CHETAN

Bone growth is achieved by adding
newly synthesized bone to existing
bone by two mechanisms:
Endochondral ossification
Intramembranous ossification
Bone growth

 bone forms via a cartilaginous intermediate
 physis best reflects this process
 From 9 to 10 weeks' gestational age to skeletal
maturity at 15 to 17 years, they are responsible for
the longitudinal growth of bone
Endochondral
ossification

 Physeal injuries represent 15% to 30% of all fractures
in children.
 The incidence varies with age and has been reported
to peak in adolescents.
 Physeal injuries involving the phalanges account for
over 30% of all physeal fractures (wrist jt more
common)

Growth hormone increases the number of cells in
the physeal columns;
Thyroid hormone potentiates cytoplasmic
proliferation;
Oestrogens play an important role in triggering
physeal closure.
Hormonal effects upon
skeletal growth

zones
/resting zone



 Epiphyaseal vessels—supply germinal layer
 Metaphyseal vessels—supply central ¾ of physis
 Periosteal physis—supply peripheral
Types of epiphysis
 Pressure epiphysis
 Traction
 Atavistic
 aberrant
Blood supply of physis

 The first two zones have an abundant extracellular matrix
and, consequently, a great deal of mechanical integrity,
particularly in response to shear forces.
 The third layer, the hypertrophic zone, contains scant
extracellular matrix and is weaker. On the metaphyseal side
of the hypertrophic zone there is an area of provisional
calcification leading to the zone of enchondral ossification.
The calcification in these areas provides additional
resistance to shear.
 Thus, the area of the hypertrophic zone just above the area
of provisional calcification is the weakest area of the physis,
and it is here that most injuries to the physis occur

 It is a wedge-shaped group of germinal cells that is
continuous with the physis
 The zone of Ranvier consists of three cell types—
 Osteoblasts form the bony portion of the perichondral ring
at the metaphysis;
 chondrocytes contribute to latitudinal growth;
 fibroblasts circumscribe the zone and anchor it to
perichondrium above and below the growth plate.
Zone of Ranvier

 It is a fibrous structure that is continuous with the
fibroblasts of the zone of Ranvier and the periosteum
of the metaphysis.
 It provides strong mechanical support for the bone–
cartilage junction of the growth plate
Perichondrial Ring
(LaCroix)


Epiphyseal blood supply
by Dale and Harris
Type A,
The epiphysis is nearly entirely covered by
articular cartilage.
Consequently, the blood supply traverses the
metaphysis and may be damaged on
separation of the metaphysis and epiphysis.
Type B,
The epiphysis is only partially covered by
articular cartilage.
Because the blood supply enters through the
epiphysis, separation of the metaphysis and
epiphysis will not compromise the blood
supply to the germinal layer
Proximal femur
Proximal
humerus
Distal femur
Proximal & distal
tibia
Distal radius

 The most frequent mechanism of injury is fracture
 M C, fracture injury is direct, with the fracture pattern
involving the physis itself. Occasionally, physeal injury from
trauma is indirect and associated with a fracture elsewhere in
the limb segment, either as a result of ischemia or perhaps
compression
 infection, disruption by tumour, cysts, tumour-like disorders,
vascular insult, repetitive stress, irradiation, and other rare
etiologies
Etiology

 Long bone osteomyelitis or septic arthritis
(particularly of theshoulder, hip, and knee) can cause
physeal damage resulting in either physeal growth
disturbance or frank growth arrest

 Partial or complete growth arrests can occur from a
pure vascular injury to an extremity.
 Salter–Harris type V injuries; the most common
location for this is the tibial tubercle after femoral
shaft or distal femoral physeal fractures
Vascular Insult

EPIDEMIOLOGY
 SH2 MC
 PHALNGES 44%, DISTAL RADIUS 18%,DISTAL
TIBIA11%
 MALE:FEMALE -2:1- 14YEARS:12YEARS
 occurred twice as often in the upper extremities as
in the lower extremities.

Salter and harris
 Based on the Radiographic appearance of fracture.
 The first three types were adopted from Poland
(types I, II, and III) and Aitken (Aitken type III
became Salter-Harris type IV)
 The higher the classification the more likely Is
physeal arrest or joint incongurity to occur

 Is a separation of the epiphysis from
the metaphysis occurring entirely
through the physis. It usually occurs
in the zone of hypertrophy (weakest)
 It is rare and seen most frequently in
infants or in pathologic fractures, such
as those secondary to rickets or
scurvy.
 Because the germinal layer remains
with the epiphysis, growth is not
disturbed unless the blood supply is
interrupted, as frequently occurs with
traumatic separation of the proximal
femoral epiphysis.
Phalenges
Metacarpals
 Radiographs of undisplaced type I physeal fractures, are
normal except for associated soft tissue swelling.
 type I fractures occurred most frequently in the phalanges,
metacarpals, distal tibia, and distal ulna.
 Epiphyseal separations in infants occur most commonlyin the
proximal humerus, distal humerus, and proximal femur
 Ultrasound is particularly helpful for assessing epiphyseal
separations in infants (especially in the proximal femur and
elbow regions) without the need for sedation

The fracture extends along the
hypertrophic zone of the physis and at
some point exits through the metaphysis.
The epiphyseal fragment contains the entire
germinal layer as well as a metaphyseal
fragment of varying size. This fragment is
known as Thurston Holland's sign.
The periosteum on the side of the
metaphyseal fragment is intact and
provides stability once the fracture is
reduced. Growth disturbance is rare
because the germinal layer remains intact.
distal radius
By def:- they cross the germinal layer and
are usually intra-articular.
Often ass with high-energy or
compression mechanisms of injury, which
imply greater potential disruption of the
physis & higher risk of subsequent growth
disturbance
Consequently, if displaced, they require
an anatomic reduction, which may need to
be achieved open
Tillaux #
distal
humerus

 Salter–Harris type III fractures begin in the epiphysis
as a fracture through the articular surface and extend
vertically toward the physis. The fracture then courses
peripherally through the physis.
 The articular surface is involved and the fracture line
involves the germinal and proliferative layers of the
physis.

Vertical shear # Extend from the metaphysis
across the physis and into the epiphysis.
Thus, the # crosses the germinal layer of
the physis and usually extends into the
joint. (articular)
This # pattern is frequent around the medial malleolus, Lateral condylar
fractures of the distal humerus (milch type 1) and intra-articular two-part
triplane fractures of the distal tibia
with displacement, may result in metaphyseal–epiphyseal
cross union there by causing growh disurbance
treatment principles include obtaining anatomic reduction and adequate
stabilization to restore the articular surface and prevent metaphyseal–
epiphyseal cross union

Is a crushing injury to the physis from a
pure compression force.
Those authors who have reported type V
injuries have noted a poor prognosis, with
almost universal growth disturbance
Eg:- Of such an injury is closure of the
tibial tubercle, often with the development
of recurvatum deformity of the proximal
tibia, after fractures of the femur or distal
femoral epiphysis
unrecognized on initial radiographs.
Undoubtedly, more sophisticated imaging of
injured extremities (such as with MRI) will
identify physeal injuries in the presence of normal
plain radiographs

 Injury to the perichondrial ring
Type 6 injury RANG

 Trauma to epiphysis (chondral to osteochondral)
 Isolated injury of the epiphyseal plate
Type 7 ogden

 # of metaphysis
 Isolated injury of the metaphysis with possible
impairement of enchondral ossification
Type 8

 Avulsion injury to periosteum
 which may impair intramembranous ossification
Type 9


 The higher the classification, the more likely is
physeal arrest or joint incongruity to occur.

Peterson

 Type I is a fracture of the metaphysis extending to
the physis. Types II to V are the equivalents of Salter-
Harris types I, II, III, and IV, respectively. Peterson
type VI is epiphyseal (and usually articular surface)
loss. Lawnmower injuries are a frequent mechanism
for type VI injuries
PETERSON 1
# METAPHYSIS
EXTENDING
INTO PHYSIS
P 2
META
+ PHYSIS
P 2
PHYSIS
P4
EPI+
PHYSIS
P5
META+
EPI+
PHYSIS
P6
PHYSIS
MISSING
SH2 SH1 SH3 SH4
POLAND 2 PO1 PO3,4
AITKEN 1 A2 A3

Salter-Harris I fracture of the distal
femur. (widening)

Displaced Salter-Harris II fracture of the distal femur (with the
Thurstan Holland fragment)

Salter-Harris II fracture of the
distal tibial epiphysis

CT scan -Salter-Harris III fracture of
distal anterolateral tibial epiphysis
(ie, Tillaux fracture).

Displaced Salter-Harris IV fracture of the proximal tibia
The lateral portion of the epiphysis and the medial
portion of the epiphysis are independently displaced

initial injury radiograph of ankle subjected to significant
compressive and inversion forces. minimally displaced
fractures of tibia and fibula with apparent maintenance
of distal tibial physeal architecture.

Follow-up radiograph - growth arrest secondary to Salter-
Harris V injury. Note the markedly asymmetric Park-
Harris growth recovery line, indicating that the lateral
portion of the growth plate continues to function and the
medial portion does not.

Mortise radiograph -The Salter-Harris VI pattern. In this
case, the radiograph indicates that it is quite likely that a
small portion of the peripheral medial physis (as well as
a small amount of adjacent epiphyseal and metaphyseal
bone) has been avulsed

Growth plate (physeal)
fractures. Radiographic
evidence of a pediatric stubbed
great toe.

 Xray of the injured limb in atleast 2 views
 Classification of injury types usually done by
radiograph
 Ct scans may clarify complex # patterns
 Mri may show considerably more physeal damage

Treatment

Generally all type 1 and type 2 # do well
with closed reduction
All type 3 &4 # should be treated by ORIF
regardless of the amount of displacement
In type V fractures, the cartilage cells of the physis are crushed,
and regardless of the form of treatment, growth disturbance can
occur.
A type V fracture usually is diagnosed only in retrospect when a
growth disturbance develops

General principles of Rx
 Most SH I and II injuries can be treated with closed
reduction and casting or splinting and then
reexamination in 7-10 days to evaluate maintenance
of the reduction.
 Displaced injuries:- require reduction (within 48
hours) because growth arrest is common after late
reduction.
Greater angular deformity can be tolerated in the
upper extremity than in the lower extremity,
More valgus deformity can be tolerated than varus,
More flexion deformity can be tolerated than
extension.

 More proximal deformities of the lower extremity (in
the hip) are better compensated for than distal
deformities (the knee and, least of all, the ankle).
Spontaneous correction of angular deformities is
greatest when the asymmetry is in the plane of flexion
or extension (ie, the plane of joint motion),
 Function often returns to normal unless the fracture
occurs near the end of growth.

younger the patient,:- more remodeling
potential, greater degrees of displacement are
acceptable. But have greater potential for
deformity.
A growth plate that requires higher energy to
cause failure tends to have a higher rate of
growth arrest. For instance, the distal femoral
and proximal tibial

 (SH III and IV)-require ORIF.
 Smooth pins should parallel physis in epiphysis or metaphysis,
avoid physis.
 Oblique application of pins across physis considered only
when satisfactory internal fixation is unattainable with
transverse fixation.
 Type V fractures - rarely diagnosed acutely, treatment often
delayed until formation of a bony bar across physis.
 A high level of clinical suspicion is necessary

Correct placement of
cannulated screws across
epiphysis and metaphysis

 Crossing the physis with any form of fixation should
be avoided if possible
 In type III and IV fractures the pins should cross the
epiphysis in the fractured areas
 In type II and IV fractures they should cross the
metaphysis and epiphysis rather than the physis if
possible.
 Small cannulated screws are well suited for these
fractures.

COMPLICATIONS
1)Growth acceleration
first 6-18 months after injury.
increased vascular response.
use of fixation devices that may stimulate
longitudinal growth.
 Treatment in adolescents may involve an
epiphysiodesis.
If more than 6 cm of correction is desired-
lengthening procedures for bilateral limb-length
equilibration.

2) Growth arrest
 Premature partial growth arrest is far more common and
can appear as peripheral or central closures. Complete
growth arrest is uncommon.
 angular deformities and limb-length discrepancies.
 Peripheral arrests are produced when (bone bar/bridge)
forms, connecting metaphysis to epiphysis, traversing the
physis. If bar is located medially, the normal physis
continues to grow laterally, producing a varus deformity.
Anterior bone bars - recurvatum deformity.

 Central growth arrests - tented lesions of physis and
epiphysis due to a central osseous tether with metaphysis,
resulting in physeal coning.
 Some longitudinal growth continues in patients with
growth retardation, though at a much slower rate; thus, a
progressive shortening of the limb occurs.
 Partial growth arrests may be visible on radiographs as
early as 3-4 months postinjury or may be delayed as long
as 18-24 months.
 Follow-up checks may be necessary for 1-2 years
postinjury to monitor physeal healing and growth
response.


Physeal injuries

  • 1.
  • 2.
     Bone growth isachieved by adding newly synthesized bone to existing bone by two mechanisms: Endochondral ossification Intramembranous ossification Bone growth
  • 3.
      bone formsvia a cartilaginous intermediate  physis best reflects this process  From 9 to 10 weeks' gestational age to skeletal maturity at 15 to 17 years, they are responsible for the longitudinal growth of bone Endochondral ossification
  • 4.
      Physeal injuriesrepresent 15% to 30% of all fractures in children.  The incidence varies with age and has been reported to peak in adolescents.  Physeal injuries involving the phalanges account for over 30% of all physeal fractures (wrist jt more common)
  • 5.
     Growth hormone increasesthe number of cells in the physeal columns; Thyroid hormone potentiates cytoplasmic proliferation; Oestrogens play an important role in triggering physeal closure. Hormonal effects upon skeletal growth
  • 6.
  • 7.
  • 8.
  • 9.
      Epiphyaseal vessels—supplygerminal layer  Metaphyseal vessels—supply central ¾ of physis  Periosteal physis—supply peripheral Types of epiphysis  Pressure epiphysis  Traction  Atavistic  aberrant Blood supply of physis
  • 10.
      The firsttwo zones have an abundant extracellular matrix and, consequently, a great deal of mechanical integrity, particularly in response to shear forces.  The third layer, the hypertrophic zone, contains scant extracellular matrix and is weaker. On the metaphyseal side of the hypertrophic zone there is an area of provisional calcification leading to the zone of enchondral ossification. The calcification in these areas provides additional resistance to shear.  Thus, the area of the hypertrophic zone just above the area of provisional calcification is the weakest area of the physis, and it is here that most injuries to the physis occur
  • 11.
      It isa wedge-shaped group of germinal cells that is continuous with the physis  The zone of Ranvier consists of three cell types—  Osteoblasts form the bony portion of the perichondral ring at the metaphysis;  chondrocytes contribute to latitudinal growth;  fibroblasts circumscribe the zone and anchor it to perichondrium above and below the growth plate. Zone of Ranvier
  • 12.
      It isa fibrous structure that is continuous with the fibroblasts of the zone of Ranvier and the periosteum of the metaphysis.  It provides strong mechanical support for the bone– cartilage junction of the growth plate Perichondrial Ring (LaCroix)
  • 13.
  • 14.
     Epiphyseal blood supply byDale and Harris Type A, The epiphysis is nearly entirely covered by articular cartilage. Consequently, the blood supply traverses the metaphysis and may be damaged on separation of the metaphysis and epiphysis. Type B, The epiphysis is only partially covered by articular cartilage. Because the blood supply enters through the epiphysis, separation of the metaphysis and epiphysis will not compromise the blood supply to the germinal layer Proximal femur Proximal humerus Distal femur Proximal & distal tibia Distal radius
  • 15.
      The mostfrequent mechanism of injury is fracture  M C, fracture injury is direct, with the fracture pattern involving the physis itself. Occasionally, physeal injury from trauma is indirect and associated with a fracture elsewhere in the limb segment, either as a result of ischemia or perhaps compression  infection, disruption by tumour, cysts, tumour-like disorders, vascular insult, repetitive stress, irradiation, and other rare etiologies Etiology
  • 16.
      Long boneosteomyelitis or septic arthritis (particularly of theshoulder, hip, and knee) can cause physeal damage resulting in either physeal growth disturbance or frank growth arrest
  • 17.
      Partial orcomplete growth arrests can occur from a pure vascular injury to an extremity.  Salter–Harris type V injuries; the most common location for this is the tibial tubercle after femoral shaft or distal femoral physeal fractures Vascular Insult
  • 18.
     EPIDEMIOLOGY  SH2 MC PHALNGES 44%, DISTAL RADIUS 18%,DISTAL TIBIA11%  MALE:FEMALE -2:1- 14YEARS:12YEARS  occurred twice as often in the upper extremities as in the lower extremities.
  • 19.
     Salter and harris Based on the Radiographic appearance of fracture.  The first three types were adopted from Poland (types I, II, and III) and Aitken (Aitken type III became Salter-Harris type IV)  The higher the classification the more likely Is physeal arrest or joint incongurity to occur
  • 20.
      Is aseparation of the epiphysis from the metaphysis occurring entirely through the physis. It usually occurs in the zone of hypertrophy (weakest)  It is rare and seen most frequently in infants or in pathologic fractures, such as those secondary to rickets or scurvy.  Because the germinal layer remains with the epiphysis, growth is not disturbed unless the blood supply is interrupted, as frequently occurs with traumatic separation of the proximal femoral epiphysis. Phalenges Metacarpals
  • 21.
     Radiographs ofundisplaced type I physeal fractures, are normal except for associated soft tissue swelling.  type I fractures occurred most frequently in the phalanges, metacarpals, distal tibia, and distal ulna.  Epiphyseal separations in infants occur most commonlyin the proximal humerus, distal humerus, and proximal femur  Ultrasound is particularly helpful for assessing epiphyseal separations in infants (especially in the proximal femur and elbow regions) without the need for sedation
  • 22.
     The fracture extendsalong the hypertrophic zone of the physis and at some point exits through the metaphysis. The epiphyseal fragment contains the entire germinal layer as well as a metaphyseal fragment of varying size. This fragment is known as Thurston Holland's sign. The periosteum on the side of the metaphyseal fragment is intact and provides stability once the fracture is reduced. Growth disturbance is rare because the germinal layer remains intact. distal radius
  • 23.
    By def:- theycross the germinal layer and are usually intra-articular. Often ass with high-energy or compression mechanisms of injury, which imply greater potential disruption of the physis & higher risk of subsequent growth disturbance Consequently, if displaced, they require an anatomic reduction, which may need to be achieved open Tillaux # distal humerus
  • 24.
      Salter–Harris typeIII fractures begin in the epiphysis as a fracture through the articular surface and extend vertically toward the physis. The fracture then courses peripherally through the physis.  The articular surface is involved and the fracture line involves the germinal and proliferative layers of the physis.
  • 25.
     Vertical shear #Extend from the metaphysis across the physis and into the epiphysis. Thus, the # crosses the germinal layer of the physis and usually extends into the joint. (articular) This # pattern is frequent around the medial malleolus, Lateral condylar fractures of the distal humerus (milch type 1) and intra-articular two-part triplane fractures of the distal tibia with displacement, may result in metaphyseal–epiphyseal cross union there by causing growh disurbance treatment principles include obtaining anatomic reduction and adequate stabilization to restore the articular surface and prevent metaphyseal– epiphyseal cross union
  • 26.
     Is a crushinginjury to the physis from a pure compression force. Those authors who have reported type V injuries have noted a poor prognosis, with almost universal growth disturbance Eg:- Of such an injury is closure of the tibial tubercle, often with the development of recurvatum deformity of the proximal tibia, after fractures of the femur or distal femoral epiphysis unrecognized on initial radiographs. Undoubtedly, more sophisticated imaging of injured extremities (such as with MRI) will identify physeal injuries in the presence of normal plain radiographs
  • 27.
      Injury tothe perichondrial ring Type 6 injury RANG
  • 28.
      Trauma toepiphysis (chondral to osteochondral)  Isolated injury of the epiphyseal plate Type 7 ogden
  • 29.
      # ofmetaphysis  Isolated injury of the metaphysis with possible impairement of enchondral ossification Type 8
  • 30.
      Avulsion injuryto periosteum  which may impair intramembranous ossification Type 9
  • 31.
  • 32.
      The higherthe classification, the more likely is physeal arrest or joint incongruity to occur.
  • 33.
  • 34.
      Type Iis a fracture of the metaphysis extending to the physis. Types II to V are the equivalents of Salter- Harris types I, II, III, and IV, respectively. Peterson type VI is epiphyseal (and usually articular surface) loss. Lawnmower injuries are a frequent mechanism for type VI injuries
  • 35.
    PETERSON 1 # METAPHYSIS EXTENDING INTOPHYSIS P 2 META + PHYSIS P 2 PHYSIS P4 EPI+ PHYSIS P5 META+ EPI+ PHYSIS P6 PHYSIS MISSING SH2 SH1 SH3 SH4 POLAND 2 PO1 PO3,4 AITKEN 1 A2 A3
  • 36.
     Salter-Harris I fractureof the distal femur. (widening)
  • 37.
     Displaced Salter-Harris IIfracture of the distal femur (with the Thurstan Holland fragment)
  • 38.
     Salter-Harris II fractureof the distal tibial epiphysis
  • 39.
     CT scan -Salter-HarrisIII fracture of distal anterolateral tibial epiphysis (ie, Tillaux fracture).
  • 40.
     Displaced Salter-Harris IVfracture of the proximal tibia The lateral portion of the epiphysis and the medial portion of the epiphysis are independently displaced
  • 41.
     initial injury radiographof ankle subjected to significant compressive and inversion forces. minimally displaced fractures of tibia and fibula with apparent maintenance of distal tibial physeal architecture.
  • 42.
     Follow-up radiograph -growth arrest secondary to Salter- Harris V injury. Note the markedly asymmetric Park- Harris growth recovery line, indicating that the lateral portion of the growth plate continues to function and the medial portion does not.
  • 43.
     Mortise radiograph -TheSalter-Harris VI pattern. In this case, the radiograph indicates that it is quite likely that a small portion of the peripheral medial physis (as well as a small amount of adjacent epiphyseal and metaphyseal bone) has been avulsed
  • 44.
     Growth plate (physeal) fractures.Radiographic evidence of a pediatric stubbed great toe.
  • 45.
      Xray ofthe injured limb in atleast 2 views  Classification of injury types usually done by radiograph  Ct scans may clarify complex # patterns  Mri may show considerably more physeal damage
  • 46.
  • 47.
     Generally all type1 and type 2 # do well with closed reduction All type 3 &4 # should be treated by ORIF regardless of the amount of displacement In type V fractures, the cartilage cells of the physis are crushed, and regardless of the form of treatment, growth disturbance can occur. A type V fracture usually is diagnosed only in retrospect when a growth disturbance develops
  • 48.
     General principles ofRx  Most SH I and II injuries can be treated with closed reduction and casting or splinting and then reexamination in 7-10 days to evaluate maintenance of the reduction.  Displaced injuries:- require reduction (within 48 hours) because growth arrest is common after late reduction.
  • 49.
    Greater angular deformitycan be tolerated in the upper extremity than in the lower extremity, More valgus deformity can be tolerated than varus, More flexion deformity can be tolerated than extension.
  • 50.
      More proximaldeformities of the lower extremity (in the hip) are better compensated for than distal deformities (the knee and, least of all, the ankle). Spontaneous correction of angular deformities is greatest when the asymmetry is in the plane of flexion or extension (ie, the plane of joint motion),  Function often returns to normal unless the fracture occurs near the end of growth.
  • 51.
     younger the patient,:-more remodeling potential, greater degrees of displacement are acceptable. But have greater potential for deformity. A growth plate that requires higher energy to cause failure tends to have a higher rate of growth arrest. For instance, the distal femoral and proximal tibial
  • 52.
      (SH IIIand IV)-require ORIF.  Smooth pins should parallel physis in epiphysis or metaphysis, avoid physis.  Oblique application of pins across physis considered only when satisfactory internal fixation is unattainable with transverse fixation.  Type V fractures - rarely diagnosed acutely, treatment often delayed until formation of a bony bar across physis.  A high level of clinical suspicion is necessary
  • 53.
     Correct placement of cannulatedscrews across epiphysis and metaphysis
  • 54.
      Crossing thephysis with any form of fixation should be avoided if possible  In type III and IV fractures the pins should cross the epiphysis in the fractured areas  In type II and IV fractures they should cross the metaphysis and epiphysis rather than the physis if possible.  Small cannulated screws are well suited for these fractures.
  • 55.
     COMPLICATIONS 1)Growth acceleration first 6-18months after injury. increased vascular response. use of fixation devices that may stimulate longitudinal growth.  Treatment in adolescents may involve an epiphysiodesis. If more than 6 cm of correction is desired- lengthening procedures for bilateral limb-length equilibration.
  • 56.
     2) Growth arrest Premature partial growth arrest is far more common and can appear as peripheral or central closures. Complete growth arrest is uncommon.  angular deformities and limb-length discrepancies.  Peripheral arrests are produced when (bone bar/bridge) forms, connecting metaphysis to epiphysis, traversing the physis. If bar is located medially, the normal physis continues to grow laterally, producing a varus deformity. Anterior bone bars - recurvatum deformity.
  • 57.
      Central growtharrests - tented lesions of physis and epiphysis due to a central osseous tether with metaphysis, resulting in physeal coning.  Some longitudinal growth continues in patients with growth retardation, though at a much slower rate; thus, a progressive shortening of the limb occurs.  Partial growth arrests may be visible on radiographs as early as 3-4 months postinjury or may be delayed as long as 18-24 months.  Follow-up checks may be necessary for 1-2 years postinjury to monitor physeal healing and growth response.
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