Every year children are involved in accidents or
born with conditions that require medical care not
always available by their local provider.
• Shriners Hospitals for Children® – Cincinnati
has experts in the field of burn care, pediatric
plastic reconstruction and rehabilitation
• Our goal is provide comprehensive services
regardless of the patient’s ability to pay
There are about 486,000 burn injuries per
year in the US that require medical
attention and 3,275 deaths
ABA 2016 fact sheet
Burn Injuries
40,000 of 486,000 injuries are hospitalized – 30,000
of those into the 128 burn centers
The other 4500 U.S. acute care hospitals average less
than 3 burn admissions/year
Place of occurrence: 73% home, 8% job, 5% street,
5% recreational, 9% other
More males than females burned – 2.5:1
ABA 2016 fact sheet
Etiology of Admitted Burns
• Fire & flame 43%
• Scalds 34%
• Hot object contact 9%
• Electrical 4%
• Chemical 3%
• Other 7%
ABA 2016 fact
sheet
• Protection
1st line of defense against infection
• Loss of Body Fluids
After a burn, large fluid loss occurs
• Temperature Regulation
Full thickness injuries destroy sweat glands
• Sensory
Pain, pressure, temperature, and touch
Change in sensory perception
Skin is largest organ in the body
• Vitamin D activation
Skin + sunlight activates vitamin D
Partial thickness burns reduce this ability and full
thickness burns result in complete loss of ability to
activate Vitamin D from the area burned
• A person’s Physical identity is closely tied to how
their skin looks
Self-image issues are common after a burn injury
Skin
• Infants (up to age 1) & children (up to age 15) have a greater surface
area per unit of body weight than adults.
• Results in greater contact with the environment & greater evaporative
water loss per unit of body weight than adults;
• Therefore infants & children need more fluid per unit of body weight
during resuscitation than adults.
Body Surface Area
• 2 layers
• Epidermis
• Outer layer
• Contains no blood
vessels
• Healing occurs
from the dermal
layer underneath
the epidermis
Anatomy of Skin
• Thicker than the
epidermis
• Contains blood vessels,
sensory nerves, hair
follicles, and sweat
glands
• Healing can occur as
long as there is some
dermis present after the
burn injury
Dermis
• When the whole
dermal layer is
destroyed from
the burn, the skin
can no longer
restore itself
• Beneath the
dermis is
subcutaneous fat,
muscles, tendons,
and bones
Dermis
Types of Burns
• Flames
• Scalds
• Chemicals
• Electrical/Lightning
Flames
• House fires
• Explosions – gas
• Gasoline
• Campfires
• Fireworks
Firework Injuries
• Consumption of fireworks has been increasing significantly
over recent years with relaxed laws
• In 2017, there were 8 deaths from fireworks and 12,900
injuries. Two-thirds happened around the July Fourth
holiday. The large majority of the injuries were burns.
• Children under 15 years of age experienced about 36
percent of the injuries, and males of all ages were involved
in 70 percent.
• Insurance Journal: July 2018
Scald Injuries
• Occur when the child has a hot liquid spilled on him
• How bad the burn will be depends on what the hot
liquid is.
Example: Water boils at 100C, but soup with chicken fat
will boil at a higher temperature so soup will burn
deeper faster
Scald Injuries
• Most scald injuries in children occur while
bathing
• Most common burn injury in children under 4
years of age
• 95% of scalds occur in the residence
Scald Injuries
• Time of contact and water temperature to cause a
burn
- 120 degrees – 5 minutes
- 130 degrees - 30 seconds
- 140 degrees - 5 seconds
- 160 degrees - instantaneous
• Young children and older adult may burn deeper
faster because their skin is often thinner
• Set hot water heaters appropriately
Accidental Scald Burns
 Splash marks present
 Irregular pattern of burn
 Run-down pattern
 Consistent history
Identifying Abuse and Neglect
• Verify burn history
• Assess burn pattern
• 60% of burn histories do not match the
pattern of injury
• The child is pre verbal so they are unable to
explain the injury
Patterns of Injury
Classic Abuse:
• Sock-glove like distribution
• No splash marks
• Symmetrical deep second or third degree
burns of the hands or feet
Chemicals
Most common types
•Alkalis 
Home & work: cleaning, hobbies
•Acids 
•Organic compounds – petroleum products
Treatment
• Protect yourself – use PPE
• Remove saturated clothing, jewelry, contacts
• Brush off powder agents
• Continuously irrigate area with copious amounts of water
* Neutralizing chemical contraindicated
Potential for heat generation
* Continue irrigation until pain
decreases or patient is evaluated at a
burn center
Treatment
• Support ABC’s. Chemical agents can
impact respiratory &/or circulatory status
• Establish IV access for large
chemical injuries
• If irrigating a large burn, avoid
hypothermia
• Identify agent if possible but do not delay
therapy until agent is identified
• Contact Poison Control Center if needed
Chemical Injuries to Eyes
• Alkalis are twice as common as acid injuries
• Alkalis bond to tissue protein: require prolonged irrigation
• Water or saline irrigation – begin at scene & continue until
seen by qualified professional
• Consult ophthalmologist
Anhydrous Ammonia
• Seen commonly in fertilizer or industrial refrigerant
• Used in the manufacture of methamphetamine
• Is a strong base with a penetrating odor – pH 12
• Skin blistering with exposure, eye irritant
• If fumes are inhaled  Increased secretions combined with
sloughed epithelium, necrotic debris, tissue edema & reactive
bronchospasm
Treat with copious irrigation to
wounds; intubate as necessary
may need
Tar Burns
• Tar creates a thermal injury, not a chemical one
• Bitumen compound not absorbed, not toxic
• Cool tar to stop the burning process
• Facilitate removal with use of a petroleum based ointment
or medically safe solvent to emulsify the tar
Electrical Injury: The Grand Masquerader
* Small surface injuries may be associated with devastating internal
injuries
* Many work related with
significant economic impact
Electrical Injuries
• Low-voltage <1,000 V
Localized to area surrounding
the area
•
• High-Voltage >1,000 V
Deep extension and underlying
tissue damage
The extent of the injury is dependent upon…
•Type of current
- Alternate Current (AC) – 120v, back & forth flow of current;
usually contact burn only
- Direct Current (DC)– flows in 1 direction; entrance and exit
points Usually high voltage
•Pathway of flow through body
•Local tissue resistance
•How long the body is in contact with the electrical source
Tissue Injury
• Bone & skin have high resistance
• Once through the skin, the electrical current flows through the
tissue under it, especially along bones creating more heat
• Heat damages the muscle around it
• Deep muscle injury may occur, including compartment
syndrome, even when superficial muscle appears normal
Tissue Resistance
Treatment
• Scene Safety
• Assess Entrance and Exit wounds
• Monitor for dysrhythmias
• May require increased fluids during
resuscitation to flush out myoglobin
• NaHCO3 may be considered if myoglobin is
present in urine
Lightning
• Over the last 10 yrs, caused average of 28 deaths/yr.
• The spectrum of burn injury varies widely from pt to pt.
• Many survivors suffer serious complications related to the
cardiac and neurological systems.
• Not always associated with deep burns as the current
generated from lightning often travels on the surface of the
body and not through it.
• The cutaneous burns are typically superficial presenting
what has been called a “splashed on” spidery pattern.
Burns From Other Sources
• Curling Iron
• Irons
• Grills
• Cigarettes
Suspect abuse if you notice several pattern burns
Frostbite
Injury due to intracellular water turning to ice and
extreme cold constricting, damaging, and blocking the
blood supply to exposed parts.
Treatment protocol involves re-warming and care in a
specialized treatment unit for wound care
•Determining the Severity of a Burn
• Burn severity is based on how much of the body
surface area is injured and the depth of the burn
• Skin thickness also affects burn depth
Young children and older adults tend to have
thinner skin, resulting in deeper injuries in these age
groups, even when the temperature is lower
Depth of Burn Injury
• Only involves the epidermis – also
called 1st degree burn
• The cells needed for re-growth
remain
• Caused by prolonged exposure to
low intensity heat or short exposure
to high intensity heat
• Flash burn
Superficial Thickness Wounds
• Reddened, Painful, Mild Swelling
• No Blisters
• Peeling occurs 2-3 days after the burn
• Heals Within 3-6 Days
• No Scarring
• Care: Lotion or aloe for comfort
• Acetaminophen or ibuprofen for mild pain
Superficial Thickness Wounds
• Involves the entire epidermis and varying depths
of the dermis – also called 2nd degree burn
• Called a superficial partial thickness or deep partial
thickness burn depending on how far down into
the dermis the injury goes
Partial Thickness Wounds
• Involves the upper third of the dermis
• Small vessels bringing blood to this area are damaged,
resulting in large amounts of plasma leaking, which is trapped
between the dermis and epidermis resulting in a blister
• Pink, Moist, Blisters, Swollen, Blanches
• Painful because nerve endings are exposed when the blisters
are broken
• Heals within 2-3 weeks with proper care
• No scarring, but pigmentation changes can result and be
permanent
Superficial Partial Thickness Injuries
43
Deep Partial Thickness
• Burn injury extends in into the lower 2/3 of the dermis
• Blisters don’t usually form because the layer of dead tissue
is thick, and sticks to the dermis underneath, so does not
easily lift off the surface to make the blister
• Wound is dryer than a superficial partial thickness injury
because fewer blood vessels are left that have not been
injured
• Often looks red with patches of white
• Blanching either occurs slowly or not at all
Deep Partial Thickness Injuries
• Moderate amounts of edema
• Less pain than with superficial burns because more of the
nerve endings have been destroyed
• Can convert to full thickness wounds if there is infection,
or poor blood flow to the area because there is further
tissue damage
• Heals within 2-6 weeks
• Skin grafting can reduce healing time and often
produces a better looking scar
• These burns will scar
• Massage and pressure garments will reduce thickness
and redness of scars
Deep Partial Thickness Injuries
• Burn injury destroys the entire epidermis and all of the
dermis, leaving no skin cells to regenerate. Also called a 3rd
degree burn
• Will not heal without grafting
• No pain
• No blanching
• Hard, dry leathery appearance called eschar forms from the
destroyed skin
• Eschar is dead tissue and must be removed before any
healing can occur
• If the eschar is not removed, patient can become
septic and even die from toxins given off by the eschar
Full Thickness Injuries
• Severe edema is present under the
eschar
• Circumferential injuries:
• Because of the leathery texture of
the eschar and the severe edema,
blood flow to and movement of
the area may be reduced
• Escharotomies or fasciotomies may
be needed to relieve pressure,
allowing normal blood flow
Full Thickness Injuries
• May be called a 4th degree
burn
• The burn injury damages
muscle, bone, and/or
tendons
• Looks black and sunken in
• All feeling is gone
• Amputation may be
needed when an extremity
is involved
Deep Full Thickness Injuries
• Affects all body systems
• Vascular changes
• Circulation to the burned skin is immediately
altered
• Inflammation that develops causes the blood
vessels near the wound to dilate and have
increased capillary permeability causing fluids to
leak into the interstitial space = “3rd spacing”
Initial Clinical Manifestations of Burn Injury
• Continuous leak of plasma from the vascular space
into the interstitial space
• Leads to loss of plasma and proteins which decreases
blood volume and decreases blood pressure
• This leakage occurs in surrounding tissues and may
cause edema even in areas not burned
• If TBSA is >25%, capillary leak and edema occurs in
both burned and unburned areas
• This fluid shift occurs within the first 12 hours and
continues for 36 hours after burn injury
Third Spacing
• Fluid shifts lead to fluid/electrolyte and acid/base
changes
• Hypovolemia-3rd spacing
• Metabolic acidosis-cell destruction
• Hyperkalemia (↑K)-cell destruction releases
large amounts of potassium (K)
• Hyponatremia (↓Na) - 3rd spacing
• Sodium stays in the body but it is in the
interstitial space, not the vascular system
Third Spacing con’t …
• Increased hemoglobin and hematocrit
• Vascular dehydration- the volume is outside the blood
vessels in the 3rd space
• Problem as it increases blood viscosity, decreases
general blood flow throughout the body and
increases tissue hypoxia
Hemoconcentration from Fluid Shift
• Capillary leak stops
• Amount of edema seen on
the outside is matched in
the airway and pulmonary
system
24-36 Hours After Injury
• Tachycardia
• Decreased cardiac output because of fluid shifts and hypovolemia
initially
• May improve with fluid resuscitation
• Proper resuscitation and administration of oxygen helps prevent
complications
Cardiac
• Direct lung injury from flames rarely
occurs
• Superheated air, steam, toxic fumes, and
smoke can cause respiratory injury
• Respiratory issues are the major
cause of death in burn patients and
much more likely to happen when
the injury occurred indoors e.g.
trailers
• Difficult to have inhalation injury
with an outdoor fire
Respiratory
• Inhalation injuries can occur in
upper airway, major airways, and
lung tissue
• Upper airway- inhaled smoke or
irritants cause edema and blocks the
trachea
• Inflammatory response results in edema
of throat and mouth with possible
airway obstruction
• Same level of edema seen on outside is
also in the airway
• Worsens with fluid resuscitation but it is
necessary
Respiratory
• Toxic irritants ( chemicals & gases) can damage the lung
tissue including alveoli and capillaries
• Leaking capillaries in the lungs can cause alveolar edema
which leads to decreased oxygen exchange
• The leaking fluids contain protein which forms fibrinous
membranes and leads to respiratory distress and
pulmonary failure
• Smoke and gases also slow the ciliary activity allowing
particles to enter the bronchi
• Lining of trachea and bronchi may slough at 48-72
hours after injury and obstruct the lower airway
Respiratory
Effects of Edema on Airway Resistance
in the Infant versus the Adult
Infant
Adult
Normal Edema 1mm Resistance
increase
Diameter
Decrease
16x
3x
75%
44%
• As respiratory failure can follow, endotracheal
intubation or tracheostomy is sometimes needed
to maintain an open airway
• Chest burns with eschar can restrict chest
movement
• Carbon monoxide poisoning
Respiratory
• Reduced cardiac output and fluid shifts decrease
blood flow to the GI tract
• Impairs gastric motility and stress can cause a
gastro-duodenal ulcer called a Curlings ulcer
• At worst, destroys sections of the intestine
• As peristalsis decreases an ileus may develop
• Secretions and gases collect in GI tract causing
abdominal distention
GI Changes
• Increased metabolism
• Oxygen use and caloric needs are high
• Caloric needs may double and cause catabolic
activity
• **Can remain elevated even months after all
wounds are healed
• Increases core body temperature:
Lose heat through burned areas and the body tries to
heat the body to maintain equilibrium, often resulting in
low-grade fever
Metabolic Changes
• Diuretic phase- fluid goes back from the interstitial space into
the vascular space
• Blood volume increases leading to ↑ blood flow to kidneys
and diuresis occurs
• Hyponatremia continues- now because the kidneys are
excreting the sodium
• Hypokalemia- potassium moving back into cells and excreted
in urine
• Sodium & potassium will need to be replaced by IV
• Protein continues to be lost from the wounds
• Metabolic acidosis due to ↑ metabolic rate & loss of
bicarbonate in the urine
48-72 Hours After Burn Injury
1. Safety
2. Stop the Burning Process
3. Primary Survey and Support
– Airway - maintain airway with cervical-spine control
– Breathing - assess and support (Oxygen)
– Circulation -assess, support, control external
hemorrhage, monitor ECG; begin fluid replacement
– Disability - evaluate neurologic status (AVPU Method-
alert, responds to verbal, pain, unresponsive)
– Exposure - Allow for detailed exam
Primary Survey of Burn Patient
Predisposing Factors
• Closed space
Physical Examination
• Facial burns, singed nasal hairs
• Mucosal edema of nose and mouth
• Carbonaceous (black) sputum
• Hoarseness, strider (laryngeal)
• Difficulty breathing, wheezing (small airway)
• Decreased level of consciousness
Airway & Breathing
Assessment for Inhalation Injury
• Non-Rebreather mask
• Bag-Valve Mask Assist – bag patient
• Intubation
Oxygen
• Assessment Findings
• Absent or poor respiratory effort
• Absent peripheral pulses (Central pulse is weak
or strong)
• Unconscious
• Elective vs. Emergent is preferred
Intubation
Carbon Monoxide
• Colorless Odorless Gas
• 200x Affinity for Hemoglobin
• Does not allow for O2 transport
Treatment for CO Poisoning is removal from source,
followed by 100% O2.
• Monitor Heart Rate, Pulses, Color, Capillary Refill, O2
level, bleeding
• IV’s – large bore IV (2 if a large burn) – through unburned
skin if possible but can use burned skin
• Pre-hospital amounts before TBSA is calculated:
5 yrs & younger: 125ml Lactated Ringers (LR)/hr
6-13 yrs: 250ml LR/hr
14 yrs & older: 500ml LR/hr
Circulation
Disability
• Level of consciousness: A burn injury, even 100%
will not render a victim unconscious
• If the patient is unconscious or incoherent, look for
a cause other than the burn.
• Heart Attack
• Anoxia
• Stroke
• Head Injury
• Pain levels
• Glascow scale; APVU to determine LOC
• Quick Head to Toe examination without clothes – strip
& flip
• Temperature control
• Clean dry dressings
Exposure
Exposure
Wet vs. Dry Dressing?
• Always place in dry clean dressings.
• Topicals antibiotics are not necessary if being
transferred to a burn facility
• Ice and cold is absolutely not applied! It causes
vasoconstriction to an area that already has poor blood
flow
One of the major functions of the skin is
temperature regulation. If the skin is damaged the
body cannot maintain this function.
Hypothermia can result.
Temperature Regulation
• Temperature Regulation in infants and children, is affected by
their relatively greater body surface area so body heat is lost
quickly. (Cover head also!)
• Heat generation by shivering is hard for children due to their
relatively small muscle mass.
• Temperature regulation in infants < 6 months old depends
less on shivering and more on the body’s metabolic processes
and the air temperature around them.
Temperature Regulation
• Increased fluid needs
• Clotting factors are inhibited
• End Organ Perfusion is decreased
• DEATH
Complications of Hypothermia
The secondary survey does not begin until the
primary survey is completed
Secondary Survey
• History, burning agent & location of injury, associated
trauma
• Medical history – AMPLET (allergies, medications,
previous illnesses, last meal, events related to injury,
tetanus & immunizations
• Accurate pre-burn patient weight
• Complete Head to Toe Evaluation
• Severity & depth of burn
Secondary Survey
• TBSA involved*
• Adjusted fluid rates after TBSA determined*
• Monitor fluid resuscitation & urine output*
• Obtain labs & x-rays as needed
• Pain & anxiety management*
• Psychosocial support
• Wound Care*
Secondary Survey

No px slides1-78

  • 2.
    Every year childrenare involved in accidents or born with conditions that require medical care not always available by their local provider.
  • 3.
    • Shriners Hospitalsfor Children® – Cincinnati has experts in the field of burn care, pediatric plastic reconstruction and rehabilitation • Our goal is provide comprehensive services regardless of the patient’s ability to pay
  • 4.
    There are about486,000 burn injuries per year in the US that require medical attention and 3,275 deaths ABA 2016 fact sheet
  • 5.
    Burn Injuries 40,000 of486,000 injuries are hospitalized – 30,000 of those into the 128 burn centers The other 4500 U.S. acute care hospitals average less than 3 burn admissions/year Place of occurrence: 73% home, 8% job, 5% street, 5% recreational, 9% other More males than females burned – 2.5:1 ABA 2016 fact sheet
  • 6.
    Etiology of AdmittedBurns • Fire & flame 43% • Scalds 34% • Hot object contact 9% • Electrical 4% • Chemical 3% • Other 7% ABA 2016 fact sheet
  • 7.
    • Protection 1st lineof defense against infection • Loss of Body Fluids After a burn, large fluid loss occurs • Temperature Regulation Full thickness injuries destroy sweat glands • Sensory Pain, pressure, temperature, and touch Change in sensory perception Skin is largest organ in the body
  • 8.
    • Vitamin Dactivation Skin + sunlight activates vitamin D Partial thickness burns reduce this ability and full thickness burns result in complete loss of ability to activate Vitamin D from the area burned • A person’s Physical identity is closely tied to how their skin looks Self-image issues are common after a burn injury Skin
  • 9.
    • Infants (upto age 1) & children (up to age 15) have a greater surface area per unit of body weight than adults. • Results in greater contact with the environment & greater evaporative water loss per unit of body weight than adults; • Therefore infants & children need more fluid per unit of body weight during resuscitation than adults. Body Surface Area
  • 10.
    • 2 layers •Epidermis • Outer layer • Contains no blood vessels • Healing occurs from the dermal layer underneath the epidermis Anatomy of Skin
  • 11.
    • Thicker thanthe epidermis • Contains blood vessels, sensory nerves, hair follicles, and sweat glands • Healing can occur as long as there is some dermis present after the burn injury Dermis
  • 12.
    • When thewhole dermal layer is destroyed from the burn, the skin can no longer restore itself • Beneath the dermis is subcutaneous fat, muscles, tendons, and bones Dermis
  • 13.
    Types of Burns •Flames • Scalds • Chemicals • Electrical/Lightning
  • 14.
    Flames • House fires •Explosions – gas • Gasoline • Campfires • Fireworks
  • 15.
    Firework Injuries • Consumptionof fireworks has been increasing significantly over recent years with relaxed laws • In 2017, there were 8 deaths from fireworks and 12,900 injuries. Two-thirds happened around the July Fourth holiday. The large majority of the injuries were burns. • Children under 15 years of age experienced about 36 percent of the injuries, and males of all ages were involved in 70 percent. • Insurance Journal: July 2018
  • 16.
    Scald Injuries • Occurwhen the child has a hot liquid spilled on him • How bad the burn will be depends on what the hot liquid is. Example: Water boils at 100C, but soup with chicken fat will boil at a higher temperature so soup will burn deeper faster
  • 17.
    Scald Injuries • Mostscald injuries in children occur while bathing • Most common burn injury in children under 4 years of age • 95% of scalds occur in the residence
  • 18.
    Scald Injuries • Timeof contact and water temperature to cause a burn - 120 degrees – 5 minutes - 130 degrees - 30 seconds - 140 degrees - 5 seconds - 160 degrees - instantaneous • Young children and older adult may burn deeper faster because their skin is often thinner • Set hot water heaters appropriately
  • 19.
    Accidental Scald Burns Splash marks present  Irregular pattern of burn  Run-down pattern  Consistent history
  • 20.
    Identifying Abuse andNeglect • Verify burn history • Assess burn pattern • 60% of burn histories do not match the pattern of injury • The child is pre verbal so they are unable to explain the injury
  • 21.
    Patterns of Injury ClassicAbuse: • Sock-glove like distribution • No splash marks • Symmetrical deep second or third degree burns of the hands or feet
  • 22.
    Chemicals Most common types •Alkalis Home & work: cleaning, hobbies •Acids  •Organic compounds – petroleum products
  • 23.
    Treatment • Protect yourself– use PPE • Remove saturated clothing, jewelry, contacts • Brush off powder agents • Continuously irrigate area with copious amounts of water * Neutralizing chemical contraindicated Potential for heat generation * Continue irrigation until pain decreases or patient is evaluated at a burn center
  • 24.
    Treatment • Support ABC’s.Chemical agents can impact respiratory &/or circulatory status • Establish IV access for large chemical injuries • If irrigating a large burn, avoid hypothermia • Identify agent if possible but do not delay therapy until agent is identified • Contact Poison Control Center if needed
  • 25.
    Chemical Injuries toEyes • Alkalis are twice as common as acid injuries • Alkalis bond to tissue protein: require prolonged irrigation • Water or saline irrigation – begin at scene & continue until seen by qualified professional • Consult ophthalmologist
  • 26.
    Anhydrous Ammonia • Seencommonly in fertilizer or industrial refrigerant • Used in the manufacture of methamphetamine • Is a strong base with a penetrating odor – pH 12 • Skin blistering with exposure, eye irritant • If fumes are inhaled  Increased secretions combined with sloughed epithelium, necrotic debris, tissue edema & reactive bronchospasm Treat with copious irrigation to wounds; intubate as necessary may need
  • 27.
    Tar Burns • Tarcreates a thermal injury, not a chemical one • Bitumen compound not absorbed, not toxic • Cool tar to stop the burning process • Facilitate removal with use of a petroleum based ointment or medically safe solvent to emulsify the tar
  • 28.
    Electrical Injury: TheGrand Masquerader * Small surface injuries may be associated with devastating internal injuries * Many work related with significant economic impact
  • 29.
    Electrical Injuries • Low-voltage<1,000 V Localized to area surrounding the area • • High-Voltage >1,000 V Deep extension and underlying tissue damage
  • 30.
    The extent ofthe injury is dependent upon… •Type of current - Alternate Current (AC) – 120v, back & forth flow of current; usually contact burn only - Direct Current (DC)– flows in 1 direction; entrance and exit points Usually high voltage •Pathway of flow through body •Local tissue resistance •How long the body is in contact with the electrical source Tissue Injury
  • 31.
    • Bone &skin have high resistance • Once through the skin, the electrical current flows through the tissue under it, especially along bones creating more heat • Heat damages the muscle around it • Deep muscle injury may occur, including compartment syndrome, even when superficial muscle appears normal Tissue Resistance
  • 32.
    Treatment • Scene Safety •Assess Entrance and Exit wounds • Monitor for dysrhythmias • May require increased fluids during resuscitation to flush out myoglobin • NaHCO3 may be considered if myoglobin is present in urine
  • 33.
    Lightning • Over thelast 10 yrs, caused average of 28 deaths/yr. • The spectrum of burn injury varies widely from pt to pt. • Many survivors suffer serious complications related to the cardiac and neurological systems. • Not always associated with deep burns as the current generated from lightning often travels on the surface of the body and not through it. • The cutaneous burns are typically superficial presenting what has been called a “splashed on” spidery pattern.
  • 34.
    Burns From OtherSources • Curling Iron • Irons • Grills • Cigarettes Suspect abuse if you notice several pattern burns
  • 36.
    Frostbite Injury due tointracellular water turning to ice and extreme cold constricting, damaging, and blocking the blood supply to exposed parts. Treatment protocol involves re-warming and care in a specialized treatment unit for wound care
  • 37.
  • 38.
    • Burn severityis based on how much of the body surface area is injured and the depth of the burn • Skin thickness also affects burn depth Young children and older adults tend to have thinner skin, resulting in deeper injuries in these age groups, even when the temperature is lower Depth of Burn Injury
  • 39.
    • Only involvesthe epidermis – also called 1st degree burn • The cells needed for re-growth remain • Caused by prolonged exposure to low intensity heat or short exposure to high intensity heat • Flash burn Superficial Thickness Wounds
  • 40.
    • Reddened, Painful,Mild Swelling • No Blisters • Peeling occurs 2-3 days after the burn • Heals Within 3-6 Days • No Scarring • Care: Lotion or aloe for comfort • Acetaminophen or ibuprofen for mild pain Superficial Thickness Wounds
  • 41.
    • Involves theentire epidermis and varying depths of the dermis – also called 2nd degree burn • Called a superficial partial thickness or deep partial thickness burn depending on how far down into the dermis the injury goes Partial Thickness Wounds
  • 42.
    • Involves theupper third of the dermis • Small vessels bringing blood to this area are damaged, resulting in large amounts of plasma leaking, which is trapped between the dermis and epidermis resulting in a blister • Pink, Moist, Blisters, Swollen, Blanches • Painful because nerve endings are exposed when the blisters are broken • Heals within 2-3 weeks with proper care • No scarring, but pigmentation changes can result and be permanent Superficial Partial Thickness Injuries
  • 43.
  • 44.
    • Burn injuryextends in into the lower 2/3 of the dermis • Blisters don’t usually form because the layer of dead tissue is thick, and sticks to the dermis underneath, so does not easily lift off the surface to make the blister • Wound is dryer than a superficial partial thickness injury because fewer blood vessels are left that have not been injured • Often looks red with patches of white • Blanching either occurs slowly or not at all Deep Partial Thickness Injuries
  • 45.
    • Moderate amountsof edema • Less pain than with superficial burns because more of the nerve endings have been destroyed • Can convert to full thickness wounds if there is infection, or poor blood flow to the area because there is further tissue damage • Heals within 2-6 weeks • Skin grafting can reduce healing time and often produces a better looking scar • These burns will scar • Massage and pressure garments will reduce thickness and redness of scars Deep Partial Thickness Injuries
  • 46.
    • Burn injurydestroys the entire epidermis and all of the dermis, leaving no skin cells to regenerate. Also called a 3rd degree burn • Will not heal without grafting • No pain • No blanching • Hard, dry leathery appearance called eschar forms from the destroyed skin • Eschar is dead tissue and must be removed before any healing can occur • If the eschar is not removed, patient can become septic and even die from toxins given off by the eschar Full Thickness Injuries
  • 47.
    • Severe edemais present under the eschar • Circumferential injuries: • Because of the leathery texture of the eschar and the severe edema, blood flow to and movement of the area may be reduced • Escharotomies or fasciotomies may be needed to relieve pressure, allowing normal blood flow Full Thickness Injuries
  • 48.
    • May becalled a 4th degree burn • The burn injury damages muscle, bone, and/or tendons • Looks black and sunken in • All feeling is gone • Amputation may be needed when an extremity is involved Deep Full Thickness Injuries
  • 50.
    • Affects allbody systems • Vascular changes • Circulation to the burned skin is immediately altered • Inflammation that develops causes the blood vessels near the wound to dilate and have increased capillary permeability causing fluids to leak into the interstitial space = “3rd spacing” Initial Clinical Manifestations of Burn Injury
  • 51.
    • Continuous leakof plasma from the vascular space into the interstitial space • Leads to loss of plasma and proteins which decreases blood volume and decreases blood pressure • This leakage occurs in surrounding tissues and may cause edema even in areas not burned • If TBSA is >25%, capillary leak and edema occurs in both burned and unburned areas • This fluid shift occurs within the first 12 hours and continues for 36 hours after burn injury Third Spacing
  • 52.
    • Fluid shiftslead to fluid/electrolyte and acid/base changes • Hypovolemia-3rd spacing • Metabolic acidosis-cell destruction • Hyperkalemia (↑K)-cell destruction releases large amounts of potassium (K) • Hyponatremia (↓Na) - 3rd spacing • Sodium stays in the body but it is in the interstitial space, not the vascular system Third Spacing con’t …
  • 53.
    • Increased hemoglobinand hematocrit • Vascular dehydration- the volume is outside the blood vessels in the 3rd space • Problem as it increases blood viscosity, decreases general blood flow throughout the body and increases tissue hypoxia Hemoconcentration from Fluid Shift
  • 54.
    • Capillary leakstops • Amount of edema seen on the outside is matched in the airway and pulmonary system 24-36 Hours After Injury
  • 55.
    • Tachycardia • Decreasedcardiac output because of fluid shifts and hypovolemia initially • May improve with fluid resuscitation • Proper resuscitation and administration of oxygen helps prevent complications Cardiac
  • 56.
    • Direct lunginjury from flames rarely occurs • Superheated air, steam, toxic fumes, and smoke can cause respiratory injury • Respiratory issues are the major cause of death in burn patients and much more likely to happen when the injury occurred indoors e.g. trailers • Difficult to have inhalation injury with an outdoor fire Respiratory
  • 57.
    • Inhalation injuriescan occur in upper airway, major airways, and lung tissue • Upper airway- inhaled smoke or irritants cause edema and blocks the trachea • Inflammatory response results in edema of throat and mouth with possible airway obstruction • Same level of edema seen on outside is also in the airway • Worsens with fluid resuscitation but it is necessary Respiratory
  • 58.
    • Toxic irritants( chemicals & gases) can damage the lung tissue including alveoli and capillaries • Leaking capillaries in the lungs can cause alveolar edema which leads to decreased oxygen exchange • The leaking fluids contain protein which forms fibrinous membranes and leads to respiratory distress and pulmonary failure • Smoke and gases also slow the ciliary activity allowing particles to enter the bronchi • Lining of trachea and bronchi may slough at 48-72 hours after injury and obstruct the lower airway Respiratory
  • 59.
    Effects of Edemaon Airway Resistance in the Infant versus the Adult Infant Adult Normal Edema 1mm Resistance increase Diameter Decrease 16x 3x 75% 44%
  • 60.
    • As respiratoryfailure can follow, endotracheal intubation or tracheostomy is sometimes needed to maintain an open airway • Chest burns with eschar can restrict chest movement • Carbon monoxide poisoning Respiratory
  • 61.
    • Reduced cardiacoutput and fluid shifts decrease blood flow to the GI tract • Impairs gastric motility and stress can cause a gastro-duodenal ulcer called a Curlings ulcer • At worst, destroys sections of the intestine • As peristalsis decreases an ileus may develop • Secretions and gases collect in GI tract causing abdominal distention GI Changes
  • 62.
    • Increased metabolism •Oxygen use and caloric needs are high • Caloric needs may double and cause catabolic activity • **Can remain elevated even months after all wounds are healed • Increases core body temperature: Lose heat through burned areas and the body tries to heat the body to maintain equilibrium, often resulting in low-grade fever Metabolic Changes
  • 63.
    • Diuretic phase-fluid goes back from the interstitial space into the vascular space • Blood volume increases leading to ↑ blood flow to kidneys and diuresis occurs • Hyponatremia continues- now because the kidneys are excreting the sodium • Hypokalemia- potassium moving back into cells and excreted in urine • Sodium & potassium will need to be replaced by IV • Protein continues to be lost from the wounds • Metabolic acidosis due to ↑ metabolic rate & loss of bicarbonate in the urine 48-72 Hours After Burn Injury
  • 64.
    1. Safety 2. Stopthe Burning Process 3. Primary Survey and Support – Airway - maintain airway with cervical-spine control – Breathing - assess and support (Oxygen) – Circulation -assess, support, control external hemorrhage, monitor ECG; begin fluid replacement – Disability - evaluate neurologic status (AVPU Method- alert, responds to verbal, pain, unresponsive) – Exposure - Allow for detailed exam Primary Survey of Burn Patient
  • 65.
    Predisposing Factors • Closedspace Physical Examination • Facial burns, singed nasal hairs • Mucosal edema of nose and mouth • Carbonaceous (black) sputum • Hoarseness, strider (laryngeal) • Difficulty breathing, wheezing (small airway) • Decreased level of consciousness Airway & Breathing Assessment for Inhalation Injury
  • 66.
    • Non-Rebreather mask •Bag-Valve Mask Assist – bag patient • Intubation Oxygen
  • 67.
    • Assessment Findings •Absent or poor respiratory effort • Absent peripheral pulses (Central pulse is weak or strong) • Unconscious • Elective vs. Emergent is preferred Intubation
  • 68.
    Carbon Monoxide • ColorlessOdorless Gas • 200x Affinity for Hemoglobin • Does not allow for O2 transport Treatment for CO Poisoning is removal from source, followed by 100% O2.
  • 69.
    • Monitor HeartRate, Pulses, Color, Capillary Refill, O2 level, bleeding • IV’s – large bore IV (2 if a large burn) – through unburned skin if possible but can use burned skin • Pre-hospital amounts before TBSA is calculated: 5 yrs & younger: 125ml Lactated Ringers (LR)/hr 6-13 yrs: 250ml LR/hr 14 yrs & older: 500ml LR/hr Circulation
  • 70.
    Disability • Level ofconsciousness: A burn injury, even 100% will not render a victim unconscious • If the patient is unconscious or incoherent, look for a cause other than the burn. • Heart Attack • Anoxia • Stroke • Head Injury • Pain levels • Glascow scale; APVU to determine LOC
  • 71.
    • Quick Headto Toe examination without clothes – strip & flip • Temperature control • Clean dry dressings Exposure
  • 72.
    Exposure Wet vs. DryDressing? • Always place in dry clean dressings. • Topicals antibiotics are not necessary if being transferred to a burn facility • Ice and cold is absolutely not applied! It causes vasoconstriction to an area that already has poor blood flow
  • 73.
    One of themajor functions of the skin is temperature regulation. If the skin is damaged the body cannot maintain this function. Hypothermia can result. Temperature Regulation
  • 74.
    • Temperature Regulationin infants and children, is affected by their relatively greater body surface area so body heat is lost quickly. (Cover head also!) • Heat generation by shivering is hard for children due to their relatively small muscle mass. • Temperature regulation in infants < 6 months old depends less on shivering and more on the body’s metabolic processes and the air temperature around them. Temperature Regulation
  • 75.
    • Increased fluidneeds • Clotting factors are inhibited • End Organ Perfusion is decreased • DEATH Complications of Hypothermia
  • 76.
    The secondary surveydoes not begin until the primary survey is completed Secondary Survey
  • 77.
    • History, burningagent & location of injury, associated trauma • Medical history – AMPLET (allergies, medications, previous illnesses, last meal, events related to injury, tetanus & immunizations • Accurate pre-burn patient weight • Complete Head to Toe Evaluation • Severity & depth of burn Secondary Survey
  • 78.
    • TBSA involved* •Adjusted fluid rates after TBSA determined* • Monitor fluid resuscitation & urine output* • Obtain labs & x-rays as needed • Pain & anxiety management* • Psychosocial support • Wound Care* Secondary Survey