Pneumonia in equine
‫أ‬.‫د‬/‫غانم‬ ‫محمدي‬ ‫محمد‬
‫الباطنة‬ ‫األمراض‬ ‫أستاذ‬
‫الحيوان‬ ‫طب‬ ‫قسم‬ ‫ورئيس‬
1
‫البيطري‬ ‫الطب‬ ‫كلية‬–‫بنها‬ ‫جامعة‬
2
PNEUMONIA
 Definition :
 Pneumonia is inflammation of pulmonary
parenchyma usually accompanied by inflammation
of the bronchioles and often by pleurisy
3
4
Etiology
 (A) predisposing factors:
 predisposing factors weaken the defense
mechanisms of the animals e.g.
 1- Inclement weather.
 2- poor ventilated housing.
 3- stress of transportation.
 4- Stress of malnutrition.
5
 (B) infectious causes:
 a- Viral pneumonia
 1- Adenovirus pneumonia (in immune –
deficient foals).
 2- Equine herpes virus (older foals)
 3- Equine influenza virus (older foals)
 4- Equine viral arteritis (adult horses).
 5- Equine viral rhinopneumonitis (adult
horses).
6
 b- Bacterial pneumonia :
 1- E. coli, streptococcus equi,
Actinobacillus equuli which causes
newborn foal septicemia.
 2- Corynebacterium equi (older
foals)
 3- Strangles
 4- Glanders
 5- Pleuropneumonia due to
anaerobic bacteria.
7
 C- Mycotic pneumonia:
 1- Epizootic lymphangtitis
 2- pneumonic aspergillosis
 d- Verminous pneumonia:
 1-Dictyocaulus arnifeldi.
 2- Parascaris equorum.
8
Clinical findings
 1- rapid, shallow respiration is the cardinal
sign of early pneumonia.
 - Dyspnea occurring in the later stages when
much of the lung tissue is non – functional
 2- Cough which is:
 - Dry, frequent, hacking cough in interstitial
pneumonia.
 - Moist, painful cough in bronchopneumonia.
 3- Cyanosis: Not a common sign, occurs only
when large areas of the lung are affected.
9
 4- Nasal discharge: May or may not
present depending upon
 the amount of exudates present in
bronchioles and
 whether or not, there is accompanying
inflammation of the upper respiratory
tract.
 5- Abnormal breath odour: Decay –
putrid
 – Decay when there is a large
accumulation of inspissated pus.
 - Putrid when pulmonary gangrene is
present.
10
 6-Auscultation of the lunges
 In early congestive stage of bronchopneumonia
and interstitial pneumonia there is increased
breath sound (bronchial sound)
 - Crackles (moist rales) develop in
bronchopneumonia as bronchiolar exudation
increases.
 - Clear, harsh bronchial sounds are audible in
uncomplicated interstitial pneumonia.
 - Loud bronchial sound when complete
consolidation in either form occurs (
consolidation also causes increased audibility of
heart sounds).
 - Pleuritic friction rub in early stages when
pleurisy is also present, and muffling of
bronchial sounds in the late exudative stages.
11
12
13
Diagnosis
 (I) History
 (II) Clinical
 (III) Endoscopic examination
 (IV) Nasal swabs
 (V) Ultrasonography
14
Treatment outline
24
Hygienic
treatment
Medicated
treatment
Antibiotics BronchodilatorsAntiparasitic antihistaminics
Anti-inflammatory?
25
Treatment
 1- Isolation of affected animals (particularly if
infectious disease suspected)
 in warm, well ventilated, draft free place and
 provide with ample fresh water and
 light nourishing food
 parenteral feeding if animal does not eat
 2- The choice of antibacterial agent (antibiotic or
sulfonamide) based on culture and sensitivity
testing.
 Suitable antiparasitic agent if verminous
pneumonia suspected. (thiabenzole, piperazine
citrate)
26
3- The use of bronchodilator to improve
ventilation and oxygen exchange.
 The most commonly used agents are
aminophylline and theophylline .
 Sympathomimetic drugs such as
epinephrine is effective but little used
because of their short– term action.
 The beta-2 adrenergic receptor selective
bronchodilators, such ad clenbuterol, exert
a very beneficial effect.
27
 4- Antihistaminic e.g. Avil injection,
histacure , ….
 5- The use of corticosteroid as and anti-
inflammatory agent e.g. Dexamethazone,
Betamethazone.
29
ASPIRATION PNEUMONLA
(Drenching pneumonia, Inhalation
Pneumonia)
 Etiology :
 1- Careless drenching or passage of a stomach
tube during treatment for other illness.
 2- Following vomiting
 3- Rupture of pharyngeal abscess.
 4- Paralysis or obstruction of larynx, pharynx
and esophagus result in aspiration of foal of
water when attempting to swallow
 Following guttural pouch diseases
30
Clinical signs
1-If large quantities of fluid are aspirated death may
occur suddenly
2- If small quantities are aspirated, the outcome may
depend on the composition of the aspirated, material
e.g
 -With soluble such as chloral hydrate and magnesium
sulphate, very rapid absorption from the lung occurs.
 -With insoluble substances and vomitus, pneumonia
with toxaemia result, which is usually fatal in 48-72
hrs.
3- The severity of aspiration pneumonia depends largely
upon the bacteria which are introduced, causing in
many cases an acute gangrenous pneumonia.
31
Treatment
 As mentioned before with pneumonia.

Pneumonia in equine 2017

  • 1.
    Pneumonia in equine ‫أ‬.‫د‬/‫غانم‬‫محمدي‬ ‫محمد‬ ‫الباطنة‬ ‫األمراض‬ ‫أستاذ‬ ‫الحيوان‬ ‫طب‬ ‫قسم‬ ‫ورئيس‬ 1 ‫البيطري‬ ‫الطب‬ ‫كلية‬–‫بنها‬ ‫جامعة‬
  • 2.
    2 PNEUMONIA  Definition : Pneumonia is inflammation of pulmonary parenchyma usually accompanied by inflammation of the bronchioles and often by pleurisy
  • 3.
  • 4.
    4 Etiology  (A) predisposingfactors:  predisposing factors weaken the defense mechanisms of the animals e.g.  1- Inclement weather.  2- poor ventilated housing.  3- stress of transportation.  4- Stress of malnutrition.
  • 5.
    5  (B) infectiouscauses:  a- Viral pneumonia  1- Adenovirus pneumonia (in immune – deficient foals).  2- Equine herpes virus (older foals)  3- Equine influenza virus (older foals)  4- Equine viral arteritis (adult horses).  5- Equine viral rhinopneumonitis (adult horses).
  • 6.
    6  b- Bacterialpneumonia :  1- E. coli, streptococcus equi, Actinobacillus equuli which causes newborn foal septicemia.  2- Corynebacterium equi (older foals)  3- Strangles  4- Glanders  5- Pleuropneumonia due to anaerobic bacteria.
  • 7.
    7  C- Mycoticpneumonia:  1- Epizootic lymphangtitis  2- pneumonic aspergillosis  d- Verminous pneumonia:  1-Dictyocaulus arnifeldi.  2- Parascaris equorum.
  • 8.
    8 Clinical findings  1-rapid, shallow respiration is the cardinal sign of early pneumonia.  - Dyspnea occurring in the later stages when much of the lung tissue is non – functional  2- Cough which is:  - Dry, frequent, hacking cough in interstitial pneumonia.  - Moist, painful cough in bronchopneumonia.  3- Cyanosis: Not a common sign, occurs only when large areas of the lung are affected.
  • 9.
    9  4- Nasaldischarge: May or may not present depending upon  the amount of exudates present in bronchioles and  whether or not, there is accompanying inflammation of the upper respiratory tract.  5- Abnormal breath odour: Decay – putrid  – Decay when there is a large accumulation of inspissated pus.  - Putrid when pulmonary gangrene is present.
  • 10.
    10  6-Auscultation ofthe lunges  In early congestive stage of bronchopneumonia and interstitial pneumonia there is increased breath sound (bronchial sound)  - Crackles (moist rales) develop in bronchopneumonia as bronchiolar exudation increases.  - Clear, harsh bronchial sounds are audible in uncomplicated interstitial pneumonia.  - Loud bronchial sound when complete consolidation in either form occurs ( consolidation also causes increased audibility of heart sounds).  - Pleuritic friction rub in early stages when pleurisy is also present, and muffling of bronchial sounds in the late exudative stages.
  • 11.
  • 12.
  • 13.
    13 Diagnosis  (I) History (II) Clinical  (III) Endoscopic examination  (IV) Nasal swabs  (V) Ultrasonography
  • 14.
  • 15.
  • 16.
    25 Treatment  1- Isolationof affected animals (particularly if infectious disease suspected)  in warm, well ventilated, draft free place and  provide with ample fresh water and  light nourishing food  parenteral feeding if animal does not eat  2- The choice of antibacterial agent (antibiotic or sulfonamide) based on culture and sensitivity testing.  Suitable antiparasitic agent if verminous pneumonia suspected. (thiabenzole, piperazine citrate)
  • 17.
    26 3- The useof bronchodilator to improve ventilation and oxygen exchange.  The most commonly used agents are aminophylline and theophylline .  Sympathomimetic drugs such as epinephrine is effective but little used because of their short– term action.  The beta-2 adrenergic receptor selective bronchodilators, such ad clenbuterol, exert a very beneficial effect.
  • 18.
    27  4- Antihistaminice.g. Avil injection, histacure , ….  5- The use of corticosteroid as and anti- inflammatory agent e.g. Dexamethazone, Betamethazone.
  • 19.
    29 ASPIRATION PNEUMONLA (Drenching pneumonia,Inhalation Pneumonia)  Etiology :  1- Careless drenching or passage of a stomach tube during treatment for other illness.  2- Following vomiting  3- Rupture of pharyngeal abscess.  4- Paralysis or obstruction of larynx, pharynx and esophagus result in aspiration of foal of water when attempting to swallow  Following guttural pouch diseases
  • 20.
    30 Clinical signs 1-If largequantities of fluid are aspirated death may occur suddenly 2- If small quantities are aspirated, the outcome may depend on the composition of the aspirated, material e.g  -With soluble such as chloral hydrate and magnesium sulphate, very rapid absorption from the lung occurs.  -With insoluble substances and vomitus, pneumonia with toxaemia result, which is usually fatal in 48-72 hrs. 3- The severity of aspiration pneumonia depends largely upon the bacteria which are introduced, causing in many cases an acute gangrenous pneumonia.
  • 21.
    31 Treatment  As mentionedbefore with pneumonia.