Unit 8 neurotic stress and somatoform, PSYCHIATRIC NURSING
1. The document discusses various neurotic, stress-related and somatoform disorders including anxiety disorders, phobic disorders, obsessive compulsive disorder, and somatoform disorders.
2. It provides classifications of these disorders based on the ICD-10 system and describes key features, symptoms, etiologies, and treatment approaches for each disorder type.
3. Treatment typically involves a multimodal approach including psychotherapy, relaxation techniques, drug therapies like antidepressants, and in more severe cases of OCD, electroconvulsive therapy or psychosurgery may be used.
Unit 8 neurotic stress and somatoform, PSYCHIATRIC NURSING
1.
NURSING MANAGEMENT
OF PATIENTWITH
NEUROTIC, STRESS-
RELATED AND
SOMATOFORM DISORDERS
Prepared by:
Mr. Vipin Chandran
1
2.
The term “Neurosis”is derived from two greek
words, ‘Neuron’ means ‘nerve’ with the suffix ‘osis’
means ‘diseased’ or ‘abnormal condition’.
Majority of people are affected by neurosis in some
mild form or other, which may include physical
symptoms e.g anxiety, hysteria, phobia,
depression, obsessive compulsive tendencies.
NEUROSIS 2
Anxiety is a‘normal’ phenomenon which is
characterized by a state of apprehension or unease
arising out of anticipation of danger.
1.ANXIETY DISORDER: 5
6.
Types of anxiety:
1):Trait theory: this is habitual tendency to be
anxious in general (a trait). “I often feel anxious”
2): State anxiety: this is the anxiety felt at the
present. “I feel anxious now”
6
It is characterizedby an Insidious onset in third decade,
usually chronic course which may or may not be with
panic attacks (episodes of acute anxiety).
Symptoms should last for at least 6 months to diagnose
G.A.D.
GENERALIZED ANXIETY DISORDER
9
10.
It is characterizedby discrete episodes of acute anxiety
onset is in early third decade, with chronic course.
Panic attack occur recurrently every few days last for few
minutes & characterized by very severe anxiety.
PANIC DISORDER 10
11.
1. Psychodynamic Theory:
Anxietyis a signal that disturb internal
psychological equilibrium. This is called signal
anxiety.
It arouses ego to take defensive action
(Repression) primary. When it fails (Conversion,
Isolation) secondary.
ETIOLOGY: 11
12.
2. Behavioral Theory:
Accordingto this theory, Anxiety is an
unconditioned inherent response of organism to
painful or dangerous stimuli.
3. Cognitive Behavioral Theory:
There is evidence of selective information
processing (more attention paid to threat- related
information), cognitive distortions, Negative
automatic thoughts.
ETIOLOGY: 12
13.
4. Biological Theory:
a).Genetic evidence: 15-20% Ist degree relative
Monozygotic- 80%
Dizygotic- 20%
b). Chemically Induced:
GABA it is the most prevalent inhibitory
neurotransmitter in the CNS. Alteration in GABA
levels lead to production of Anxiety.
ETIOLOGY: 13
14.
Treatment of anxietydisorder is usually multimodal
1. Psychotherapy (Supportive & CBT etc)
2. Relaxation techniques (Exercise, Yoga, Pranayama,
Meditation)
3. Other behavior therapies (Biofeedback &
Hyperventilation control)
4. Drug treatment:
- Benzodiazepines (GAD)
- Antidepressant (Panic Disorder)
- Beta Blockers (Propranolol & atenolol)
- Buspirone (Anti-anxiety Drug)
TREATMENT: 14
15.
Phobia is definedas irrational fear of a specific
object, situation or activity, often leading to
persistent avoidance of the feared object, situation
or activity.
2. PHOBIC DISORDER: 15
16.
a): Presence offear of an object, situation or
activity.
b): Patient recognizes the fear as irrational &
unjustified
c): Patient is unable to control fear
d): It leads to persistent avoidance of a particular
object, situation or activity.
CHARACTERISTIC FEATURES: 16
17.
It is characterizedby an irrational fear of situations or
being in places away from the familiar setting of home. It
includes fear of open spaces, public spaces, crowded
places, where there is no escape.
AGORAPHOBIA: 17
18.
Irrational fear ofactivities or social interaction,
characterized by irrational fear of performing
activities in the presence of other people or
interacting with others.
SOCIAL PHOBIA: 18
19.
It is characterizedby an irrational fear of a
specified object or situation. Some of the example
of simple phobia include-
*Acrophobia
*Zoophobia
*Xenophobia
*Algophobia
*Claustrophobia
SPECIFIC PHOBIA: 19
20.
1. Psychodynamic Theory:
Anxietyis usually dealt with defense mechanism of
(Repression) when it fails, secondary defense
mechanism (Displacement) come into action.
2. Behavioral Theory:
It explains phobia as a conditioned reflex.
3. Biological Theory:
All phobias especially agoraphobia are closely
linked to panic disorders..
ETIOLOGY: 20
An obsession isdefined as:
1): An Idea, impulse or image which intrudes into the conscious
awareness repeatedly.
2): It is recognized as irrational & absurd. (Insight is present)
3): Patient tries to resist against it but is unable to.
3. OBSESSIVE COMPULSIVE DISORDER:
22
23.
Compulsion is definedas :
1): A form of behavior which usually follows
obsessions.
2): The behavior is not realistic and is either
irrational or excessive.
23
24.
In India, OCDis more common in married males,
while In other countries, no gender differences are
reported. Average onset is late third decade in
India.
EPIDEMIOLOGY: 24
25.
*ICD-10 Classifies OCDinto three clinical
subtypes:
1): Predominately Obsessive thoughts or ruminations.
2): Predominately Compulsive acts (Compulsive rituals).
3): Mixed Obsessional thoughts & acts.
* Depression is very common associated with OCD
CLINICAL SYMPTOMS: 25
Four clinical syndromehave been described :
4|. Primary Obsessive Slowness:
Relatively rare syndrome, characterized by
severe obsessive ideas & extensive compulsive
rituals.
29
1. Neurotransmitters:
Serotonin andNoradrenalin were found to have
higher level in brain.
2. Genetics:
It is transmitted genetically.
3. Electrophysiological Studies:
-Electroencephalography: Temporal lobes spikes
and increased theta waves
ETIOLOGY: 33
3]. BEHAVIORAL THEORIES35
From the learning theory perspective,
obsessions and compulsion are
understood as the result of interplay
of classical and operant conditioning
paradigms.
PHARMACOLOGICAL MANAGEMENT 37
1):Clomipramine (25 to 75 mg in divided doses)
2): Fluoxetine (Antidepressant)
3): Fluvoxamine (Significant in Obsessive-
Compulsive Symptoms)
38.
2]. BEHAVIOR THERAPY:38
Classical Behavioral Therapy techniques have been
used in treatment of OCD. These include:
1) Systematic desensitization
2) Flooding
3) Modeling
4) Response prevention
5) Negative practice
6) Implosion
7) Thought stopping
Somatoform disorders isdefined as the use of
physical symptoms to express emotional problems
and psychosocial stress. it is also known as
Hysteria or Briquet Syndrome.
4. SOMATOFORM DISORDERS:
41
42.
Its main featureis pattern of multiple, recurring
and significant physical complaints
Classification of Somatoform Disorders:
1) Somatization Disorder
2) Undifferentiated Somatoform
3) Hypochondriacal Disorders
4) Somatoform Autonomic Dysfunction
5) Persistent Somatoform Pain Disorder
6) Other Somatoform Disorder
7) Somatoform Disorder Unspecified
42
43.
It is characterizedby presence of recurrent and
multiple frequently changing somatic complaints
of several years duration for which medical
attention has been sought, but these apparently
are not due to any physical disorder.
1.SOMATIZATION DISORDER:
43
44.
Exact Etiology isnot known but probably due to:
1. Familial Factors:
It has been found that risk to develop disorder is
10-20% in female first- degree relatives.
2. Socio- Cultural Factors:
It has been documented that the tendency to
perceive and report distress in psychological or
somatic term is influenced by various social &
cultural factors including stigma.
ETIOLOGY: 44
45.
Symptoms may referto any part of the body:
- Gastrointestinal symptoms
(Abdominal pain, Bowel problems,
Nausea, Vomiting, Regurgitation etc)
- Pain in various body part
(extremities, back, joint etc)
- Conversion symptoms
(Pseudoseizures, Fainting,
Incoordination, loss of voice,
difficulty in swalloing)
CLINICAL SYMPTOMS: 45
Morrison has summarizedthe management of
somatization disorder in ABC as follows:
A. Accommodate initially
B. Behavior modification
C. Confrontation later about effects of behavior
D. Decrease drugs gradually
E. Educate about course and meaning of illness
F. Family involvement to give information
G. Guilt should be assuaged in physicians
H. Hospitalize
I. Intercurrent depression should be treated
conservatively.
47
48.
2. Undifferentiated SomatoformDisorder:
When physical complaint are multiple, varying and
persistent but the complete and typical clinical
picture of somatisation disorder is not fulfilled, this
category is to be considered.
3. Hypochondriacal Disorder:
Essential features is a persistent preoccupation
with the possibility of having one or more serious
& progressive physical disorders.
48
49.
4. Somatoform AutonomicDysfunction:
Patient present the symptoms as if they were due
to a physical disorder of a system or organ that is
largely or completely under Autonomic innervation
& control like. (Cardio-vascular, GI, Respiratory
systems, Genito Urinary etc)
5. Persistent Somatoform Pain Disorder:
Predominant complaint is of persistent, severe,
distressing pain which cannot be explained fully
by a physiological process or a physical disorder.
49
50.
6. Other SomatoformDisorder:
In these disorders the presenting complaints are
not mediated through the autonomic nervous
system and are limited to specific systems or parts
of the body.
50
51.
It is characterizedby a preoccupation with fear of having
or developing a serious physical illness. The fear is often
a result of unrealistic interpretation of physical signs or
sensations as evidence of disease.
5. HYPOCHONDRAISIS:
51
52.
There are threetheories of origin of hypochondriasis:
1. Psychodynamic theory:
Aggressive and Hostile wishes towards others are
transformed into physical complaints through
repression or displacement .
2. Socio- Cultural theory:
Sick role serves to convey about their distress and
disability to others, serving nonverbal
communication.
ETIOLOGY: 52
- Patient believesthat he has serious disease
- Pain
- Patient comes with a detailed
pathophysiological model
explaining his symptoms
CLINICAL SYMPTOMS: 54
55.
55
MANAGEMENT:
Hypochondraisis are oneof the most difficult patient to
treat. It can be managed by general physicians. But
patient “Doctor Shopping” Behavior also elicits negative
reaction from the treating physician.
Basic principles of treatment are as follows:
- Treatment by a single physician
- Supportive approach & regularly scheduled visit
- Avoidance of hospitalization, diagnostic procedures &
medications with abuse potential
- Focusing on symptoms & brief examination in initial visit.
56.
1. Flashbacks
2. Hypervigilance
3. Avoidance
4. Numbness
TERMINOLOGIES RELATED TO PTSD:
56
57.
PTSD is aset of reactions to an extreme stressor
such as intense fear, helplessness or horror that
leads individual to relieve the trauma.
5. POST- TRAUMATIC STRESS DISORDER
57
58.
-Episodes of repeatedrelieving of the trauma
(“Flashbacks”) or dreams.
- Flashbacks occurring (against persisting background
of a sense of “numbness” and emotional blunting)
- Detachment from other people
- Unresponsiveness to surroundings
- Anhedonia
CLINICAL SYMPTOMS: 58
59.
- 1-14% developPTSD from a few week to months
- Rarely exceeds 6 months
INCIDENCE & ONSET OF SYMPTOMS:
59
60.
- Evidence oftrauma
- Onset within 6 months of a traumatic event
- Repetitive intrusive recollection
- Day time imagery or dreams
- Conspicuous emotional detachment
- Numbing of feeling
DIAGNOSTIC CRITERIA: 60
61.
1. Stressor:
- Presenceof childhood trauma.
- Borderline, Paranoid
- Antisocial personality disorder
- Inadequate support system
- Recent stressful life changes
2. Psychodynamic factors:
* Cognitive model composites that affected person are
unable to process, rationalize the trauma that precipitated
the disorder
* Behavioral model has two phases:
First the trauma, Second instrumental learning
ETIOLOGY:
61
62.
3. Biological factors:
-Many neurotransmitter system have been responsible
for PTSD ( Nor epinephrine, Dopamine, Benzodiazepine
receptor and Hypothalamic- pituitary- adrenal axis)
ETIOLOGY:
62