
Newer modality– few studies
Ozone– antimicrobial activity
Useful adjunct to conventional sequence
of endotherapy followed by periodontal
treatment
OZONE GAS
THANK YOU

ENDO-PERIO LESIONS

  • 4.
    PULP PERIODONTIUM Confusion &controversy Pathways of spread of infection Diagnosis, prognosis, treatment EMBRYONIC ANATOMIC FUNCTIONAL
  • 5.
    ENDO- PERIO LESION Lesionsdue to inflammatory products found in varying degrees in both periodontium and pulpal tissues.
  • 22.
    • Tooth withnecrotic pulp reaching apical periodontium • Root perforations • Root fractures • Chronic periapical abscess with sinus tract • Replants • Transplants • Teeth requiring hemisection • Occlusal trauma causing reversible pulpitis • Supra/infra bony pockets caused during periodontal treatment resulting in pulpal inflammation • Occlusal trauma and gingival inflammation resulting in pocket formation • Any lesion of type I which result in irreversible reaction to periodontium requiring periodontal treatment • Any lesion of type II which results in irreversible damage to pulp tissue requiring endodontic
  • 23.
    - Pulpal infectionmay cause a tissue destructive process that proceeds from the apical or furcal region of a tooth towards the gingival margin, as opposed to MARGINAL PERIODONTITIS in which infection spreads from the gingival margin toward the root apex.
  • 24.
    Endo-periodontal lesion withroot damage Root fracture or cracking Root canal or pulp chamber perforation External root resorption Endo-periodontal lesion without root damage Endo-periodontal lesion in periodontitis patients Grade 1- narrow deep periodontal pocket in 1 tooth surface Grade 2- wide deep periodontal pocket in 1 tooth surface Grade 3- deep periodontal pockets in more than 1 tooth surface Endo-periodontal lesion in non-periodontitis patients Grade 1- narrow deep periodontal pocket in 1 tooth surface Grade 2- wide deep periodontal pocket in 1 tooth surface
  • 26.
  • 30.
    Pulpal inflammation/necrosis Inflammatory responsein PDL Gingival sulcus of involved tooth Gingival sulcus of adjacent tooth Severe – destruction of supporting apparatus viz., PDL, socket, etc. Localized swelling Draining sinus tract Diffuse swelling Attached gingiva Alveolar mucosa
  • 31.
  • 37.
    Pathogenic bacteria & inflammatoryproducts of periodontal disease Accessory canals/ lateral canals/apical foramen Retrograde pulpitis
  • 38.
    Pulpal response –remaining dentin thickness Negligible response Repair & Healing Pulpal response Necrosis Prophylactic root canal treatment to be completed before periodontal treatment  SCALING & ROOT PLANING - REASONS: 1. Dissolved smear layer– - increased hydraulic conductance of involved dentinal tubules - decreased peripheral resistance to fluid flow across dentin 2. Open dentinal tubules– pathway for diffuse transport of bacterial elements in oral cavity to the pulp 3. Peripheral sensitization of pulp nociceptors due to release of inflammatory mediators as seen by A∂ fibers.  IN CONCLUSION, unless periodontal disease extends all the way to the tooth apex, the effect of periodontal disease treatment on dental pulp is ineffective
  • 40.
    ENDODONTIC LESION PERIODONTALLESION Pain: Acute, severe, throbbing type, poor localization, postural variation, referred to other sites, not much response to analgesics etc. Chronic onset. Mild to moderate in nature, able to localize pain, no postural variation, responds to mild analgesics etc. Swelling: Mucobuccal fold area, spreads to facial plane, extraoral swelling Attached gingiva, rarely spreads beyond MGJ, rarely facial swellings Mobility: Isolated tooth mobility if acute abscess Usually generalized mobility Periodontal probing: Precipitous drop in probing depth, lesion is narrow coronally than at the apex, no calculus Generalized deep probing, lesion is broader at its point of origin at the apex, calculus on the root Fistulation: Traced to radiographic apex with negative vitality Traced to mid-root Percussion: vertical Horizontal Pulp vitality: Nonvital Vital
  • 41.
    MOBILITY HISTORY PULP VITALITY VISUAL EXAMINATION PALPATIONPERIODONTAL PROBING RADIOGRAPH PERCUSSION SINUS TRACING, CRACKED TOOTH TEST (TRANSILLUMINATION, WEDGING), SELECTIVE ANESTHESIA TEST to localize pain to specific site
  • 42.
    PRIMARY ENDODONTIC LESION Soft tissue-presence of sinus opening Tooth- presence of decay/large restoration/ # restoration or tooth/ erosions/abrasions/cracks/discolorations/poor RCT PRIMARY PERIODONTAL LESION Inflamed gingiva/gingival recession around multiple teeth Accumulation of plaque and subgingival calculus around multiple teeth Intact teeth Presence of swelling indicating periodontal abscess PRIMARY ENDO- SECONDARY PERIO Plaque forms at the gingival margin of the sinus tract and leads to inflammation of marginal gingiva exudate Root perforation/fracture/misplaced post PRIMARY PERIO- SECONDARY ENDO Presence of plaque, subgingival calculus and swelling around multiple teeth Presence of pus, exudate Presence of localized/generalized gingival recession and exposure of root TRUE COMBINED LESION/ CONCOMITANT PULPAL Plaque, calculus and periodontitis will be present in varying degrees
  • 43.
    PRIMARY ENDODONTIC LESION Sharp Does notindicate whether inflammation is of periodontal/endodontic origin PRIMARY PERIODONTAL LESION Usually dull ache Sharp only in acute condition PRIMARY ENDO- SECONDARY PERIO Usually sharp shooting pain Dull ache in chronic conditions PRIMARY PERIO- SECONDARY ENDO Usually dull ache Sharp only in periodontal abscess TRUE COMBINED LESION/ CONCOMITANT PULPAL AND PERIODONTAL LESIONS Dull ache Severe pain only in acute conditions
  • 44.
    PRIMARY ENDODONTIC LESION Normally tenderon percussion PRIMARY PERIODONTAL LESION The sensitivity of the proprioceptive fibres in an inflamed periodontal ligament will help identify the location of the pain PRIMARY ENDO- SECONDARY PERIO Tender on percussion PRIMARY PERIO- SECONDARY ENDO Tender on percussion TRUE COMBINED LESION/ CONCOMITANT PULPAL AND PERIODONTAL LESIONS Tender on percussion
  • 45.
    PRIMARY ENDODONTIC LESION Fractured rootsand recently traumatized teeth often present high mobility PRIMARY PERIODONTAL LESION Localized to generalized mobility PRIMARY ENDO- SECONDARY PERIO Localized mobility PRIMARY PERIO- SECONDARY ENDO Generalized mobility TRUE COMBINED LESION/ CONCOMITANT PULPAL AND PERIODONTAL LESIONS Generalized mobility with higher grade of mobility related to the involved tooth
  • 46.
    PRIMARY ENDODONTIC LESION A lingeringresponse– irreversible pulpitis No response– necrotic pulp (non-vital) PRIMARY PERIODONTAL LESION The pulp is vital and responsive to testing PRIMARY ENDO- SECONDARY PERIO Pulp vitality tests negative PRIMARY PERIO- SECONDARY ENDO Pulp vitality may be positive in multirooted teeth TRUE COMBINED LESION/ CONCOMITANT PULPAL AND PERIODONTAL LESIONS Usually negative because of non-vital pulp. Vitality tests may give a positive response in multirooted teeth
  • 47.
    PRIMARY ENDODONTIC LESION A deepnarrow solitary pocket in the absence of periodontal disease may indicate the presence of a lesion of endodontic origin or a vertical root fracture PRIMARY PERIODONTAL LESION Multiple wide and deep pockets PRIMARY ENDO- SECONDARY PERIO Presence of solitary wide pocket but if periodontal lesion is due to fracture of root then solitary deep narrow pocket (mainly localized) PRIMARY PERIO- SECONDARY ENDO Presence of multiple wide and deep periodontal pockets TRUE COMBINED LESION/ CONCOMITANT PULPAL AND PERIODONTAL LESIONS Probing reveals the typical conical periodontal type of probing with the exception that at the base of the periodontal lesion, the probe will abruptly drop further down the lateral root surface and may even extend to the apex of the tooth
  • 48.
    PRIMARY ENDODONTIC LESION Presence ofdeep carious lesions/extensive or defective restorations/ previous poor RCT/ possible mishaps/root fracture/root resorption with periapical radiolucency PRIMARY PERIODONTAL LESION Vertical bone loss and more generalized than to lesions of endodontic origin Bone loss wider coronally PRIMARY ENDO- SECONDARY PERIO Presence of deep carious lesions/extensive or defective restorations/previous poor RCT/diminution of the pulp canal space/possible mishaps/root fractures/root resorption with a wide base radiolucency around the apex of the root PRIMARY PERIO- SECONDARY ENDO Angular bone loss in multiple teeth with a wide base coronally and narrow at the apex of the root TRUE COMBINED LESION/ CONCOMITANT PULPAL AND PERIODONTAL LESIONS The radiographic appearance of combined endodontic-periodontal disease may be similar to that of a vertically fractured tooth
  • 49.
    PRIMARY ENDODONTIC LESION A radiographwith gutta percha points to apex or furcation area in molars PRIMARY PERIODONTAL LESION Sinus tract mainly at the lateral aspect of the root PRIMARY ENDO- SECONDARY PERIO Sinus tract mainly at the apex of=r furcation area PRIMARY PERIO- SECONDARY ENDO Sinus tract mainly at the lateral aspect of the root TRUE COMBINED LESION/ CONCOMITANT PULPAL AND PERIODONTAL LESIONS Difficult to trace out the origin of the lesion, if a sinus tract is present, it maybe necessary to raise a flap to determine the etiology of the lesion
  • 50.
    PRIMARY ENDODONTIC LESION Painful responseat the time of chewing, especially on releasing the biting pressure PRIMARY PERIODONTAL LESION No symptoms PRIMARY ENDO- SECONDARY PERIO Painful response at the time of chewing, especially on releasing the biting pressure PRIMARY PERIO- SECONDARY ENDO No symptoms TRUE COMBINED LESION/ CONCOMITANT PULPAL AND PERIODONTAL LESIONS Painful response at the time of chewing, especially on releasing the biting pressure
  • 51.
    Parolia A, GaitTC, Porto IC, Mala K. Endo-perio lesion: A dilemma from 19 th until 21 st century. Journal of Interdisciplinary Dentistry. 2013 Jan Hoyle P, Tank M, Modarres-Simmons S, Storey CA. Assessment and management of endo-periodontal lesions. Dental Update. 2019 Nov 2;46(10):930-41.
  • 52.
     Identify occlusaltrauma and eliminate– reduce risk of exacerbating inflamed periodontium  C/F: fremitus test +ve on lateral/protrusive movements  Splinting– indicated when not possible to eliminate such forces absolutely - Also indicated for accurate occlusal adjustment (mobility)  Primary endodontic disease– necrotic pulp– clinically seen with drainage from sulcus/sinus + swelling in buccal attached gingiva  Patient may be ASYMPTOMATIC  In absence of adequate healing– further periodontal treatment is indicated.
  • 54.
    Herrera D, Retamal‐ValdesB, Alonso B, Feres M. Acute periodontal lesions (periodontal abscesses and necrotizing periodontal diseases) and endo‐periodontal lesions. J Clin Periodontol 2018
  • 55.
     Combination treatment Endodontic therapy BEFORE periodontal therapy
  • 56.
    TREATMENT Endodontic treatment only-root canal therapy Bakland LK, Andreasen FM, Andreasen JO. Management of traumatized teeth. In: Walton RE, Torabinejad T, editors. Principles and Practice of Endodontics. 3rd ed. Philadelphia, PA: WB
  • 57.
    Bakland LK, AndreasenFM, Andreasen JO. Management of traumatized teeth. In: Walton RE, Torabinejad T, editors. Principles and Practice of Endodontics. 3rd ed. Philadelphia, PA: WB TREATMENT 1. Periodontal treatment only 2. SRP 3. Removal of poorly contoured restorations and developmental grooves
  • 58.
    Bakland LK, AndreasenFM, Andreasen JO. Management of traumatized teeth. In: Walton RE, Torabinejad T, editors. Principles and Practice of Endodontics. 3rd ed. Philadelphia, PA: WB TREATMENT 1) Endodontic treatment, evaluate 2) Periodontal treatment– initially a close procedure 3) Revaluation at 4-6 wks. post-treatmen
  • 59.
    Bakland LK, AndreasenFM, Andreasen JO. Management of traumatized teeth. In: Walton RE, Torabinejad T, editors. Principles and Practice of Endodontics. 3rd ed. Philadelphia, PA: WB
  • 60.
    Bakland LK, AndreasenFM, Andreasen JO. Management of traumatized teeth. In: Walton RE, Torabinejad T, editors. Principles and Practice of Endodontics. 3rd ed. Philadelphia, PA: WB
  • 61.
    Bakland LK, AndreasenFM, Andreasen JO. Management of traumatized teeth. In: Walton RE, Torabinejad T, editors. Principles and Practice of Endodontics. 3rd ed. Philadelphia, PA: WB
  • 63.
    2014 Periodontal considerations: - Severevertical bone loss involving 1 root - Through-and-through furcation destruction - Proximity of adjacent roots-prevent oral hygiene maintenance - Root exposure- dehiscence - GOOD PROGNOSIS observed with proper occlusion, absence of mobility, and healthy periodontal condition with regular follow-up. HEMISECTIONING
  • 64.
    2016 Advocated the roleof endotherapy preceding periodontal treatment Periodontal regenerative therapy of furcation-involved teeth is MORE COST EFFECTIVE than extraction/implant. Use of enamel matrix derivative>> more effective in class 1 and 2 furca involved ACCESS FLAP+ EMD
  • 65.
    2019 Periodontal regenerative procedures Endodontic treatmentbefore definitive periodontal regenerative procedures  SUPPORTS PERIODONTAL HEALING in case of apical involvement Strict SPT program DBBM+ 10% COLLAGEN
  • 66.
    2020 Newer modality– fewstudies Ozone– antimicrobial activity Useful adjunct to conventional sequence of endotherapy followed by periodontal treatment OZONE GAS
  • 69.