WORM INFESTATION
Medicine and medical Nursing
Presenter :George masiye
5th
year students
Unza
Supervisor Mr P. Mwanza
GENERAL OBJECTIVE
• At the end of this lecture / discussion students
should be able demonstrate an understanding
of pin worms infestation
SPECIFIC OBJECTIVES:
• At the end of the lecture, student nurses
should be able to;
• Define pin worm infestation
• Describe the life cycle of the pin worms.
• Outline how pin worms are diagnosed
• Describe the treatment of pin worms.
• Outline the preventive measure of pin worms
INTRODUCTION
• Pin worm infestation occur world wide and is
most common among children 5 – 10 years old .
• In the USA it is estimated that there are more
than 40 million cases each year.
• Pinworm infections occur in all socioeconomic
groups; however, human-to-human spread is
favored by close, crowded living conditions
PINWORM INFESTATION
• Pinworm infestation is a type of human
intestinal infection by the pinworm.
• Pinworms are tiny, narrow worms that are
white about one cm long.
• Pinworms are also known as Enterobius
Vermiacularis Schull (2005)
LIFE CYCLE
• The cycle of infection begins with the ingestion
of the eggs.
• The eggs enter the small intestine until they
hatch into larvae and mature.
• The adult female pinworms moves into the
colon where they live.
• At night the female adult exit the body through
the anus and lay eggs in the folds of skin around
the anus.
• The presence of these eggs often causes
itching and irritation.
• When a person scratches the affected area,
the pinworm eggs are transferred to the
fingers.
• The eggs can survive for several hours on the
hands
• . If the infected person touches bedding,
clothing, toilet seats, toys, or other household
objects, the eggs are transferred to these
surfaces.
• Pinworm eggs can survive on these
contaminated surfaces for up to three weeks.
• The transfer of pinworm eggs is easier among
children, who may put infected toys or other
objects into their mouths.
• The eggs can also be transferred from
contaminated fingers to food or liquids.
• it’s also possible for adults to inhale eggs that
become airborne when bedding or clothing is
shaken.
Signs and symptoms
• frequent and strong itching of the anal area
• restless sleep due to the itching and
discomfort
• pain, rash, or other skin irritation around the
anus
• the presence of pinworms in the area of the
child’s anus
• the presence of pinworms in stools
diagnosis
• Finding eggs on the cellophane tape or scotch tape
placed on the skin around the anus.
• The laid eggs will stick to the tape.
• The tape is placed over night and check the
following morning.
• The tape is placed on the slide and examined
under the microscope.
• The pin worm eggs are seen.
DRUGS
• Mebendazole 100mg BD for 3 days.
• Aldendazole 400 mg single dose.
• Piperazine citrate 50 to 75mg per kg bwt OD
for 2 days.
• Lavemisole 150mg for adult and 5mg per kg
bwt for children
• Nitazoxanide 200 mg BD x 3 days
• Tribendimine 400 mg single dose
prevention
• Ensure infected person and family members
practice hand washing with warm water and
soap before eating.
• .
• Clean everyone’s fingernails and cut them
short.
• infected person and others to stop biting their
nails.
• Avoid shaking clothing and bedding to prevent
pinworm eggs from spreading into the air.
• Do not allow children to bathe together to
prevent spreading pinworm eggs in the bath
water.
• Encourage everyone to shower and change their
underwear every morning
• infected person to refrain from scratching the
anal area.
• Use hot water to launder all bedding, towels,
washcloths, and clothes in the affected house.
• Thoroughly clean any surfaces that may be
infected, including toys, floors, countertops,
and toilet seats.
NEXT IS
HOOKWORMS INFESTATION
SPECIFIC OBJECTIVES
• At the end of the lecture/discussion, the
students should be able to
• Define hookworm infestation
• Describe the life cycle of hookworms.
• Explain the signs and symptoms of hookworms
• Describe how hookworms are diagnosed.
• Mention the treatment of hookworms.
• Outline the prevention of hook worms
DEFINITION
• Hookworm infestation is the infection of the
small intestine with hook worms.(Scull 2005)
• There are two types of hookworms namely:
1. Necator Americanus
2. Ancylostoma Dueodenale
LIFE CYCLE OF HOOKWORMS
• The infective larvae penetrate the intact skin
especially the feet that are in contact with the
ground.
• The infective larvae are carried through the
bloodstream to the heart then to the lungs..
• The infective larvae penetrate the pulmonary
alveoli.
• The larvae enter the bronchial tree into the
trachea, the pharynx and are swallowed.
• The larvae go into the small intestine where
they mature and become adult worms.
• The adult hookworms attach to the wall of the
intestine with buccal capsure
–
• The adult male and female met and the female
lay eggs that come out in the faces.
• The eggs are passed into the soil and under
favourable condition hatch into infective larvae.
• The favourable condition includes moisture,
warm and shady environment.
SIGNS AND SYMPTOMS
• Ground itch itchiness and small rash caused
by an allergic reaction as the larvae enter the
skin.
• Diarrhoea
• Abdominal pain or colic
• Intestinal cramps
• Nausea
• Fever
• Blood in stool
• Loss of appetite
• Other signs are
• Anemia due to the worms feeding on blood
• Malnutrition due to the worms feed feeding
on the food that the person eats.
• Ascites due to protein deficiency.
diagnosis
• Stool examination
• Eggs in fresh stool less than 24hrs
• Larvae in old stool more than 24hrs
• Blood test
• Eosinophilia raised eosinophils level
Prevention of hookworms
• Using protective clothes like gumboots for the
farmers and fishermen
• Dress the children in shoes.
• Mass treatment of all the under 5 children
• Mass treatment of the people in the location
where many people are diagnosed with hook
worms
• Use of toilets for defecation to prevent soil
contamination
• Good hand hygiene after toilet and before
eating.
• Food hygiene that is washing all the fruits and
other raw foods before eating.
• Information education communication to the
community on sanitation
NEXT IS
ROUND WORMS
DEFINITION
• Round worm infestation is the infection of the
intestine by round worms.
• Round worm also called Ascaris Lumbricoides.
are the largest nematode parasitizing the
human intestine.
• Adult females: 20 to 35 cm; adult male: 15 to
30 cm.
Life cycle
• The infective eggs are swallowed goes into
the small intestine
• In the small intestine the eggs hatch into
larvae.
• The larvae penetrates the intestinal wall into
the portal vein and go to the liver.
• The larvae then goes to the lungs passing
through the heart.
• In the lungs the larvae mature after 10 to 14
days.
• The larvae penetrates the alveolar wall,
ascend the bronchial tree to the throat and
are swallowed.
• Upon reaching the small intestine, they
develop into adult worms.
• Between 2 and 3 months are required from
ingestion of the infective eggs to when the
adult female lay eges.
• Adult worms can live 1 to 2 years in the
lumen of the small intestine
• A female may produce approximately 200,000
eggs per day.
• Eggs are passed in the feces.
• Unfertilized eggs are not infective.
• Fertile embryonate eggs become infective
after 18 days to several weeks, depending on
the environmental conditions
Signs and stmptoms
• Pulmonary manifestation called Loeffler,s
syndrome includes:
• . mild to severe cough
• . dyspnoe
• .thoracic pain
• . fever
• Abdominal signs and symptoms.
• .Abdominal pain
• .Anorexia
• . Nausea
• .Diarrhoea
• .Heavy infestation leads to impaired food
nutrients absorption
diagnosis
• Stool examination
• Detection of eggs in faces-no concentration
technique is needed worm lay 200,000 eggs
per day.
• Full blood count
• There is significant eosinophilia during lung
passage
• x-ray of the intestine
• Show one or more worms .worms form a long
thin dark area if using barium contrast.
• Ultra sound of the pancreas biliary tract and
gall bladder show ectopic migration
COMPLICATION OF ROUND WORMS
• Intestinal obstruction
• numerous adult worms may form a ball and cause mechanical
intestinal obstruction.
•
• Biliary obstruction
• Migration into the biliary tract may lead to biliary obstruction
(cholestasis) with possibly infection (cholangitis, liver abscess).
•
• Pancreatic duct obstruction
• Migration into the pancreatic duct can cause
obstruction( pancreatitis)
• Appendicitis
• Sometimes there is migration to the appendix
with inflammation.
• Sometimes an adult Ascaris is present in vomitus.
• Occasionally, an adult can penetrate the lacrimal
duct.
Prevention of round worms
• Mass treatment of the under 5 children
• Mass treatment of the people in the location
where there man people are being diagnosed
with worm infestation
• Use of toilets for defecation to prevent soil
contamination
• Good hand hygiene after toilet and before
eating.
• Food hygiene that is washing all the fruits and
other raw foods before eating.
• Information education communication to the
community on sanitation
NEXT IS
• TAPE WORMS
• Tape worms are flat segmented worms that
live in the intestine of animals including man.
• The most common tape worms to man are
beef tape worms called taenia saginata and
pork tape worm called taenia solium.
• Man gets infected with tape worms by eating
raw or under cooked beef or pork from
infected animals.
BEEF TAPE WORMS(Taenia saginata)
• Cause taeniasis.
• The head ( scolex) has 4 hemispherical suckers.
The scolex imbeds in the mucosa of the intestine.
• The neck is the budding zone from which
segments are formed.
• The strobilus consists of immature, mature and
pregnant proglottides. Proglottides are segments
• There are hermaphroditic (there contain both
male and female reproductive organs in every
mature proglottid).
• Digestive tract is absent. Nutrition is absorbed
by the villi of the body surface.
• All adult worms infest the digestive tract of
mammals.
PORK TAPE WORM(taenia solium)
• The scolex(head) is global with 4 suckers and 1
rostellum
• The rostellum is for attaching to the mucosa
and the suckers are for sucking blood.
• The rostellum has 25 -50 hooklets for
attaching to the mucosa
PORK SCOLEX
LIFE CYCLE
• IN MAN
• infection is by ingesting of raw or under
cooked containing cystececus (pork and beef).
• In the intestine the cystececus develop into
adult tape worms after two months which can
survive for years.
• The adult worm attaches to the mucosa by the
scolex and reside in the small intestine
• The adult tape worm produce proglottids
which mature, become gravid and are passed
into stool.
• The eggs are released after the proglottids are
passed in stool.
• The taenia solium cystececus invades the
intestinal mucosa enter the bloodstream and
lodge in the brain, skin, tongue or eyes.
IN ANIMALS
• Cattle or pig get infected by ingesting eggs or
gravid proglottids during grazing.
• In the animal intestine the eggs hatch into
larvae (oncospheres).
• The oncospheres invades the intestinal wall,
migrate through blood stream to the muscles
where it encysts, forming cysticerci
Signs and symptoms
• Taeniasis
• Taeniasis is caused by the adult tape worm
residing in the small intestine.
• The adult worm irritates the small intestine
causing:
• Abdominal pain
• Anorexia
• Chronic indigestion
• Diarrhea
• Weight loss.
CYSTICERCOSIS
• Caused by cysticerci living in human tissue.
Cysticercosis is divided into three types
•
• Subcutaneous type:
• The subcutaneous nodules usually found on
the head, limbs, abdomen and back.
• Are movable and painless
• Ocular type
• The Cysticercus is usually found in the vitreous
body or subretina.
• Visual disturbance often occurs.
• The dead worms may provoke local
inflammation causing blindness.
• Brain type:
• The patients may manifest with headache, nausea,
vomiting, and epilepsy, and paralysis, weakness in
limbs, dizziness, and mental disorder.
• Epilepsy is the most frequent symptom of brain
cysticercosis.
Diagnosis
• 1.Taeniasis:
•
• Finding gravid proglottides or eggs in stool.
• 2. Cysticercosis:
• Biopsy of subcutaneous nodules,
• X-ray, CT or MR for brain type
• Ophthalmoscope examination:
• for ocular type.
TREATMENT
Taeniasis
• Praziquantel 5 -10 mg per kg body weight
single dose
• Niclosamide adults and children above 6
years give 2g single dose after light breakfast
followed after 2 hours by a laxative
Cysticercus
Excision - removal of cyst.
• Praziquantel - destroys Cysticercus in brain
• Metrifonate- for cutaneous type
• Corticosteroid reduce the inflammatory
process
PREVENTION
• Health education on how to cook meat
• Avoid eating raw pork.
• Avoid pigs eating human stool.
• Inspection of slaughter houses.
• Sanitary disposal of night soil.
• Improved meat inspection
• Improved meat processing
• Deworming all the pigs
SUMMARY
• Tape worms are contracted by ingestion of
cysticerci in the raw or under cooked meat .
Man pass proglottids which contain eggs in
stool.
• Pigs or cattle get infected by ingesting the
eggs, the larvae form cysticerci the muscle.
Tape worms are prevented by eating well
cooked meat
• REFERENCES
• Cook.G.C and Zumla (2003), Manson’s Tropical
Disease, 21st
Edition, Saunders, Elsevier
Sciences Division, 32 Jamestown Road London.
• Schull C.R (2005), Common Medical Problems in
the Tropics, 2nd
Edition macmillan Education
Between
•

WORM INFESTATION medicine POWER POINT.pptx

  • 1.
    WORM INFESTATION Medicine andmedical Nursing Presenter :George masiye 5th year students Unza Supervisor Mr P. Mwanza
  • 2.
    GENERAL OBJECTIVE • Atthe end of this lecture / discussion students should be able demonstrate an understanding of pin worms infestation
  • 3.
    SPECIFIC OBJECTIVES: • Atthe end of the lecture, student nurses should be able to; • Define pin worm infestation • Describe the life cycle of the pin worms. • Outline how pin worms are diagnosed • Describe the treatment of pin worms. • Outline the preventive measure of pin worms
  • 4.
    INTRODUCTION • Pin worminfestation occur world wide and is most common among children 5 – 10 years old . • In the USA it is estimated that there are more than 40 million cases each year. • Pinworm infections occur in all socioeconomic groups; however, human-to-human spread is favored by close, crowded living conditions
  • 5.
    PINWORM INFESTATION • Pinworminfestation is a type of human intestinal infection by the pinworm. • Pinworms are tiny, narrow worms that are white about one cm long. • Pinworms are also known as Enterobius Vermiacularis Schull (2005)
  • 7.
    LIFE CYCLE • Thecycle of infection begins with the ingestion of the eggs. • The eggs enter the small intestine until they hatch into larvae and mature. • The adult female pinworms moves into the colon where they live. • At night the female adult exit the body through the anus and lay eggs in the folds of skin around the anus.
  • 8.
    • The presenceof these eggs often causes itching and irritation. • When a person scratches the affected area, the pinworm eggs are transferred to the fingers. • The eggs can survive for several hours on the hands
  • 9.
    • . Ifthe infected person touches bedding, clothing, toilet seats, toys, or other household objects, the eggs are transferred to these surfaces. • Pinworm eggs can survive on these contaminated surfaces for up to three weeks.
  • 10.
    • The transferof pinworm eggs is easier among children, who may put infected toys or other objects into their mouths. • The eggs can also be transferred from contaminated fingers to food or liquids. • it’s also possible for adults to inhale eggs that become airborne when bedding or clothing is shaken.
  • 11.
    Signs and symptoms •frequent and strong itching of the anal area • restless sleep due to the itching and discomfort • pain, rash, or other skin irritation around the anus • the presence of pinworms in the area of the child’s anus • the presence of pinworms in stools
  • 12.
    diagnosis • Finding eggson the cellophane tape or scotch tape placed on the skin around the anus. • The laid eggs will stick to the tape. • The tape is placed over night and check the following morning. • The tape is placed on the slide and examined under the microscope. • The pin worm eggs are seen.
  • 13.
    DRUGS • Mebendazole 100mgBD for 3 days. • Aldendazole 400 mg single dose. • Piperazine citrate 50 to 75mg per kg bwt OD for 2 days. • Lavemisole 150mg for adult and 5mg per kg bwt for children • Nitazoxanide 200 mg BD x 3 days • Tribendimine 400 mg single dose
  • 14.
    prevention • Ensure infectedperson and family members practice hand washing with warm water and soap before eating. • . • Clean everyone’s fingernails and cut them short. • infected person and others to stop biting their nails.
  • 15.
    • Avoid shakingclothing and bedding to prevent pinworm eggs from spreading into the air. • Do not allow children to bathe together to prevent spreading pinworm eggs in the bath water. • Encourage everyone to shower and change their underwear every morning
  • 16.
    • infected personto refrain from scratching the anal area. • Use hot water to launder all bedding, towels, washcloths, and clothes in the affected house. • Thoroughly clean any surfaces that may be infected, including toys, floors, countertops, and toilet seats.
  • 17.
  • 18.
    SPECIFIC OBJECTIVES • Atthe end of the lecture/discussion, the students should be able to • Define hookworm infestation • Describe the life cycle of hookworms. • Explain the signs and symptoms of hookworms • Describe how hookworms are diagnosed. • Mention the treatment of hookworms. • Outline the prevention of hook worms
  • 19.
    DEFINITION • Hookworm infestationis the infection of the small intestine with hook worms.(Scull 2005) • There are two types of hookworms namely: 1. Necator Americanus 2. Ancylostoma Dueodenale
  • 21.
    LIFE CYCLE OFHOOKWORMS • The infective larvae penetrate the intact skin especially the feet that are in contact with the ground. • The infective larvae are carried through the bloodstream to the heart then to the lungs.. • The infective larvae penetrate the pulmonary alveoli.
  • 22.
    • The larvaeenter the bronchial tree into the trachea, the pharynx and are swallowed. • The larvae go into the small intestine where they mature and become adult worms. • The adult hookworms attach to the wall of the intestine with buccal capsure –
  • 24.
    • The adultmale and female met and the female lay eggs that come out in the faces. • The eggs are passed into the soil and under favourable condition hatch into infective larvae. • The favourable condition includes moisture, warm and shady environment.
  • 25.
    SIGNS AND SYMPTOMS •Ground itch itchiness and small rash caused by an allergic reaction as the larvae enter the skin. • Diarrhoea • Abdominal pain or colic • Intestinal cramps • Nausea • Fever
  • 26.
    • Blood instool • Loss of appetite • Other signs are • Anemia due to the worms feeding on blood • Malnutrition due to the worms feed feeding on the food that the person eats. • Ascites due to protein deficiency.
  • 27.
    diagnosis • Stool examination •Eggs in fresh stool less than 24hrs • Larvae in old stool more than 24hrs • Blood test • Eosinophilia raised eosinophils level
  • 28.
    Prevention of hookworms •Using protective clothes like gumboots for the farmers and fishermen • Dress the children in shoes. • Mass treatment of all the under 5 children • Mass treatment of the people in the location where many people are diagnosed with hook worms
  • 29.
    • Use oftoilets for defecation to prevent soil contamination • Good hand hygiene after toilet and before eating. • Food hygiene that is washing all the fruits and other raw foods before eating. • Information education communication to the community on sanitation
  • 30.
  • 31.
    DEFINITION • Round worminfestation is the infection of the intestine by round worms. • Round worm also called Ascaris Lumbricoides. are the largest nematode parasitizing the human intestine. • Adult females: 20 to 35 cm; adult male: 15 to 30 cm.
  • 33.
    Life cycle • Theinfective eggs are swallowed goes into the small intestine • In the small intestine the eggs hatch into larvae. • The larvae penetrates the intestinal wall into the portal vein and go to the liver. • The larvae then goes to the lungs passing through the heart.
  • 34.
    • In thelungs the larvae mature after 10 to 14 days. • The larvae penetrates the alveolar wall, ascend the bronchial tree to the throat and are swallowed. • Upon reaching the small intestine, they develop into adult worms.
  • 35.
    • Between 2and 3 months are required from ingestion of the infective eggs to when the adult female lay eges. • Adult worms can live 1 to 2 years in the lumen of the small intestine
  • 36.
    • A femalemay produce approximately 200,000 eggs per day. • Eggs are passed in the feces. • Unfertilized eggs are not infective. • Fertile embryonate eggs become infective after 18 days to several weeks, depending on the environmental conditions
  • 37.
    Signs and stmptoms •Pulmonary manifestation called Loeffler,s syndrome includes: • . mild to severe cough • . dyspnoe • .thoracic pain • . fever
  • 38.
    • Abdominal signsand symptoms. • .Abdominal pain • .Anorexia • . Nausea • .Diarrhoea • .Heavy infestation leads to impaired food nutrients absorption
  • 39.
    diagnosis • Stool examination •Detection of eggs in faces-no concentration technique is needed worm lay 200,000 eggs per day. • Full blood count • There is significant eosinophilia during lung passage
  • 40.
    • x-ray ofthe intestine • Show one or more worms .worms form a long thin dark area if using barium contrast. • Ultra sound of the pancreas biliary tract and gall bladder show ectopic migration
  • 41.
    COMPLICATION OF ROUNDWORMS • Intestinal obstruction • numerous adult worms may form a ball and cause mechanical intestinal obstruction. • • Biliary obstruction • Migration into the biliary tract may lead to biliary obstruction (cholestasis) with possibly infection (cholangitis, liver abscess). • • Pancreatic duct obstruction • Migration into the pancreatic duct can cause obstruction( pancreatitis)
  • 42.
    • Appendicitis • Sometimesthere is migration to the appendix with inflammation. • Sometimes an adult Ascaris is present in vomitus. • Occasionally, an adult can penetrate the lacrimal duct.
  • 43.
    Prevention of roundworms • Mass treatment of the under 5 children • Mass treatment of the people in the location where there man people are being diagnosed with worm infestation • Use of toilets for defecation to prevent soil contamination
  • 44.
    • Good handhygiene after toilet and before eating. • Food hygiene that is washing all the fruits and other raw foods before eating. • Information education communication to the community on sanitation
  • 45.
  • 46.
    • Tape wormsare flat segmented worms that live in the intestine of animals including man. • The most common tape worms to man are beef tape worms called taenia saginata and pork tape worm called taenia solium.
  • 47.
    • Man getsinfected with tape worms by eating raw or under cooked beef or pork from infected animals.
  • 48.
    BEEF TAPE WORMS(Taeniasaginata) • Cause taeniasis. • The head ( scolex) has 4 hemispherical suckers. The scolex imbeds in the mucosa of the intestine. • The neck is the budding zone from which segments are formed. • The strobilus consists of immature, mature and pregnant proglottides. Proglottides are segments
  • 49.
    • There arehermaphroditic (there contain both male and female reproductive organs in every mature proglottid). • Digestive tract is absent. Nutrition is absorbed by the villi of the body surface. • All adult worms infest the digestive tract of mammals.
  • 50.
    PORK TAPE WORM(taeniasolium) • The scolex(head) is global with 4 suckers and 1 rostellum • The rostellum is for attaching to the mucosa and the suckers are for sucking blood. • The rostellum has 25 -50 hooklets for attaching to the mucosa
  • 51.
  • 52.
    LIFE CYCLE • INMAN • infection is by ingesting of raw or under cooked containing cystececus (pork and beef). • In the intestine the cystececus develop into adult tape worms after two months which can survive for years. • The adult worm attaches to the mucosa by the scolex and reside in the small intestine
  • 53.
    • The adulttape worm produce proglottids which mature, become gravid and are passed into stool. • The eggs are released after the proglottids are passed in stool. • The taenia solium cystececus invades the intestinal mucosa enter the bloodstream and lodge in the brain, skin, tongue or eyes.
  • 54.
    IN ANIMALS • Cattleor pig get infected by ingesting eggs or gravid proglottids during grazing. • In the animal intestine the eggs hatch into larvae (oncospheres). • The oncospheres invades the intestinal wall, migrate through blood stream to the muscles where it encysts, forming cysticerci
  • 56.
    Signs and symptoms •Taeniasis • Taeniasis is caused by the adult tape worm residing in the small intestine. • The adult worm irritates the small intestine causing: • Abdominal pain
  • 57.
    • Anorexia • Chronicindigestion • Diarrhea • Weight loss.
  • 58.
    CYSTICERCOSIS • Caused bycysticerci living in human tissue. Cysticercosis is divided into three types • • Subcutaneous type: • The subcutaneous nodules usually found on the head, limbs, abdomen and back. • Are movable and painless
  • 59.
    • Ocular type •The Cysticercus is usually found in the vitreous body or subretina. • Visual disturbance often occurs. • The dead worms may provoke local inflammation causing blindness.
  • 60.
    • Brain type: •The patients may manifest with headache, nausea, vomiting, and epilepsy, and paralysis, weakness in limbs, dizziness, and mental disorder. • Epilepsy is the most frequent symptom of brain cysticercosis.
  • 61.
    Diagnosis • 1.Taeniasis: • • Findinggravid proglottides or eggs in stool. • 2. Cysticercosis: • Biopsy of subcutaneous nodules, • X-ray, CT or MR for brain type • Ophthalmoscope examination: • for ocular type.
  • 62.
    TREATMENT Taeniasis • Praziquantel 5-10 mg per kg body weight single dose • Niclosamide adults and children above 6 years give 2g single dose after light breakfast followed after 2 hours by a laxative
  • 63.
    Cysticercus Excision - removalof cyst. • Praziquantel - destroys Cysticercus in brain • Metrifonate- for cutaneous type • Corticosteroid reduce the inflammatory process
  • 64.
    PREVENTION • Health educationon how to cook meat • Avoid eating raw pork. • Avoid pigs eating human stool. • Inspection of slaughter houses. • Sanitary disposal of night soil. • Improved meat inspection • Improved meat processing • Deworming all the pigs
  • 65.
    SUMMARY • Tape wormsare contracted by ingestion of cysticerci in the raw or under cooked meat . Man pass proglottids which contain eggs in stool. • Pigs or cattle get infected by ingesting the eggs, the larvae form cysticerci the muscle. Tape worms are prevented by eating well cooked meat
  • 66.
    • REFERENCES • Cook.G.Cand Zumla (2003), Manson’s Tropical Disease, 21st Edition, Saunders, Elsevier Sciences Division, 32 Jamestown Road London. • Schull C.R (2005), Common Medical Problems in the Tropics, 2nd Edition macmillan Education Between •