WORM INFESTATION
Medicine andmedical Nursing
Presenter :George masiye
5th
year students
Unza
Supervisor Mr P. Mwanza
2.
GENERAL OBJECTIVE
• Atthe end of this lecture / discussion students
should be able demonstrate an understanding
of pin worms infestation
3.
SPECIFIC OBJECTIVES:
• Atthe end of the lecture, student nurses
should be able to;
• Define pin worm infestation
• Describe the life cycle of the pin worms.
• Outline how pin worms are diagnosed
• Describe the treatment of pin worms.
• Outline the preventive measure of pin worms
4.
INTRODUCTION
• Pin worminfestation occur world wide and is
most common among children 5 – 10 years old .
• In the USA it is estimated that there are more
than 40 million cases each year.
• Pinworm infections occur in all socioeconomic
groups; however, human-to-human spread is
favored by close, crowded living conditions
5.
PINWORM INFESTATION
• Pinworminfestation is a type of human
intestinal infection by the pinworm.
• Pinworms are tiny, narrow worms that are
white about one cm long.
• Pinworms are also known as Enterobius
Vermiacularis Schull (2005)
7.
LIFE CYCLE
• Thecycle of infection begins with the ingestion
of the eggs.
• The eggs enter the small intestine until they
hatch into larvae and mature.
• The adult female pinworms moves into the
colon where they live.
• At night the female adult exit the body through
the anus and lay eggs in the folds of skin around
the anus.
8.
• The presenceof these eggs often causes
itching and irritation.
• When a person scratches the affected area,
the pinworm eggs are transferred to the
fingers.
• The eggs can survive for several hours on the
hands
9.
• . Ifthe infected person touches bedding,
clothing, toilet seats, toys, or other household
objects, the eggs are transferred to these
surfaces.
• Pinworm eggs can survive on these
contaminated surfaces for up to three weeks.
10.
• The transferof pinworm eggs is easier among
children, who may put infected toys or other
objects into their mouths.
• The eggs can also be transferred from
contaminated fingers to food or liquids.
• it’s also possible for adults to inhale eggs that
become airborne when bedding or clothing is
shaken.
11.
Signs and symptoms
•frequent and strong itching of the anal area
• restless sleep due to the itching and
discomfort
• pain, rash, or other skin irritation around the
anus
• the presence of pinworms in the area of the
child’s anus
• the presence of pinworms in stools
12.
diagnosis
• Finding eggson the cellophane tape or scotch tape
placed on the skin around the anus.
• The laid eggs will stick to the tape.
• The tape is placed over night and check the
following morning.
• The tape is placed on the slide and examined
under the microscope.
• The pin worm eggs are seen.
13.
DRUGS
• Mebendazole 100mgBD for 3 days.
• Aldendazole 400 mg single dose.
• Piperazine citrate 50 to 75mg per kg bwt OD
for 2 days.
• Lavemisole 150mg for adult and 5mg per kg
bwt for children
• Nitazoxanide 200 mg BD x 3 days
• Tribendimine 400 mg single dose
14.
prevention
• Ensure infectedperson and family members
practice hand washing with warm water and
soap before eating.
• .
• Clean everyone’s fingernails and cut them
short.
• infected person and others to stop biting their
nails.
15.
• Avoid shakingclothing and bedding to prevent
pinworm eggs from spreading into the air.
• Do not allow children to bathe together to
prevent spreading pinworm eggs in the bath
water.
• Encourage everyone to shower and change their
underwear every morning
16.
• infected personto refrain from scratching the
anal area.
• Use hot water to launder all bedding, towels,
washcloths, and clothes in the affected house.
• Thoroughly clean any surfaces that may be
infected, including toys, floors, countertops,
and toilet seats.
SPECIFIC OBJECTIVES
• Atthe end of the lecture/discussion, the
students should be able to
• Define hookworm infestation
• Describe the life cycle of hookworms.
• Explain the signs and symptoms of hookworms
• Describe how hookworms are diagnosed.
• Mention the treatment of hookworms.
• Outline the prevention of hook worms
19.
DEFINITION
• Hookworm infestationis the infection of the
small intestine with hook worms.(Scull 2005)
• There are two types of hookworms namely:
1. Necator Americanus
2. Ancylostoma Dueodenale
21.
LIFE CYCLE OFHOOKWORMS
• The infective larvae penetrate the intact skin
especially the feet that are in contact with the
ground.
• The infective larvae are carried through the
bloodstream to the heart then to the lungs..
• The infective larvae penetrate the pulmonary
alveoli.
22.
• The larvaeenter the bronchial tree into the
trachea, the pharynx and are swallowed.
• The larvae go into the small intestine where
they mature and become adult worms.
• The adult hookworms attach to the wall of the
intestine with buccal capsure
–
24.
• The adultmale and female met and the female
lay eggs that come out in the faces.
• The eggs are passed into the soil and under
favourable condition hatch into infective larvae.
• The favourable condition includes moisture,
warm and shady environment.
25.
SIGNS AND SYMPTOMS
•Ground itch itchiness and small rash caused
by an allergic reaction as the larvae enter the
skin.
• Diarrhoea
• Abdominal pain or colic
• Intestinal cramps
• Nausea
• Fever
26.
• Blood instool
• Loss of appetite
• Other signs are
• Anemia due to the worms feeding on blood
• Malnutrition due to the worms feed feeding
on the food that the person eats.
• Ascites due to protein deficiency.
27.
diagnosis
• Stool examination
•Eggs in fresh stool less than 24hrs
• Larvae in old stool more than 24hrs
• Blood test
• Eosinophilia raised eosinophils level
28.
Prevention of hookworms
•Using protective clothes like gumboots for the
farmers and fishermen
• Dress the children in shoes.
• Mass treatment of all the under 5 children
• Mass treatment of the people in the location
where many people are diagnosed with hook
worms
29.
• Use oftoilets for defecation to prevent soil
contamination
• Good hand hygiene after toilet and before
eating.
• Food hygiene that is washing all the fruits and
other raw foods before eating.
• Information education communication to the
community on sanitation
DEFINITION
• Round worminfestation is the infection of the
intestine by round worms.
• Round worm also called Ascaris Lumbricoides.
are the largest nematode parasitizing the
human intestine.
• Adult females: 20 to 35 cm; adult male: 15 to
30 cm.
33.
Life cycle
• Theinfective eggs are swallowed goes into
the small intestine
• In the small intestine the eggs hatch into
larvae.
• The larvae penetrates the intestinal wall into
the portal vein and go to the liver.
• The larvae then goes to the lungs passing
through the heart.
34.
• In thelungs the larvae mature after 10 to 14
days.
• The larvae penetrates the alveolar wall,
ascend the bronchial tree to the throat and
are swallowed.
• Upon reaching the small intestine, they
develop into adult worms.
35.
• Between 2and 3 months are required from
ingestion of the infective eggs to when the
adult female lay eges.
• Adult worms can live 1 to 2 years in the
lumen of the small intestine
36.
• A femalemay produce approximately 200,000
eggs per day.
• Eggs are passed in the feces.
• Unfertilized eggs are not infective.
• Fertile embryonate eggs become infective
after 18 days to several weeks, depending on
the environmental conditions
37.
Signs and stmptoms
•Pulmonary manifestation called Loeffler,s
syndrome includes:
• . mild to severe cough
• . dyspnoe
• .thoracic pain
• . fever
diagnosis
• Stool examination
•Detection of eggs in faces-no concentration
technique is needed worm lay 200,000 eggs
per day.
• Full blood count
• There is significant eosinophilia during lung
passage
40.
• x-ray ofthe intestine
• Show one or more worms .worms form a long
thin dark area if using barium contrast.
• Ultra sound of the pancreas biliary tract and
gall bladder show ectopic migration
41.
COMPLICATION OF ROUNDWORMS
• Intestinal obstruction
• numerous adult worms may form a ball and cause mechanical
intestinal obstruction.
•
• Biliary obstruction
• Migration into the biliary tract may lead to biliary obstruction
(cholestasis) with possibly infection (cholangitis, liver abscess).
•
• Pancreatic duct obstruction
• Migration into the pancreatic duct can cause
obstruction( pancreatitis)
42.
• Appendicitis
• Sometimesthere is migration to the appendix
with inflammation.
• Sometimes an adult Ascaris is present in vomitus.
• Occasionally, an adult can penetrate the lacrimal
duct.
43.
Prevention of roundworms
• Mass treatment of the under 5 children
• Mass treatment of the people in the location
where there man people are being diagnosed
with worm infestation
• Use of toilets for defecation to prevent soil
contamination
44.
• Good handhygiene after toilet and before
eating.
• Food hygiene that is washing all the fruits and
other raw foods before eating.
• Information education communication to the
community on sanitation
• Tape wormsare flat segmented worms that
live in the intestine of animals including man.
• The most common tape worms to man are
beef tape worms called taenia saginata and
pork tape worm called taenia solium.
47.
• Man getsinfected with tape worms by eating
raw or under cooked beef or pork from
infected animals.
48.
BEEF TAPE WORMS(Taeniasaginata)
• Cause taeniasis.
• The head ( scolex) has 4 hemispherical suckers.
The scolex imbeds in the mucosa of the intestine.
• The neck is the budding zone from which
segments are formed.
• The strobilus consists of immature, mature and
pregnant proglottides. Proglottides are segments
49.
• There arehermaphroditic (there contain both
male and female reproductive organs in every
mature proglottid).
• Digestive tract is absent. Nutrition is absorbed
by the villi of the body surface.
• All adult worms infest the digestive tract of
mammals.
50.
PORK TAPE WORM(taeniasolium)
• The scolex(head) is global with 4 suckers and 1
rostellum
• The rostellum is for attaching to the mucosa
and the suckers are for sucking blood.
• The rostellum has 25 -50 hooklets for
attaching to the mucosa
LIFE CYCLE
• INMAN
• infection is by ingesting of raw or under
cooked containing cystececus (pork and beef).
• In the intestine the cystececus develop into
adult tape worms after two months which can
survive for years.
• The adult worm attaches to the mucosa by the
scolex and reside in the small intestine
53.
• The adulttape worm produce proglottids
which mature, become gravid and are passed
into stool.
• The eggs are released after the proglottids are
passed in stool.
• The taenia solium cystececus invades the
intestinal mucosa enter the bloodstream and
lodge in the brain, skin, tongue or eyes.
54.
IN ANIMALS
• Cattleor pig get infected by ingesting eggs or
gravid proglottids during grazing.
• In the animal intestine the eggs hatch into
larvae (oncospheres).
• The oncospheres invades the intestinal wall,
migrate through blood stream to the muscles
where it encysts, forming cysticerci
56.
Signs and symptoms
•Taeniasis
• Taeniasis is caused by the adult tape worm
residing in the small intestine.
• The adult worm irritates the small intestine
causing:
• Abdominal pain
CYSTICERCOSIS
• Caused bycysticerci living in human tissue.
Cysticercosis is divided into three types
•
• Subcutaneous type:
• The subcutaneous nodules usually found on
the head, limbs, abdomen and back.
• Are movable and painless
59.
• Ocular type
•The Cysticercus is usually found in the vitreous
body or subretina.
• Visual disturbance often occurs.
• The dead worms may provoke local
inflammation causing blindness.
60.
• Brain type:
•The patients may manifest with headache, nausea,
vomiting, and epilepsy, and paralysis, weakness in
limbs, dizziness, and mental disorder.
• Epilepsy is the most frequent symptom of brain
cysticercosis.
61.
Diagnosis
• 1.Taeniasis:
•
• Findinggravid proglottides or eggs in stool.
• 2. Cysticercosis:
• Biopsy of subcutaneous nodules,
• X-ray, CT or MR for brain type
• Ophthalmoscope examination:
• for ocular type.
62.
TREATMENT
Taeniasis
• Praziquantel 5-10 mg per kg body weight
single dose
• Niclosamide adults and children above 6
years give 2g single dose after light breakfast
followed after 2 hours by a laxative
63.
Cysticercus
Excision - removalof cyst.
• Praziquantel - destroys Cysticercus in brain
• Metrifonate- for cutaneous type
• Corticosteroid reduce the inflammatory
process
64.
PREVENTION
• Health educationon how to cook meat
• Avoid eating raw pork.
• Avoid pigs eating human stool.
• Inspection of slaughter houses.
• Sanitary disposal of night soil.
• Improved meat inspection
• Improved meat processing
• Deworming all the pigs
65.
SUMMARY
• Tape wormsare contracted by ingestion of
cysticerci in the raw or under cooked meat .
Man pass proglottids which contain eggs in
stool.
• Pigs or cattle get infected by ingesting the
eggs, the larvae form cysticerci the muscle.
Tape worms are prevented by eating well
cooked meat
66.
• REFERENCES
• Cook.G.Cand Zumla (2003), Manson’s Tropical
Disease, 21st
Edition, Saunders, Elsevier
Sciences Division, 32 Jamestown Road London.
• Schull C.R (2005), Common Medical Problems in
the Tropics, 2nd
Edition macmillan Education
Between
•